Dextran sulfate triggers platelet aggregation via direct activation of PEAR1.

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Cell-Specific PEAR1 Methylation Studies Reveal a Locus that Coordinates Expression of Multiple Genes.Variants of PEAR1 Are Associated With Outcome in Patients With ACS and Stable CAD Undergoing PCI.

P2860

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Dextran sulfate triggers platelet aggregation via direct activation of PEAR1.

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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Dextran sulfate triggers platelet aggregation via direct activation of PEAR1.

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type

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label

Dextran sulfate triggers platelet aggregation via direct activation of PEAR1.

@en

prefLabel

Dextran sulfate triggers platelet aggregation via direct activation of PEAR1.

@en

P1476

Dextran sulfate triggers platelet aggregation via direct activation of PEAR1

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P2093

Christophe Vandenbriele

http://www.w3.org/2001/XMLSchema#string

Katrien Cludts

http://www.w3.org/2001/XMLSchema#string

Maarten Criel

http://www.w3.org/2001/XMLSchema#string

Marc F Hoylaerts

http://www.w3.org/2001/XMLSchema#string

Peter Verhamme

http://www.w3.org/2001/XMLSchema#string

Soetkin Van Kerckhoven

http://www.w3.org/2001/XMLSchema#string

Thomas Vanassche

http://www.w3.org/2001/XMLSchema#string

Yi Sun

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C-type lectin like receptor 2 (CLEC-2) signals independently of lipid raft microdomains in platelets.

Distinct pathways regulate Syk protein activation downstream of immune tyrosine activation motif (ITAM) and hemITAM receptors in platelets.

High glucose-induced NF-kappaB activation occurs via tyrosine phosphorylation of IkappaBalpha in human glomerular endothelial cells: involvement of Syk tyrosine kinase.

Platelet endothelial aggregation receptor 1 (PEAR1), a novel epidermal growth factor repeat-containing transmembrane receptor, participates in platelet contact-induced activation.

Activation of glycoprotein VI (GPVI) and C-type lectin-like receptor-2 (CLEC-2) underlies platelet activation by diesel exhaust particles and other charged/hydrophobic ligands.

A Human Platelet Receptor Protein Microarray Identifies the High Affinity Immunoglobulin E Receptor Subunit α (FcεR1α) as an Activating Platelet Endothelium Aggregation Receptor 1 (PEAR1) Ligand.

The biological behaviour of a new synthetic anticoagulant (dextran sulphate) possessing heparin-like properties.

The orally available spleen tyrosine kinase inhibitor 2-[7-(3,4-dimethoxyphenyl)-imidazo[1,2-c]pyrimidin-5-ylamino]nicotinamide dihydrochloride (BAY 61-3606) blocks antigen-induced airway inflammation in rodents.

PEAR1 attenuates megakaryopoiesis via control of the PI3K/PTEN pathway.

Dextran sulphate induces fibrinogen receptor activation through a novel Syk-independent PI-3 kinase-mediated tyrosine kinase pathway in platelets.

P304

365-372

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P31

scholarly article

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10.3109/09537104.2015.1111321

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Benedetta Izzi

P577

2015-11-30T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

26619766

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Dextran sulfate triggers platelet aggregation via direct activation of PEAR1.

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P2860

P2860

Dextran sulfate triggers platelet aggregation via direct activation of PEAR1.

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P1433

P1476

P2093

P2860

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P2860

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P698