Effect of 8-hydroxyquinoline and derivatives on human neuroblastoma SH-SY5Y cells under high glucose.

about

Effect of 8-hydroxyquinoline and derivatives on human neuroblastoma SH-SY5Y cells under high glucose.

Item

http://www.wikidata.org/entity/Q39382593

description

2016 nî lūn-bûn

@nan

2016年の論文

@ja

2016年学术文章

@wuu

2016年学术文章

@zh-cn

2016年学术文章

@zh-hans

2016年学术文章

@zh-my

2016年学术文章

@zh-sg

2016年學術文章

@yue

2016年學術文章

@zh

2016年學術文章

@zh-hant

name

Effect of 8-hydroxyquinoline a ...... SY5Y cells under high glucose.

@en

Effect of 8-hydroxyquinoline a ...... SY5Y cells under high glucose.

@nl

type

Item

label

Effect of 8-hydroxyquinoline a ...... SY5Y cells under high glucose.

@en

Effect of 8-hydroxyquinoline a ...... SY5Y cells under high glucose.

@nl

prefLabel

Effect of 8-hydroxyquinoline a ...... SY5Y cells under high glucose.

@en

Effect of 8-hydroxyquinoline a ...... SY5Y cells under high glucose.

@nl

P1476

Effect of 8-hydroxyquinoline a ...... SY5Y cells under high glucose.

@en

P2093

Supaluk Prachayasittikul

http://www.w3.org/2001/XMLSchema#string

Virapong Prachayasittikul

http://www.w3.org/2001/XMLSchema#string

Wilasinee Suwanjang

http://www.w3.org/2001/XMLSchema#string

P2860

Calpain-5 mutations cause autoimmune uveitis, retinal neovascularization, and photoreceptor degeneration

Histochemically-reactive zinc in amyloid plaques, angiopathy, and degenerating neurons of Alzheimer's diseased brains

The calpain system

Targeting of CaV3.2 T-type calcium channels in peripheral sensory neurons for the treatment of painful diabetic neuropathy

8-Hydroxyquinolines: a review of their metal chelating properties and medicinal applications

Amyloid beta protein forms ion channels: implications for Alzheimer's disease pathophysiology.

High glucose-induced apoptosis in human endothelial cells is mediated by sequential activations of c-Jun NH(2)-terminal kinase and caspase-3.

Clioquinol and other hydroxyquinoline derivatives inhibit Abeta(1-42) oligomer assembly.

High glucose-induced oxidative stress increases transient receptor potential channel expression in human monocytes.

Glucose or diabetes activates p38 mitogen-activated protein kinase via different pathways

P304

e2389

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.7717/PEERJ.2389

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2016-08-31T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

27635352

http://www.w3.org/2001/XMLSchema#string

P921

glycobiology

P932

5012261

http://www.w3.org/2001/XMLSchema#string