about
Regulation and function of AMPK in physiology and diseasesPast strategies and future directions for identifying AMP-activated protein kinase (AMPK) modulatorsCombined inhibition of glycolysis and AMPK induces synergistic breast cancer cell killing.Dissecting the Dual Role of AMPK in Cancer: From Experimental to Human StudiesCOH-SR4 reduces body weight, improves glycemic control and prevents hepatic steatosis in high fat diet-induced obese miceAMPK activity regulates trafficking of mitochondria to the leading edge during cell migration and matrix invasion.NDRG2 overexpression enhances glucose deprivation-mediated apoptosis in breast cancer cells via inhibition of the LKB1-AMPK pathway.The study of inducing apoptosis effect of fructose 1,6-bisphosphate on the papillary thyroid carcinoma cell and its related mechanism.Small molecule adenosine 5'-monophosphate activated protein kinase (AMPK) modulators and human diseasesGlucose-based regulation of miR-451/AMPK signaling depends on the OCT1 transcription factor.Is 5´-AMP-Activated Protein Kinase Both Jekyll and Hyde in Bladder Cancer?AMPK Promotes Aberrant PGC1β Expression To Support Human Colon Tumor Cell Survival.The KSHV K1 Protein Modulates AMPK Function to Enhance Cell Survival.Targeting AMPK signaling in combating ovarian cancers: opportunities and challenges.Perspectives of the AMP-activated kinase (AMPK) signalling pathway in thyroid cancer.The matricellular protein CYR61 promotes breast cancer lung metastasis by facilitating tumor cell extravasation and suppressing anoikis.The pentose phosphate pathway and cancer.The double-edged sword of AMPK signaling in cancer and its therapeutic implications.Metformin in pancreatic cancer treatment: from clinical trials through basic research to biomarker quantification.Deficiency of AMPK in CD8+ T cells suppresses their anti-tumor function by inducing protein phosphatase-mediated cell death.A spatiotemporal hypothesis for the regulation, role, and targeting of AMPK in prostate cancer.Dysregulation of NRF2 in Cancer: from Molecular Mechanisms to Therapeutic Opportunities.A RAS-CaMKKβ-AMPKα2 pathway promotes senescence by licensing post-translational activation of C/EBPβ through a novel 3'UTR mechanism.How does oncogene transformation render tumor cells hypersensitive to nutrient deprivation?The critical role of AMPK in driving Akt activation under stress, tumorigenesis and drug resistance
P2860
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P2860
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
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2012年學術文章
@zh-hant
name
The dark face of AMPK as an essential tumor promoter.
@en
The dark face of AMPK as an essential tumor promoter.
@nl
type
label
The dark face of AMPK as an essential tumor promoter.
@en
The dark face of AMPK as an essential tumor promoter.
@nl
prefLabel
The dark face of AMPK as an essential tumor promoter.
@en
The dark face of AMPK as an essential tumor promoter.
@nl
P2860
P356
P1433
P1476
The dark face of AMPK as an essential tumor promoter.
@en
P2093
Nissim Hay
Sang-Min Jeon
P2860
P304
P356
10.4161/CL.22651
P577
2012-10-01T00:00:00Z