Targeting oxidative stress improves disease outcomes in a rat model of acquired epilepsy. - Wikidata - wikimedia - TriplyDB

Targeting oxidative stress improves disease outcomes in a rat model of acquired epilepsy.

Targeting oxidative stress improves disease outcomes in a rat model of acquired epilepsy.

http://www.wikidata.org/entity/Q38748531

about

Biomarkers of Epileptogenesis: The Focus on Glia and Cognitive Dysfunctions.Metabolic and Homeostatic Changes in Seizures and Acquired Epilepsy-Mitochondria, Calcium Dynamics and Reactive Oxygen Species.A combination of NMDA and AMPA receptor antagonists retards granule cell dispersion and epileptogenesis in a model of acquired epilepsy Metabolic Dysfunction and Oxidative Stress in Epilepsy.Commonalities in epileptogenic processes from different acute brain insults: Do they translate?Pitfalls in the quantitative imaging of glutathione in living cells.Inflammation: A Network in the Pathogenesis of Status Epilepticus The Free Radical Scavenger N-Tert-Butyl-α-Phenylnitrone (PBN) Administered to Immature Rats During Status Epilepticus Alters Neurogenesis and Has Variable Effects, Both Beneficial and Detrimental, on Long-Term Outcomes A companion to the preclinical common data elements for pharmacologic studies in animal models of seizures and epilepsy. A Report of the TASK3 Pharmacology Working Group of the ILAE/AES Joint Translational Task Force

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Targeting oxidative stress improves disease outcomes in a rat model of acquired epilepsy.

http://www.wikidata.org/entity/Q38748531

description

2017 nî lūn-bûn

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name

Targeting oxidative stress improves disease outcomes in a rat model of acquired epilepsy.

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Targeting oxidative stress improves disease outcomes in a rat model of acquired epilepsy.

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Targeting oxidative stress improves disease outcomes in a rat model of acquired epilepsy.

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Targeting oxidative stress improves disease outcomes in a rat model of acquired epilepsy

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Alberto Pauletti

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Alessia Salamone

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Andrey Y Abramov

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Bianca R Villa

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Daniel J Antoine

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Eleonora Aronica

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Ennio Del Giudice

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Erwin A van Vliet

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Gaetano Terrone

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Li-Ping Liang

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P2860

Regulation of HMGB1 release by inflammasomes

Mitochondrial involvement and oxidative stress in temporal lobe epilepsy

Temporal and spatial increase of reactive nitrogen species in the kainate model of temporal lobe epilepsy.

Seizure activity results in calcium- and mitochondria-independent ROS production via NADPH and xanthine oxidase activation

International consensus classification of hippocampal sclerosis in temporal lobe epilepsy: a Task Force report from the ILAE Commission on Diagnostic Methods

IL-1 receptor/Toll-like receptor signaling in infection, inflammation, stress and neurodegeneration couples hyperexcitability and seizures

Metabolic dysfunction during neuronal activation in the ex vivo hippocampus from chronic epileptic rats and humans.

N-acetylcysteine, reactive oxygen species and beyond.

Molecular forms of HMGB1 and keratin-18 as mechanistic biomarkers for mode of cell death and prognosis during clinical acetaminophen hepatotoxicity

Sulforaphane: translational research from laboratory bench to clinic.

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Matthew C. Walker

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