RBM5, 6, and 10 differentially regulate NUMB alternative splicing to control cancer cell proliferation.
about
Differential Impacts of Alternative Splicing Networks on ApoptosisThe role of splicing factors in deregulation of alternative splicing during oncogenesis and tumor progressionContext-dependent control of alternative splicing by RNA-binding proteinsLarge-scale analysis of genome and transcriptome alterations in multiple tumors unveils novel cancer-relevant splicing networksRBM10 promotes transformation-associated processes in small cell lung cancer and is directly regulated by RBM5.An RRM-ZnF RNA recognition module targets RBM10 to exonic sequences to promote exon exclusionThe splicing factor RBM4 controls apoptosis, proliferation, and migration to suppress tumor progression.The tumor suppressive role of NUMB isoform 1 in esophageal squamous cell carcinoma.Binding site discovery from nucleic acid sequences by discriminative learning of hidden Markov models.The clathrin adaptor proteins ARH, Dab2, and numb play distinct roles in Niemann-Pick C1-Like 1 versus low density lipoprotein receptor-mediated cholesterol uptake.Proteomic profile and in silico analysis in metastatic melanoma with and without BRAF mutationAlternative RNA structure-coupled gene regulations in tumorigenesis.Ott1 (Rbm15) regulates thrombopoietin response in hematopoietic stem cells through alternative splicing of c-MplInsight into the role of alternative splicing within the RBM10v1 exon 10 tandem donor site.Role of six single nucleotide polymorphisms, risk factors in coronary disease, in OLR1 alternative splicingLeveraging transcript quantification for fast computation of alternative splicing profiles.Transcriptome-wide identification and study of cancer-specific splicing events across multiple tumors.Alternative signaling network activation through different insulin receptor family members caused by pro-mitogenic antidiabetic insulin analogues in human mammary epithelial cellsSrc Family Tyrosine Kinase Signaling Regulates FilGAP through Association with RBM10.Alternative splicing of the cell fate determinant Numb in hepatocellular carcinoma.Global profiling of alternative RNA splicing events provides insights into molecular differences between various types of hepatocellular carcinomaIdentification of a Novel Human E-Cadherin Splice Variant and Assessment of Its Effects Upon EMT-Related Events.Transcriptome-wide analysis of alternative RNA splicing events in Epstein-Barr virus-associated gastric carcinomasThe pathogenicity of splicing defects: mechanistic insights into pre-mRNA processing inform novel therapeutic approaches.Nuclear Magnetic Resonance Structure of a Novel Globular Domain in RBM10 Containing OCRE, the Octamer Repeat Sequence MotifRBM4 promotes neuronal differentiation and neurite outgrowth by modulating Numb isoform expressionA splicing isoform of TEAD4 attenuates the Hippo-YAP signalling to inhibit tumour proliferationThe exon junction complex regulates the splicing of cell polarity gene dlg1 to control Wingless signaling in development.Genome-wide analysis of alternative splicing during human heart development.RNA splicing factors as oncoproteins and tumour suppressors.RBM5 reduces small cell lung cancer growth, increases cisplatin sensitivity and regulates key transformation-associated pathways.Tumor-suppressor genes that escape from X-inactivation contribute to cancer sex bias.RBM5 inhibits tumorigenesis of gliomas through inhibition of Wnt/β-catenin signaling and induction of apoptosis.Functional analysis reveals that RBM10 mutations contribute to lung adenocarcinoma pathogenesis by deregulating splicingThe RNA-binding landscape of RBM10 and its role in alternative splicing regulation in models of mouse early development.LOX-1 and Its Splice Variants: A New Challenge for Atherosclerosis and Cancer-Targeted Therapies.Expression of RNA-binding motif 10 is associated with advanced tumor stage and malignant behaviors of lung adenocarcinoma cancer cells.Functional roles of alternative splicing factors in human disease.Splicing arrays reveal novel RBM10 targets, including SMN2 pre-mRNA.Decreased RNA-binding motif 5 expression is associated with tumor progression in gastric cancer.
