Glycogen synthase kinase-3 inhibitors suppress the AR-V7-mediated transcription and selectively inhibit cell growth in AR-V7-positive prostate cancer cells.

about

Glycogen synthase kinase-3 inhibitors suppress the AR-V7-mediated transcription and selectively inhibit cell growth in AR-V7-positive prostate cancer cells.

Item

http://www.wikidata.org/entity/Q38708121

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh

2017年學術文章

@zh-hant

name

Glycogen synthase kinase-3 inh ...... ositive prostate cancer cells.

@en

type

Item

label

Glycogen synthase kinase-3 inh ...... ositive prostate cancer cells.

@en

prefLabel

Glycogen synthase kinase-3 inh ...... ositive prostate cancer cells.

@en

P1476

Glycogen synthase kinase-3 inh ...... positive prostate cancer cells

@en

P2093

Kazuhide Nakayama

http://www.w3.org/2001/XMLSchema#string

Ryokichi Koyama

http://www.w3.org/2001/XMLSchema#string

Satoshi Kitazawa

http://www.w3.org/2001/XMLSchema#string

Takahito Hara

http://www.w3.org/2001/XMLSchema#string

Tatsuya Watanabe

http://www.w3.org/2001/XMLSchema#string

P2860

Regression of castrate-recurrent prostate cancer by a small-molecule inhibitor of the amino-terminus domain of the androgen receptor

Emerging mechanisms of resistance to androgen receptor inhibitors in prostate cancer

AR-V7 and resistance to enzalutamide and abiraterone in prostate cancer

A novel androgen receptor splice variant is up-regulated during prostate cancer progression and promotes androgen depletion-resistant growth

Liganded androgen receptor interaction with beta-catenin: nuclear co-localization and modulation of transcriptional activity in neuronal cells

Linking beta-catenin to androgen-signaling pathway

Suppression of androgen receptor-mediated transactivation and cell growth by the glycogen synthase kinase 3 beta in prostate cells

The renaissance of GSK3

Ligand-independent androgen receptor variants derived from splicing of cryptic exons signify hormone-refractory prostate cancer

Castration resistance in human prostate cancer is conferred by a frequently occurring androgen receptor splice variant.

P304

955-961

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1002/PROS.23351

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Daisuke Nakata

P577

2017-04-10T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

28397338

http://www.w3.org/2001/XMLSchema#string