To "grow" or "go": TMEM16A expression as a switch between tumor growth and metastasis in SCCHN.
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Ani9, A Novel Potent Small-Molecule ANO1 Inhibitor with Negligible Effect on ANO2Modulating Ca²⁺ signals: a common theme for TMEM16, Ist2, and TMC.TMEM16A overexpression contributes to tumor invasion and poor prognosis of human gastric cancer through TGF-β signaling.Identification of the Novel TMEM16A Inhibitor Dehydroandrographolide and Its Anticancer Activity on SW620 CellsEffects of new-generation inhibitors of the calcium-activated chloride channel anoctamin 1 on slow waves in the gastrointestinal tract.Meta-Analyses of Microarray Datasets Identifies ANO1 and FADD as Prognostic Markers of Head and Neck CancerANO1 plays a critical role in prostatic hyperplasia.Clinical significance of Anoctamin-1 gene at 11q13 in the development and progression of head and neck squamous cell carcinomas.TMEM16A/ANO1 is differentially expressed in HPV-negative versus HPV-positive head and neck squamous cell carcinoma through promoter methylationEnhanced cell migration and apoptosis resistance may underlie the association between high SERPINE1 expression and poor outcome in head and neck carcinoma patients.Single-cell triple omics sequencing reveals genetic, epigenetic, and transcriptomic heterogeneity in hepatocellular carcinomas.CRISPR-Mediated VHL Knockout Generates an Improved Model for Metastatic Renal Cell Carcinoma.TMEM16 proteins: unknown structure and confusing functions.Nrf2-Keap1 pathway promotes cell proliferation and diminishes ferroptosisRole of volume-regulated and calcium-activated anion channels in cell volume homeostasis, cancer and drug resistanceTMEM16A/ANO1 suppression improves response to antibody-mediated targeted therapy of EGFR and HER2/ERBB2.Inhibition of calcium-activated chloride channel ANO1 suppresses proliferation and induces apoptosis of epithelium originated cancer cells.The Mechanistic Role of the Calcium-Activated Chloride Channel ANO1 in Tumor Growth and Signaling.Ion channels in regulated cell death.ANO1/TMEM16A interacts with EGFR and correlates with sensitivity to EGFR-targeting therapy in head and neck cancerCopy number gain of 11q13.3 genes associates with pathological stage in hypopharyngeal squamous cell carcinoma.Cell-specific mechanisms of TMEM16A Ca2+-activated chloride channel in cancer.CLCA1 suppresses colorectal cancer aggressiveness via inhibition of the Wnt/beta-catenin signaling pathway.DNA methylation regulates TMEM16A/ANO1 expression through multiple CpG islands in head and neck squamous cell carcinoma.Cell-specific regulation of proliferation by Ano1/TMEM16A in breast cancer with different ER, PR, and HER2 status.TMEM16A/ANO1 Inhibits Apoptosis Via Downregulation of Bim Expression.Expression of ANO1/DOG1 is associated with shorter survival and progression of breast carcinomas.TMEM16A regulates portal vein smooth muscle cell proliferation in portal hypertension.CLCA2 is a positive regulator of store-operated calcium entry and TMEM16A.Tumors of the upper aerodigestive tract
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P2860
To "grow" or "go": TMEM16A expression as a switch between tumor growth and metastasis in SCCHN.
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2014 nî lūn-bûn
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2014年の論文
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name
To "grow" or "go": TMEM16A exp ...... rowth and metastasis in SCCHN.
@en
type
label
To "grow" or "go": TMEM16A exp ...... rowth and metastasis in SCCHN.
@en
prefLabel
To "grow" or "go": TMEM16A exp ...... rowth and metastasis in SCCHN.
@en
P2093
P2860
P50
P1476
To "grow" or "go": TMEM16A exp ...... growth and metastasis in SCCHN
@en
P2093
Carol A Bertrand
Chunbo Shao
Jeffery N Myers
Jennifer Grandis
L Alex Gaither
Patrick Ha
Raja S Seethala
P2860
P304
P356
10.1158/1078-0432.CCR-14-0363
P407
P577
2014-06-11T00:00:00Z