EGFR inhibitor BIBU induces apoptosis and defective autophagy in glioma cells.

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Glial progenitors as targets for transformation in glioma.ATM-NFκB axis-driven TIGAR regulates sensitivity of glioma cells to radiomimetics in the presence of TNFα.Cotargeting EGFR and autophagy signaling: A novel therapeutic strategy for non-small-cell lung cancer.

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EGFR inhibitor BIBU induces apoptosis and defective autophagy in glioma cells.

Item

http://www.wikidata.org/entity/Q39321309

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2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

@zh-hans

2012年学术文章

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2012年学术文章

@zh-sg

2012年學術文章

@yue

2012年學術文章

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2012年學術文章

@zh-hant

name

EGFR inhibitor BIBU induces apoptosis and defective autophagy in glioma cells.

@en

EGFR inhibitor BIBU induces apoptosis and defective autophagy in glioma cells.

@nl

type

Item

label

EGFR inhibitor BIBU induces apoptosis and defective autophagy in glioma cells.

@en

EGFR inhibitor BIBU induces apoptosis and defective autophagy in glioma cells.

@nl

prefLabel

EGFR inhibitor BIBU induces apoptosis and defective autophagy in glioma cells.

@en

EGFR inhibitor BIBU induces apoptosis and defective autophagy in glioma cells.

@nl

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EGFR inhibitor BIBU induces apoptosis and defective autophagy in glioma cells.

@en

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Deobrat Dixit

http://www.w3.org/2001/XMLSchema#string

Ellora Sen

http://www.w3.org/2001/XMLSchema#string

Ruchi Ghildiyal

http://www.w3.org/2001/XMLSchema#string

P2860

Bicyclic triterpenoid Iripallidal induces apoptosis and inhibits Akt/mTOR pathway in glioma cells

p62/SQSTM1 binds directly to Atg8/LC3 to facilitate degradation of ubiquitinated protein aggregates by autophagy

Interleukin-6-type cytokine signalling through the gp130/Jak/STAT pathway

Inflammation and cancer

Mutations in the EGFR kinase domain mediate STAT3 activation via IL-6 production in human lung adenocarcinomas

Interaction of a receptor tyrosine kinase, EGF-R, with caveolins. Caveolin binding negatively regulates tyrosine and serine/threonine kinase activities

Antitumor activity of rapamycin in a Phase I trial for patients with recurrent PTEN-deficient glioblastoma

S9, a novel anticancer agent, exerts its anti-proliferative activity by interfering with both PI3K-Akt-mTOR signaling and microtubule cytoskeleton

EGFR tyrosine kinase inhibitors activate autophagy as a cytoprotective response in human lung cancer cells

Tyrosine-kinase inhibition results in EGFR clustering at focal adhesions and consequent exocytosis in uPAR down-regulated cells of head and neck cancers.

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970-982

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scholarly article

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10.1002/MC.21938

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2012-06-29T00:00:00Z

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228104968

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P698

22753156

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P921

apoptotic process

autophagy

EGFR inhibitor BIBU induces apoptosis and defective autophagy in glioma cells.

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P2860

P2860

EGFR inhibitor BIBU induces apoptosis and defective autophagy in glioma cells.

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P1433

P1476

P2093

P2860

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P31

P356

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P5875

P698

P921

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P1433

P1476

P2093

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P5875

P698

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