P2860
Q28078238-C2C77977-6FC2-46DC-98D8-1D7DA6B22382Q28083838-BB406EDE-57CD-4564-9368-B64EE520C52DQ28245737-53EA4C6F-A16A-478B-B8E1-D22D8F159C48Q28275692-A0DD81D7-51B8-4676-945E-D97139F813A7Q33853533-43674D4A-1D76-47B7-9178-0B52C74EBC04Q33878647-84CE39AD-FE49-4834-ACCC-C768C8EE9BF7Q34157516-E1E96179-1D6A-4934-99C1-96DB054AF43CQ34221736-CDD8D633-1294-4278-BDB0-3A87581EBEC8Q34579733-03314DED-036D-4117-AE20-1D31172A4998Q34580436-698505B2-57D4-4DBF-A6F5-83D6B7FAE8B6Q34599527-742D9D75-02BE-469E-A990-5D72F0088BD7Q35016456-997F9A5E-F4FD-4327-96E4-F2D6547222E2Q35056295-A15FEEE4-8C04-4FEF-8063-DDBA2C524494Q35107709-7E968262-6353-49FA-8DE7-371BC00A6DC6Q35621461-53DCA850-1BA6-4B36-88F5-C77BFC722463Q35693081-78B0E1B9-F40C-4A2D-9057-4A22173F25D2Q35740083-1D5769A7-221F-40CE-90B0-9BAA9BD1EBD5Q35863765-BF61DFDA-E129-470D-8008-D3A188949BBFQ35890184-2C8F8D16-499D-45CD-A17E-7C81752919FEQ36108838-9AD3B875-B3DF-4808-A1FF-351308159778Q36114523-88B4E974-276D-4456-A64E-12EDC7C66AECQ36147466-6290C83B-4236-456F-B0FC-B015B15FCDA9Q36368952-22FD6C5C-688A-408D-BD19-EF3D4DE5283EQ36410173-49B87F77-05AF-4B49-B7DC-D1FCF83587C9Q36446661-F2A8EB35-C8D9-4090-B010-496B63EA70C2Q36896328-05DE87DD-FFFC-45F7-A132-A4D97BEA5B2DQ37010219-FDEDC57D-24F5-402B-8C0F-AD85E62273EFQ37226999-B8C6237D-AAD5-4ED1-9F3B-DD98EA5B3B39Q37345542-368AD302-7584-4138-BC30-367BA02ECA26Q37391660-10FE8E83-FEBA-4246-AE9D-D4D7F958F36AQ37465032-60644DD7-C375-44FE-8E6D-6ACEFAFFCE33Q37550949-23CA760F-62BC-43B0-BECE-F2FCD9B49D28Q37569674-8FA188ED-5766-424B-B486-7F252AF85F2DQ37587671-B818CDF8-0516-46A3-879D-9C763FB508CFQ37610037-DA3DBF8F-45C9-401E-B23C-4FB20A997A68Q37690728-E1B6F9E6-03CD-4A07-B25D-7CFF1CD91B9DQ38288535-7FEEC62B-D7A2-493A-9868-F7FDDF41E2FFQ38335759-C5FB16D1-6F47-44BF-A406-A1F6803BC5BDQ38429727-5C4484B7-F001-45D9-A9AA-272869F14058Q38710241-07C452F6-730E-4C9B-B202-8A8B1DBBFAAA
P2860
RBM5, 6, and 10 differentially regulate NUMB alternative splicing to control cancer cell proliferation.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
RBM5, 6, and 10 differentially ...... rol cancer cell proliferation.
@en
RBM5, 6, and 10 differentially ...... rol cancer cell proliferation.
@nl
type
label
RBM5, 6, and 10 differentially ...... rol cancer cell proliferation.
@en
RBM5, 6, and 10 differentially ...... rol cancer cell proliferation.
@nl
prefLabel
RBM5, 6, and 10 differentially ...... rol cancer cell proliferation.
@en
RBM5, 6, and 10 differentially ...... rol cancer cell proliferation.
@nl
P50
P1433
P1476
RBM5, 6, and 10 differentially ...... rol cancer cell proliferation.
@en
P2093
Elias G Bechara
Isabella Bernardis
P304
P356
10.1016/J.MOLCEL.2013.11.010
P577
2013-12-01T00:00:00Z