about
Microglial CD33-related Siglec-E inhibits neurotoxicity by preventing the phagocytosis-associated oxidative burstTREM2-transduced myeloid precursors mediate nervous tissue debris clearance and facilitate recovery in an animal model of multiple sclerosis.A retroviral packaging cell line for pseudotype vectors based on glioma-infiltrating progenitor cells.Dual induction of TREM2 and tolerance-related transcript, Tmem176b, in amyloid transgenic mice: implications for vaccine-based therapies for Alzheimer's disease.Attenuated inflammatory response in triggering receptor expressed on myeloid cells 2 (TREM2) knock-out mice following stroke.Activated microglia/macrophage whey acidic protein (AMWAP) inhibits NFκB signaling and induces a neuroprotective phenotype in microglia.Polysialic acid blocks mononuclear phagocyte reactivity, inhibits complement activation, and protects from vascular damage in the retinaAge-related macular degeneration associated polymorphism rs10490924 in ARMS2 results in deficiency of a complement activator.Anti-inflammatory activity of low molecular weight polysialic acid on human macrophages.Integrated systems approach identifies genetic nodes and networks in late-onset Alzheimer's disease.Neuronal 'On' and 'Off' signals control microglia.Debris clearance by microglia: an essential link between degeneration and regeneration.Microglial clearance function in health and disease.Protective effects of microglia in multiple sclerosis.Translocator protein (18 kDa) (TSPO) is expressed in reactive retinal microglia and modulates microglial inflammation and phagocytosisMicroglial carbohydrate-binding receptors for neural repair.Microglial activatory (immunoreceptor tyrosine-based activation motif)- and inhibitory (immunoreceptor tyrosine-based inhibition motif)-signaling receptors for recognition of the neuronal glycocalyx.Janus-faced microglia: beneficial and detrimental consequences of microglial phagocytosis.Siglec functions of microglia.Sensing the neuronal glycocalyx by glial sialic acid binding immunoglobulin-like lectins.Neuronal injury mediated via stimulation of microglial toll-like receptor-9 (TLR9).Characterization of inflammatory markers and transcriptome profiles of differentially activated embryonic stem cell-derived microglia.Microglia: a cellular vehicle for CNS gene therapy.Targeting the cytosolic innate immune receptors RIG-I and MDA5 effectively counteracts cancer cell heterogeneity in glioblastoma.Microglial precursors derived from mouse embryonic stem cells.Brain endothelial PPARgamma controls inflammation-induced CD4+ T cell adhesion and transmigration in vitro.Bystander killing of malignant glioma by bone marrow-derived tumor-infiltrating progenitor cells expressing a suicide gene.Suppression of microglial inflammatory activity by myelin phagocytosis: role of p47-PHOX-mediated generation of reactive oxygen species.Sialylation of neurites inhibits complement-mediated macrophage removal in a human macrophage-neuron Co-Culture System.Clearance of apoptotic neurons without inflammation by microglial triggering receptor expressed on myeloid cells-2.Sequential proteolytic processing of the triggering receptor expressed on myeloid cells-2 (TREM2) protein by ectodomain shedding and γ-secretase-dependent intramembranous cleavageEngineered stem cell-derived microglia as therapeutic vehicle for experimental autoimmune encephalomyelitis.Unique transcriptome signature of mouse microglia.Neurodegeneration by activation of the microglial complement-phagosome pathway.Functional role of brain-derived neurotrophic factor in neuroprotective autoimmunity: therapeutic implications in a model of multiple sclerosis.Spatially pathogenic forms of tau detected in Alzheimer's disease brain tissue by fluorescence lifetime-based Förster resonance energy transfer.Intramembranous processing by γ-secretase regulates reverse signaling of ephrin-B2 in migration of microglia.Alleviation of neurotoxicity by microglial human Siglec-11.Unloading kinesin transported cargoes from the tubulin track via the inflammatory c-Jun N-terminal kinase pathway.Licensing of myeloid cells promotes central nervous system autoimmunity and is controlled by peroxisome proliferator-activated receptor γ.
P50
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P50
description
hulumtues
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Harald Neumann
@ast
Harald Neumann
@en
Harald Neumann
@es
Harald Neumann
@nl
Harald Neumann
@sl
type
label
Harald Neumann
@ast
Harald Neumann
@en
Harald Neumann
@es
Harald Neumann
@nl
Harald Neumann
@sl
prefLabel
Harald Neumann
@ast
Harald Neumann
@en
Harald Neumann
@es
Harald Neumann
@nl
Harald Neumann
@sl
P1053
A-9718-2010
P106
P21
P2798
P31
P3829
P496
0000-0002-5071-5202