Association of linear plasmid 28-1 with an arthritic phenotype of Borrelia burgdorferi.
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Outer surface protein B is critical for Borrelia burgdorferi adherence and survival within Ixodes ticksRole of outer surface protein D in the Borrelia burgdorferi life cycleDecorin binding proteins of Borrelia burgdorferi promote arthritis development and joint specific post-treatment DNA persistence in miceIncreasing the interaction of Borrelia burgdorferi with decorin significantly reduces the 50 percent infectious dose and severely impairs dissemination.Inactivation of a putative flagellar motor switch protein FliG1 prevents Borrelia burgdorferi from swimming in highly viscous media and blocks its infectivityCommon and unique contributions of decorin-binding proteins A and B to the overall virulence of Borrelia burgdorferi.Detailed analysis of sequence changes occurring during vlsE antigenic variation in the mouse model of Borrelia burgdorferi infection.High-throughput plasmid content analysis of Borrelia burgdorferi B31 by using Luminex multiplex technology.Outer surface protein C is a dissemination-facilitating factor of Borrelia burgdorferi during mammalian infection.Role of the surface lipoprotein BBA07 in the enzootic cycle of Borrelia burgdorferi.BB0250 of Borrelia burgdorferi is a conserved and essential inner membrane protein required for cell division.RpoS regulates essential virulence factors remaining to be identified in Borrelia burgdorferi.Borrelia burgdorferi lacking BBK32, a fibronectin-binding protein, retains full pathogenicity.Influence of arthritis-related protein (BBF01) on infectivity of Borrelia burgdorferi B31.Two regulatory elements required for enhancing ospA expression in Borrelia burgdorferi grown in vitro but repressing its expression during mammalian infection.Increasing RpoS expression causes cell death in Borrelia burgdorferiDifferential telomere processing by Borrelia telomere resolvases in vitro but not in vivoBosR functions as a repressor of the ospAB operon in Borrelia burgdorferi.The critical role of the linear plasmid lp36 in the infectious cycle of Borrelia burgdorferiBorrelia burgdorferi basic membrane proteins A and B participate in the genesis of Lyme arthritisEssential protective role attributed to the surface lipoproteins of Borrelia burgdorferi against innate defencesIn vitro CpG methylation increases the transformation efficiency of Borrelia burgdorferi strains harboring the endogenous linear plasmid lp56Constitutive expression of outer surface protein C diminishes the ability of Borrelia burgdorferi to evade specific humoral immunity.The dbpBA locus of Borrelia burgdorferi is not essential for infection of miceVerification and dissection of the ospC operator by using flaB promoter as a reporter in Borrelia burgdorferi.Genetic Manipulation of Borrelia Spp.Both decorin-binding proteins A and B are critical for the overall virulence of Borrelia burgdorferi
P2860
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P2860
Association of linear plasmid 28-1 with an arthritic phenotype of Borrelia burgdorferi.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Association of linear plasmid 28-1 with an arthritic phenotype of Borrelia burgdorferi.
@en
Association of linear plasmid 28-1 with an arthritic phenotype of Borrelia burgdorferi.
@nl
type
label
Association of linear plasmid 28-1 with an arthritic phenotype of Borrelia burgdorferi.
@en
Association of linear plasmid 28-1 with an arthritic phenotype of Borrelia burgdorferi.
@nl
prefLabel
Association of linear plasmid 28-1 with an arthritic phenotype of Borrelia burgdorferi.
@en
Association of linear plasmid 28-1 with an arthritic phenotype of Borrelia burgdorferi.
@nl
P2093
P2860
P1476
Association of linear plasmid 28-1 with an arthritic phenotype of Borrelia burgdorferi.
@en
P2093
Fang Ting Liang
Kristy McShan
Larry Lomax
Sunita V Seemanapalli
P2860
P304
P356
10.1128/IAI.73.11.7208-7215.2005
P407
P50
P577
2005-11-01T00:00:00Z