Cell toxicity of methacrylate monomers-the role of glutathione adduct formation.

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HEMA-induced cytotoxicity: oxidative stress, genotoxicity and apoptosis.Cell toxicity of 2-hydroxyethyl methacrylate (HEMA): the role of oxidative stress.Evaluation of Resins for Stereolithographic 3D-Printed Surgical Guides: The Response of L929 Cells and Human Gingival Fibroblasts.

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P2860

Cell toxicity of methacrylate monomers-the role of glutathione adduct formation.

Item

http://www.wikidata.org/entity/Q39161574

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2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

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2013年論文

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2013年论文

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2013年论文

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2013年论文

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name

Cell toxicity of methacrylate monomers-the role of glutathione adduct formation.

@en

Cell toxicity of methacrylate monomers-the role of glutathione adduct formation.

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type

Item

label

Cell toxicity of methacrylate monomers-the role of glutathione adduct formation.

@en

Cell toxicity of methacrylate monomers-the role of glutathione adduct formation.

@nl

prefLabel

Cell toxicity of methacrylate monomers-the role of glutathione adduct formation.

@en

Cell toxicity of methacrylate monomers-the role of glutathione adduct formation.

@nl

P1476

Cell toxicity of methacrylate monomers-the role of glutathione adduct formation.

@en

P2093

E Morisbak

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H B Kopperud

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J T Samuelsen

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V Ansteinsson

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P2860

Rapid colorimetric assay for cellular growth and survival: Application to proliferation and cytotoxicity assays

Relation of dental composite formulations to their degradation and the release of hydrolyzed polymeric-resin-derived products.

Genetic and cellular toxicology of dental resin monomers.

Independent and combined cytotoxicity and genotoxicity of triethylene glycol dimethacrylate and urethane dimethacrylate.

Bisphenol A-glycidyl methacrylate induces a broad spectrum of DNA damage in human lymphocytes.

Dental methacrylates may exert genotoxic effects via the oxidative induction of DNA double strand breaks and the inhibition of their repair.

DNA-damage, cell-cycle arrest and apoptosis induced in BEAS-2B cells by 2-hydroxyethyl methacrylate (HEMA).

Influence of TEGDMA on the mammalian cell cycle in comparison with chemotherapeutic agents.

Inhibition of TEGDMA and HEMA-induced genotoxicity and cell cycle arrest by N-acetylcysteine.

Apoptosis induced by the monomers HEMA and TEGDMA involves formation of ROS and differential activation of the MAP-kinases p38, JNK and ERK.

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3504-3510

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scholarly article

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10.1002/JBM.A.34652

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101

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2013-04-24T00:00:00Z

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236277035

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23613115

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Cell toxicity of methacrylate monomers-the role of glutathione adduct formation.

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P2860

P2860

Cell toxicity of methacrylate monomers-the role of glutathione adduct formation.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P577

P5875

P698

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P577

P5875

P698