Role and mechanism of decitabine combined with tyrosine kinase inhibitors in advanced chronic myeloid leukemia cells.

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Role and mechanism of decitabine combined with tyrosine kinase inhibitors in advanced chronic myeloid leukemia cells.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

@zh-my

2017年学术文章

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2017年學術文章

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2017年學術文章

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2017年學術文章

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Role and mechanism of decitabi ...... hronic myeloid leukemia cells.

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Role and mechanism of decitabi ...... hronic myeloid leukemia cells.

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Role and mechanism of decitabi ...... hronic myeloid leukemia cells.

@en

P1476

Role and mechanism of decitabi ...... hronic myeloid leukemia cells.

@en

P2093

Jian-Min Luo

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Li-Cai Jiang

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P2860

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Results of decitabine (5-aza-2'deoxycytidine) therapy in 130 patients with chronic myelogenous leukemia

Regulation of Bcr-Abl-induced SAP kinase activity and transformation by the SHPTP1 protein tyrosine phosphatase

Human chronic myelogenous leukemia cell-line with positive Philadelphia chromosome

STAT3- and DNA methyltransferase 1-mediated epigenetic silencing of SHP-1 tyrosine phosphatase tumor suppressor gene in malignant T lymphocytes

Chronic myeloid leukemia--advances in biology and new approaches to treatment.

From form to function: signaling by protein tyrosine phosphatases.

Imatinib mesylate--a new oral targeted therapy.

The function of the protein tyrosine phosphatase SHP-1 in cancer.

The COOH terminus of the c-Abl tyrosine kinase contains distinct F- and G-actin binding domains with bundling activity.

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1295-1302

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scholarly article

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10.3892/OL.2017.6318

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P478

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P577

2017-06-06T00:00:00Z

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P698

28789344

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P921

chronic myeloid leukemia

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5529866

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