Early-life exposure to combustion-derived particulate matter causes pulmonary immunosuppression.
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Addressing Emerging Risks: Scientific and Regulatory Challenges Associated with Environmentally Persistent Free RadicalsThe Role of Plant-Microbe Interactions and Their Exploitation for Phytoremediation of Air Pollutants.Catechol degradation on hematite/silica-gas interface as affected by gas composition and the formation of environmentally persistent free radicalsUpdate in environmental and occupational lung diseases 2013Exposure to combustion generated environmentally persistent free radicals enhances severity of influenza virus infectionImpaired gamma delta T cell-derived IL-17A and inflammasome activation during early respiratory syncytial virus infection in infantsInhibition of cytochrome P450 2B4 by environmentally persistent free radical-containing particulate matter.Respiratory Syncytial Virus Disease Is Mediated by Age-Variable IL-33.Exposure to Deepwater Horizon Crude Oil Burnoff Particulate Matter Induces Pulmonary Inflammation and Alters Adaptive Immune ResponseExposure to allergen and diesel exhaust particles potentiates secondary allergen-specific memory responses, promoting asthma susceptibility.Early Postnatal Exposure to Cigarette Smoke Leads to Later Airway Inflammation in Asthmatic Mice.Air pollution and allergic diseases.Exposure to environmentally persistent free radicals during gestation lowers energy expenditure and impairs skeletal muscle mitochondrial function in adult mice.Regulatory T cells and IL10 suppress pulmonary host defense during early-life exposure to radical containing combustion derived ultrafine particulate matter.Predictive and Prognostic Biomarkers of Respiratory Diseases due to Particulate Matter Exposure.A Scalable Field Study Protocol and Rationale for Passive Ambient Air Sampling: A Spatial Phytosampling for Leaf Data Collection.Effects of urban coarse particles inhalation on oxidative and inflammatory parameters in the mouse lung and colon.Early-life exposure to three size-fractionated ultrafine and fine atmospheric particulates in Beijing exacerbates asthma development in mature mice.Ambient particulate matter activates the aryl hydrocarbon receptor in dendritic cells and enhances Th17 polarization
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Early-life exposure to combustion-derived particulate matter causes pulmonary immunosuppression.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Early-life exposure to combust ...... s pulmonary immunosuppression.
@en
type
label
Early-life exposure to combust ...... s pulmonary immunosuppression.
@en
prefLabel
Early-life exposure to combust ...... s pulmonary immunosuppression.
@en
P2093
P2860
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Early-life exposure to combust ...... es pulmonary immunosuppression
@en
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P2860
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P304
P356
10.1038/MI.2013.88
P577
2013-10-30T00:00:00Z
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P6179
1042846964