Tubular deficiency of von Hippel-Lindau attenuates renal disease progression in anti-GBM glomerulonephritis. - Wikidata - wikimedia - TriplyDB

Tubular deficiency of von Hippel-Lindau attenuates renal disease progression in anti-GBM glomerulonephritis.

Tubular deficiency of von Hippel-Lindau attenuates renal disease progression in anti-GBM glomerulonephritis.

http://www.wikidata.org/entity/Q38867061

about

Hypoxia and fibrosis in chronic kidney disease: crossing at pericytes Renal tubular HIF-2α expression requires VHL inactivation and causes fibrosis and cysts Deletion of von Hippel-Lindau protein converts renin-producing cells into erythropoietin-producing cells.Tubulovascular cross-talk by vascular endothelial growth factor a maintains peritubular microvasculature in kidney.Central role for endothelial human deneddylase-1/SENP8 in fine-tuning the vascular inflammatory response.Impaired hypoxia-inducible factor (HIF) regulation by hyperglycemia.How tubular epithelial cells dictate the rate of renal fibrogenesis?Hypoxia and Dysregulated Angiogenesis in Kidney Disease.Inhalation treatment of pulmonary fibrosis by liposomal prostaglandin E2.Inflammatory cells of immunosuppressive phenotypes in oral lichen planus have a proinflammatory pattern of expression and are associated with clinical parameters.Action of hypoxia-inducible factor in liver and kidney from mice with Pax8-rtTA-based deletion of von Hippel-Lindau protein.

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P2860

Tubular deficiency of von Hippel-Lindau attenuates renal disease progression in anti-GBM glomerulonephritis.

http://www.wikidata.org/entity/Q38867061

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Tubular deficiency of von Hipp ...... n anti-GBM glomerulonephritis.

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Tubular deficiency of von Hipp ...... n anti-GBM glomerulonephritis.

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Tubular deficiency of von Hipp ...... n anti-GBM glomerulonephritis.

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J.AJPATH.2011.07.012

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The American Journal of Pathology

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Tubular deficiency of von Hipp ...... n anti-GBM glomerulonephritis.

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P2093

Anne Kathrin Enke

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Brigitte Scolari

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Danny Polzin

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Franziska Theilig

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Robert Koesters

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Sebastian Bachmann

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P2860

Hypoxia promotes fibrogenesis in vivo via HIF-1 stimulation of epithelial-to-mesenchymal transition

Differential roles of hypoxia-inducible factor 1alpha (HIF-1alpha) and HIF-2alpha in hypoxic gene regulation

Analysis of glomerular VEGF mRNA and protein expression in murine mesangioproliferative glomerulonephritis.

Fate tracing reveals the pericyte and not epithelial origin of myofibroblasts in kidney fibrosis

Vascular tumors in livers with targeted inactivation of the von Hippel-Lindau tumor suppressor.

Von Hippel-Lindau disease: clinical and molecular perspectives.

Increased expression of vascular endothelial growth factor in kidney leads to progressive impairment of glomerular functions.

Glomerular-specific alterations of VEGF-A expression lead to distinct congenital and acquired renal diseases.

Pathways to nephron loss starting from glomerular diseases-insights from animal models.

An efficient and versatile system for acute and chronic modulation of renal tubular function in transgenic mice.

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2177-2188

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10.1016/J.AJPATH.2011.07.012

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21925138

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glomerulonephritis

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3204192

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