DIAPH3 governs the cellular transition to the amoeboid tumour phenotype. - Wikidata - wikimedia - TriplyDB

DIAPH3 governs the cellular transition to the amoeboid tumour phenotype.

DIAPH3 governs the cellular transition to the amoeboid tumour phenotype.

http://www.wikidata.org/entity/Q39347119

about

Cell polarity signaling in the plasticity of cancer cell invasiveness Extracellular vesicles in cancer: exosomes, microvesicles and the emerging role of large oncosomes Small-molecule agonists of mammalian Diaphanous-related (mDia) formins reveal an effective glioblastoma anti-invasion strategy.Large oncosomes mediate intercellular transfer of functional microRNA.Large oncosomes contain distinct protein cargo and represent a separate functional class of tumor-derived extracellular vesicles.Exosomes from metastatic cancer cells transfer amoeboid phenotype to non-metastatic cells and increase endothelial permeability: their emerging role in tumor heterogeneity Dia-interacting protein (DIP) imposes migratory plasticity in mDia2-dependent tumor cells in three-dimensional matrices.An mDia2/ROCK signaling axis regulates invasive egress from epithelial ovarian cancer spheroids.Proteomic analyses uncover a new function and mode of action for mouse homolog of Diaphanous 2 (mDia2)Regulation of microtubule dynamics by DIAPH3 influences amoeboid tumor cell mechanics and sensitivity to taxanes An essential role of metalloprotease-disintegrin ADAM12 in triple-negative breast cancer Large oncosomes in human prostate cancer tissues and in the circulation of mice with metastatic disease.Genetic alterations and their clinical implications in gastric cancer peritoneal carcinomatosis revealed by whole-exome sequencing of malignant ascites The RhoE/ROCK/ARHGAP25 signaling pathway controls cell invasion by inhibition of Rac activity.Regulation of epithelial plasticity by miR-424 and miR-200 in a new prostate cancer metastasis model.An exploratory study of host polymorphisms in genes that clinically characterize breast cancer tumors and pretreatment cognitive performance in breast cancer survivors.Enhanced shedding of extracellular vesicles from amoeboid prostate cancer cells: potential effects on the tumor microenvironment.Genetics of breast cancer bone metastasis: a sequential multistep pattern.Regulation of prostate cancer progression by the tumor microenvironment.Loss of protocadherin-17 (PCDH-17) promotes metastasis and invasion through hyperactivation of EGFR/MEK/ERK signaling pathway in hepatocellular carcinoma.Metastasis of aggressive amoeboid sarcoma cells is dependent on Rho/ROCK/MLC signaling.Lack of Diaph3 relaxes the spindle checkpoint causing the loss of neural progenitors.Extracellular vesicles for liquid biopsy in prostate cancer: where are we and where are we headed?Trading in your spindles for blebs: the amoeboid tumor cell phenotype in prostate cancer.Dual Targeting of Mesenchymal and Amoeboid Motility Hinders Metastatic Behavior.Long Non Coding RNA Expression Intersecting Cancer and Spermatogenesis: A Systematic Review DIAPH3 promoted the growth, migration and metastasis of hepatocellular carcinoma cells by activating beta-catenin/TCF signaling.Differential Toxicity of mDia Formin-Directed Functional Agonists and Antagonists in Developing Zebrafish.Carcinoma associated fibroblasts (CAFs) promote breast cancer motility by suppressing mammalian Diaphanous-related formin-2 (mDia2).Large extracellular vesicles carry most of the tumour DNA circulating in prostate cancer patient plasma

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P2860

DIAPH3 governs the cellular transition to the amoeboid tumour phenotype.

http://www.wikidata.org/entity/Q39347119

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

@zh-tw

2012年论文

@wuu

2012年论文

@zh

2012年论文

@zh-cn

name

DIAPH3 governs the cellular transition to the amoeboid tumour phenotype.

@en

DIAPH3 governs the cellular transition to the amoeboid tumour phenotype.

@nl

type

label

DIAPH3 governs the cellular transition to the amoeboid tumour phenotype.

@en

DIAPH3 governs the cellular transition to the amoeboid tumour phenotype.

@nl

prefLabel

DIAPH3 governs the cellular transition to the amoeboid tumour phenotype.

@en

DIAPH3 governs the cellular transition to the amoeboid tumour phenotype.

@nl

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EMBO Molecular Medicine

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DIAPH3 governs the cellular transition to the amoeboid tumour phenotype.

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P2093

Diane R Bielenberg

http://www.w3.org/2001/XMLSchema#string

Dolores Di Vizio

http://www.w3.org/2001/XMLSchema#string

Francesca Demichelis

http://www.w3.org/2001/XMLSchema#string

Ghassan Mouneimne

http://www.w3.org/2001/XMLSchema#string

Henry N Higgs

http://www.w3.org/2001/XMLSchema#string

Ilona N Holcomb

http://www.w3.org/2001/XMLSchema#string

Jayoung Kim

http://www.w3.org/2001/XMLSchema#string

Keith R Solomon

http://www.w3.org/2001/XMLSchema#string

Martin H Hager

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Matteo Morello

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P2860

Regulation of epidermal growth factor receptor trafficking by lysine deacetylase HDAC6

Formins and microtubules

The basics of epithelial-mesenchymal transition

Genes involved in viral carcinogenesis and tumor initiation in hepatitis C virus-induced hepatocellular carcinoma

Disruption of the Diaphanous-related formin Drf1 gene encoding mDia1 reveals a role for Drf3 as an effector for Cdc42

Mechanism and function of formins in the control of actin assembly

Origins and evolution of the formin multigene family that is involved in the formation of actin filaments

Rac activation and inactivation control plasticity of tumor cell movement

The landscape of somatic copy-number alteration across human cancers.

Compensation mechanism in tumor cell migration: mesenchymal-amoeboid transition after blocking of pericellular proteolysis

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743-760

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10.1002/EMMM.201200242

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8

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4

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2012-05-16T00:00:00Z

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224972243

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22593025

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3494074

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