Sendai virus targets inflammatory responses, as well as the interferon-induced antiviral state, in a multifaceted manner.
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RIG-I and dsRNA-induced IFNbeta activation.Analysis of genes induced by Sendai virus infection of mutant cell lines reveals essential roles of interferon regulatory factor 3, NF-kappaB, and interferon but not toll-like receptor 3Differential role for TLR3 in respiratory syncytial virus-induced chemokine expression.Interplay between innate immunity and negative-strand RNA viruses: towards a rational modelInhibition of interleukin-6 expression by the V protein of parainfluenza virus 5Nonstructural Nipah virus C protein regulates both the early host proinflammatory response and viral virulence.Activation of the beta interferon promoter by unnatural Sendai virus infection requires RIG-I and is inhibited by viral C proteinsGeneration of recombinant human parainfluenza virus type 1 vaccine candidates by importation of temperature-sensitive and attenuating mutations from heterologous paramyxoviruses.The C proteins of human parainfluenza virus type 1 (HPIV1) control the transcription of a broad array of cellular genes that would otherwise respond to HPIV1 infectionTopoisomerase 1 inhibition suppresses inflammatory genes and protects from death by inflammation.The regulation of type I interferon production by paramyxoviruses.Disruption of MDA5-Mediated Innate Immune Responses by the 3C Proteins of Coxsackievirus A16, Coxsackievirus A6, and Enterovirus D68.Global quantitative proteomic analysis profiles host protein expression in response to Sendai virus infection.Loss of Sendai virus C protein leads to accumulation of RIG-I immunostimulatory defective interfering RNA.Targeting of the Sendai virus C protein to the plasma membrane via a peptide-only membrane anchor.Type I Interferon-Mediated Induction of Antiviral Genes and Proteins Fails to Protect Cells from the Cytopathic Effects of Sendai Virus Infection.The double-stranded RNA binding domain of the vaccinia virus E3L protein inhibits both RNA- and DNA-induced activation of interferon beta.Persistent and stable gene expression by a cytoplasmic RNA replicon based on a noncytopathic variant Sendai virus.
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Sendai virus targets inflammatory responses, as well as the interferon-induced antiviral state, in a multifaceted manner.
description
2003 nî lūn-bûn
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2003年の論文
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2003年学术文章
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2003年学术文章
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2003年学术文章
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2003年学术文章
@zh-my
2003年学术文章
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2003年學術文章
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2003年學術文章
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2003年學術文章
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name
Sendai virus targets inflammat ...... ate, in a multifaceted manner.
@en
type
label
Sendai virus targets inflammat ...... ate, in a multifaceted manner.
@en
prefLabel
Sendai virus targets inflammat ...... ate, in a multifaceted manner.
@en
P2093
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Sendai virus targets inflammat ...... ate, in a multifaceted manner.
@en
P2093
Daniel Kolakofsky
Dominique Garcin
Laura Strähle
Philippe Le Mercier
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P356
10.1128/JVI.77.14.7903-7913.2003
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2003-07-01T00:00:00Z