Interferon gamma/NADPH oxidase defense system in immunity and cancer.

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Metabolic characterization and pathway analysis of berberine protects against prostate cancer.Trial watch: Immune checkpoint blockers for cancer therapy.

P2860

Q42371497-BE38A63E-FF69-4AC1-ADE3-AA4C9403D13B Q47393365-F44920B7-B46E-4BBB-9848-7B811EB0FD4F

P2860

Interferon gamma/NADPH oxidase defense system in immunity and cancer.

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

@zh-hk

2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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name

Interferon gamma/NADPH oxidase defense system in immunity and cancer.

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Interferon gamma/NADPH oxidase defense system in immunity and cancer.

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Interferon gamma/NADPH oxidase defense system in immunity and cancer.

@en

Interferon gamma/NADPH oxidase defense system in immunity and cancer.

@nl

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Interferon gamma/NADPH oxidase defense system in immunity and cancer.

@en

Interferon gamma/NADPH oxidase defense system in immunity and cancer.

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P1476

Interferon gamma/NADPH oxidase defense system in immunity and cancer.

@en

P2093

Milan Reinis

http://www.w3.org/2001/XMLSchema#string

Pavla Vasicova

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Sona Hubackova

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Zdenek Hodny

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P2860

Interferons, immunity and cancer immunoediting

NADPH oxidase DUOX1 promotes long-term persistence of oxidative stress after an exposure to irradiation.

Inactivation of NADPH oxidases NOX4 and NOX5 protects human primary fibroblasts from ionizing radiation-induced DNA damage.

Regulation of Nox and Duox enzymatic activity and expression

IFNγ induces oxidative stress, DNA damage and tumor cell senescence via TGFβ/SMAD signaling-dependent induction of Nox4 and suppression of ANT2.

Decitabine induces delayed reactive oxygen species (ROS) accumulation in leukemia cells and induces the expression of ROS generating enzymes.

IL1- and TGFβ-Nox4 signaling, oxidative stress and DNA damage response are shared features of replicative, oncogene-induced, and drug-induced paracrine 'bystander senescence'.

ROS-generating oxidases Nox1 and Nox4 contribute to oncogenic Ras-induced premature senescence.

ROS-generating NADPH oxidase NOX4 is a critical mediator in oncogenic H-Ras-induced DNA damage and subsequent senescence.

T-helper-1-cell cytokines drive cancer into senescence.

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e1080416

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scholarly article

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10.1080/2162402X.2015.1080416

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http://www.w3.org/2001/XMLSchema#string

P478

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Jiří Bártek

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2015-09-01T00:00:00Z

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27057461

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P932

4801460

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Interferon gamma/NADPH oxidase defense system in immunity and cancer.

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P2860

P2860

Interferon gamma/NADPH oxidase defense system in immunity and cancer.

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P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P698

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P698

P932