Targeting cell death signalling in cancer: minimising 'Collateral damage'. - Wikidata - wikimedia - TriplyDB

Targeting cell death signalling in cancer: minimising 'Collateral damage'.

Targeting cell death signalling in cancer: minimising 'Collateral damage'.

http://www.wikidata.org/entity/Q38824157

about

Cancer's Achilles' Heel: Apoptosis and Necroptosis to the Rescue The Role of Oxidative Stress in Koenimbine-Induced DNA Damage and Heat Shock Protein Modulation in HepG2 Cells.Pharmacological modulation of cell death in organ transplantation.Natural Agents-Mediated Targeting of Histone Deacetylases.Novel anti-cancer drug COTI-2 synergizes with therapeutic agents and does not induce resistance or exhibit cross-resistance in human cancer cell lines.Negative regulators of cell death pathways in cancer: perspective on biomarkers and targeted therapies.Bcl-2 family in non-small cell lung cancer: its prognostic and therapeutic implications.Unlocking the NF-κB Conundrum: Embracing Complexity to Achieve Specificity.Differential mRNA expression of the main apoptotic proteins in normal and malignant cells and its relation to in vitro resistance.Essential Oil Derived From Eupatorium adenophorum Spreng. Mediates Anticancer Effect by Inhibiting STAT3 and AKT Activation to Induce Apoptosis in Hepatocellular Carcinoma.

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P2860

Targeting cell death signalling in cancer: minimising 'Collateral damage'.

http://www.wikidata.org/entity/Q38824157

description

2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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2016年论文

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2016年论文

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name

Targeting cell death signalling in cancer: minimising 'Collateral damage'.

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type

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Targeting cell death signalling in cancer: minimising 'Collateral damage'.

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Targeting cell death signalling in cancer: minimising 'Collateral damage'.

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British Journal of Cancer

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Targeting cell death signalling in cancer: minimising 'Collateral damage'

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Marion MacFarlane

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Creative Commons Attribution 4.0 International

P2860

Hallmarks of Cancer: The Next Generation

IAP antagonists induce autoubiquitination of c-IAPs, NF-kappaB activation, and TNFalpha-dependent apoptosis

IAP antagonists target cIAP1 to induce TNFalpha-dependent apoptosis

Cancer cell-autonomous TRAIL-R signaling promotes KRAS-driven cancer progression, invasion, and metastasis

Small-Molecule Bcl2 BH4 Antagonist for Lung Cancer Therapy

Autocrine TNFalpha signaling renders human cancer cells susceptible to Smac-mimetic-induced apoptosis

On respiratory impairment in cancer cells

A set of NF-κB-regulated microRNAs induces acquired TRAIL resistance in lung cancer.

A death effector domain chain DISC model reveals a crucial role for caspase-8 chain assembly in mediating apoptotic cell death.

TRAIL treatment provokes mutations in surviving cells.

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