Inhibition of TEGDMA and HEMA-induced genotoxicity and cell cycle arrest by N-acetylcysteine.
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N-Acetyl Cysteine Depletes Reactive Oxygen Species and Prevents Dental Monomer-Induced Intrinsic Mitochondrial Apoptosis In Vitro in Human Dental Pulp CellsEpigallocatechin-3-Gallate Reduces Cytotoxic Effects Caused by Dental Monomers: A Hypothesis.Methacryloxylethyl Cetyl Ammonium Chloride Induces DNA Damage and Apoptosis in Human Dental Pulp Cells via Generation of Oxidative Stress.N-acetyl cysteine mediates protection from 2-hydroxyethyl methacrylate induced apoptosis via nuclear factor kappa B-dependent and independent pathways: potential involvement of JNK.Molecular toxicology of substances released from resin-based dental restorative materials.Toll-like receptors, LPS, and dental monomers.HEMA-induced cytotoxicity: oxidative stress, genotoxicity and apoptosis.Gene expression change in human dental pulp cells exposed to a low-level toxic concentration of triethylene glycol dimethacrylate: an RNA-seq analysis.Bisphenol A-glycidyl methacrylate induces a broad spectrum of DNA damage in human lymphocytes.Cell toxicity of 2-hydroxyethyl methacrylate (HEMA): the role of oxidative stress.Cell toxicity of methacrylate monomers-the role of glutathione adduct formation.Dental methacrylates may exert genotoxic effects via the oxidative induction of DNA double strand breaks and the inhibition of their repair.Human and bovine pulp-derived cell reactions to dental resin cements.Glutathione level and genotoxicity in human oral keratinocytes exposed to TEGDMA.2-hydroxylethyl methacrylate (HEMA), a tooth restoration component, exerts its genotoxic effects in human gingival fibroblasts trough methacrylic acid, an immediate product of its degradation.The influences of N-acetyl cysteine (NAC) on the cytotoxicity and mechanical properties of Poly-methylmethacrylate (PMMA)-based dental resin.HEMA but not TEGDMA induces autophagy in human gingival fibroblasts.Insights into and relative effect of chitosan-H, chitosan-H-propolis, chitosan-H-propolis-nystatin and chitosan-H-nystatin on dentine bond strength.N-acetylcysteine protects dental pulp stromal cells from HEMA-induced apoptosis by inducing differentiation of the cells.Effects of current provisional restoration materials on the viability of fibroblasts.Cytotoxicity Evaluation of Self Adhesive Composite Resin Cements by Dentin Barrier Test on 3D Pulp Cells.Involvement of mitochondrial signalling pathway in HGFs/S. mitis coculture response to TEGDMA treatment.N-acetyl cysteine (NAC)-assisted detoxification of PMMA resin.Genotoxicity and Mutagenicity Testing of Biomaterials
P2860
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P2860
Inhibition of TEGDMA and HEMA-induced genotoxicity and cell cycle arrest by N-acetylcysteine.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Inhibition of TEGDMA and HEMA- ...... le arrest by N-acetylcysteine.
@en
type
label
Inhibition of TEGDMA and HEMA- ...... le arrest by N-acetylcysteine.
@en
prefLabel
Inhibition of TEGDMA and HEMA- ...... le arrest by N-acetylcysteine.
@en
P2093
P1433
P1476
Inhibition of TEGDMA and HEMA- ...... le arrest by N-acetylcysteine.
@en
P2093
P304
P356
10.1016/J.DENTAL.2006.06.021
P407
P577
2006-08-07T00:00:00Z