Loss of CUL4A expression is underlying cisplatin hypersensitivity in colorectal carcinoma cells with acquired trabectedin resistance.

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Altered membrane rigidity via enhanced endogenous cholesterol synthesis drives cancer cell resistance to destruxins.The thiosemicarbazone MeNNMe induces paraptosis by disrupting the ER thiol redox homeostasis based on protein disulfide isomerase inhibition

P2860

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Loss of CUL4A expression is underlying cisplatin hypersensitivity in colorectal carcinoma cells with acquired trabectedin resistance.

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2017 nî lūn-bûn

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Loss of CUL4A expression is un ...... quired trabectedin resistance.

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Loss of CUL4A expression is un ...... quired trabectedin resistance.

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Loss of CUL4A expression is un ...... quired trabectedin resistance.

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P2860

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Loss of heterozygosity of nucleotide excision repair factors in sporadic ovarian, colon and lung carcinomas: implication for their roles of carcinogenesis in human solid tumors.

Ecteinascidin 743 induces protein-linked DNA breaks in human colon carcinoma HCT116 cells and is cytotoxic independently of topoisomerase I expression.

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bjc.2016.449

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489-500

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Loss of CUL4A expression is underlying cisplatin hypersensitivity in colorectal carcinoma cells with acquired trabectedin resistance.

about

P2860

P2860

Loss of CUL4A expression is underlying cisplatin hypersensitivity in colorectal carcinoma cells with acquired trabectedin resistance.

description

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type

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P1476

P2093

P2860

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P356

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P2093

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P433

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P932