Semaphorin 3A elevates endothelial cell permeability through PP2A inactivation. - Wikidata - wikimedia - TriplyDB

Semaphorin 3A elevates endothelial cell permeability through PP2A inactivation.

Semaphorin 3A elevates endothelial cell permeability through PP2A inactivation.

http://www.wikidata.org/entity/Q39333626

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LB100, a small molecule inhibitor of PP2A with potent chemo- and radio-sensitizing potential PP2A regulatory subunit Bα controls endothelial contractility and vessel lumen integrity via regulation of HDAC7.Regulating Set-β's Subcellular Localization Toggles Its Function between Inhibiting and Promoting Axon Growth and Regeneration.Endothelial permeability and VE-cadherin: a wacky comradeship.Semaphorin3A elevates vascular permeability and contributes to cerebral ischemia-induced brain damage.Endothelial secreted factors suppress mitogen deprivation-induced autophagy and apoptosis in glioblastoma stem-like cells.The N-terminal Set-β Protein Isoform Induces Neuronal Death Luteolin Impacts on the DNA Damage Pathway in Oral Squamous Cell Carcinoma.Semaphorin-3C signals through Neuropilin-1 and PlexinD1 receptors to inhibit pathological angiogenesis.Desert Hedgehog/Patch2 Axis Contributes to Vascular Permeability and Angiogenesis in Glioblastoma.Emerging roles of Semaphorins in the regulation of epithelial and endothelial junctions Phosphatase regulation of intercellular junctions.Critical multiple angiogenic factors secreted by glioblastoma stem-like cells underline the need for combinatorial anti-angiogenic therapeutic strategies.Vascular permeability and drug delivery in cancers.Morphogens and blood-brain barrier function in health and disease.The C-terminus region of β-arrestin1 modulates VE-cadherin expression and endothelial cell permeability.Glioblastoma stem-like cells secrete the pro-angiogenic VEGF-A factor in extracellular vesicles.β-escin selectively targets the glioblastoma-initiating cell population and reduces cell viability.Protein phosphatase 2A activity is required for functional adherent junctions in endothelial cells.The E3 ubiquitin ligase MARCH3 controls the endothelial barrier.Extracellular vesicle-transported Semaphorin3A promotes vascular permeability in glioblastoma.Apelin, the Devil Inside Brain Tumors.[Tumor angiogenesis: when the Tree of Life turns bad].Isthmin is a novel vascular permeability inducer that functions through cell-surface GRP78-mediated Src activation.

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Semaphorin 3A elevates endothelial cell permeability through PP2A inactivation.

http://www.wikidata.org/entity/Q39333626

description

2012 nî lūn-bûn

@nan

2012年の論文

@ja

2012年学术文章

@wuu

2012年学术文章

@zh-cn

2012年学术文章

@zh-hans

2012年学术文章

@zh-my

2012年学术文章

@zh-sg

2012年學術文章

@yue

2012年學術文章

@zh

2012年學術文章

@zh-hant

name

Semaphorin 3A elevates endothelial cell permeability through PP2A inactivation.

@en

Semaphorin 3A elevates endothelial cell permeability through PP2A inactivation.

@nl

type

label

Semaphorin 3A elevates endothelial cell permeability through PP2A inactivation.

@en

Semaphorin 3A elevates endothelial cell permeability through PP2A inactivation.

@nl

prefLabel

Semaphorin 3A elevates endothelial cell permeability through PP2A inactivation.

@en

Semaphorin 3A elevates endothelial cell permeability through PP2A inactivation.

@nl

P1433

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P1433

Journal of Cell Science

P1476

Semaphorin 3A elevates endothelial cell permeability through PP2A inactivation.

@en

P2093

Cedric Auffray

http://www.w3.org/2001/XMLSchema#string

Eva-Maria Galan-Moya

http://www.w3.org/2001/XMLSchema#string

Garance Kettler

http://www.w3.org/2001/XMLSchema#string

Herve Chneiweiss

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Jagoda K Hebda

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Julie Dwyer

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Julie Gavard

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Nicolas Bidere

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Sonia Dubois

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P2860

Rac1-induced cell migration requires membrane recruitment of the nuclear oncogene SET

VE-PTP and VE-cadherin ectodomains interact to facilitate regulation of phosphorylation and cell contacts

Stabilizing the VE-cadherin-catenin complex blocks leukocyte extravasation and vascular permeability

Protein phosphatase 2A associates with and regulates atypical PKC and the epithelial tight junction complex

Semaphorin 3A suppresses tumor growth and metastasis in mice melanoma model

Identification of human brain tumour initiating cells

VEGF controls endothelial-cell permeability by promoting the beta-arrestin-dependent endocytosis of VE-cadherin

Targeted deficiency or cytosolic truncation of the VE-cadherin gene in mice impairs VEGF-mediated endothelial survival and angiogenesis

Class 3 semaphorins control vascular morphogenesis by inhibiting integrin function

Protein phosphatase 2A interacts with the Src kinase substrate p130(CAS)

P304

4137-4146

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scholarly article

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10.1242/JCS.108282

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P433

Pt 17

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125

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Armelle Le Guelte

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2012-06-08T00:00:00Z

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225290816

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22685328

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