Mucin production during prenatal and postnatal murine lung development.
about
Galactose 6-O-sulfotransferases are not required for the generation of Siglec-F ligands in leukocytes or lung tissueRole of epithelial mucins during airway infectionCell-based therapy in lung regenerative medicineMuc5b is required for airway defenceIn vivo imaging of the Muc5b gel-forming mucinLung epithelial stem cells and their niches: Fgf10 takes center stage.Neonatal mucosal immunology.The polymeric mucin Muc5ac is required for allergic airway hyperreactivity.Notch signaling prevents mucous metaplasia in mouse conducting airways during postnatal development.Baseline Goblet Cell Mucin Secretion in the Airways Exceeds Stimulated Secretion over Extended Time Periods, and Is Sensitive to Shear Stress and Intracellular Mucin Stores.Airway epithelial SPDEF integrates goblet cell differentiation and pulmonary Th2 inflammation.Mucus clearance, MyD88-dependent and MyD88-independent immunity modulate lung susceptibility to spontaneous bacterial infection and inflammationThe lung response to ozone is determined by age and is partially dependent on toll-Like receptor 4.CFTR, mucins, and mucus obstruction in cystic fibrosis.Overexpressing mouse model demonstrates the protective role of Muc5ac in the lungs.In utero exposure to arsenic alters lung development and genes related to immune and mucociliary function in mice.Coordinate regulation of the gel-forming mucin genes at chromosome 11p15.5.Control of lung defence by mucins and macrophages: ancient defence mechanisms with modern functions.Transcriptome profiling of the newborn mouse lung response to acute ozone exposure.Contribution of mucus concentration and secreted mucins Muc5ac and Muc5b to the pathogenesis of muco-obstructive lung diseaseCellular and molecular biology of airway mucins.Stem cells of the adult lung: their development and role in homeostasis, regeneration, and disease.MUC5AC interactions with integrin β4 enhances the migration of lung cancer cells through FAK signaling.Idiopathic Pulmonary Fibrosis: A Genetic Disease That Involves Mucociliary Dysfunction of the Peripheral Airways.Early-life exposure to combustion-derived particulate matter causes pulmonary immunosuppression.Younger patients with COME are more likely to have mucoid middle ear fluid containing mucin MUC5B.Birth delivery method affects expression of immune genes in lung and jejunum tissue of neonatal beef calves.
P2860
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P2860
Mucin production during prenatal and postnatal murine lung development.
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Mucin production during prenatal and postnatal murine lung development.
@ast
Mucin production during prenatal and postnatal murine lung development.
@en
type
label
Mucin production during prenatal and postnatal murine lung development.
@ast
Mucin production during prenatal and postnatal murine lung development.
@en
prefLabel
Mucin production during prenatal and postnatal murine lung development.
@ast
Mucin production during prenatal and postnatal murine lung development.
@en
P2093
P2860
P356
P1476
Mucin production during prenatal and postnatal murine lung development.
@en
P2093
Camille Ehre
Christopher M Evans
Jesus R Hernandez
Mahdis Rahmani
Michelle G Roy
Samantha N Alexander
Samuel B Ho
P2860
P304
P356
10.1165/RCMB.2010-0020RC
P577
2011-06-01T00:00:00Z