Enforced viral replication activates adaptive immunity and is essential for the control of a cytopathic virus. - Wikidata - wikimedia - TriplyDB

Enforced viral replication activates adaptive immunity and is essential for the control of a cytopathic virus.

Enforced viral replication activates adaptive immunity and is essential for the control of a cytopathic virus.

http://www.wikidata.org/entity/Q39439883

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Harnessing Mechanistic Knowledge on Beneficial Versus Deleterious IFN-I Effects to Design Innovative Immunotherapies Targeting Cytokine Activity to Specific Cell Types Tissue-resident macrophages The Lymphotoxin Network: orchestrating a type I interferon response to optimize adaptive immunity Subcapsular sinus macrophage fragmentation and CD169+ bleb acquisition by closely associated IL-17-committed innate-like lymphocytes The regulation of T follicular helper responses during infection Protective Capacity of the Human Anamnestic Antibody Response during Acute Dengue Virus Infection The mannose receptor High efficiency cell-specific targeting of cytokine activity Cytokine Activation by Antibody Fragments Targeted to Cytokine-Receptor Signaling Complexes.Peripheral prepositioning and local CXCL9 chemokine-mediated guidance orchestrate rapid memory CD8+ T cell responses in the lymph node.Neurovirulence and immunogenicity of attenuated recombinant vesicular stomatitis viruses in nonhuman primates.USP18 is crucial for IFN-γ-mediated inhibition of B16 melanoma tumorigenesis and antitumor immunity.A systemic macrophage response is required to contain a peripheral poxvirus infection Efficient transduction of myeloid cells by an HIV-1-derived lentiviral vector that packages the Vpx accessory protein Poly(I:C) treatment leads to interferon-dependent clearance of hepatitis B virus in a hydrodynamic injection mouse model.Selective inactivation of USP18 isopeptidase activity in vivo enhances ISG15 conjugation and viral resistance Lipid nanoparticle siRNA treatment of Ebola-virus-Makona-infected nonhuman primates.Lymphotoxin-LIGHT pathway regulates the interferon signature in rheumatoid arthritis.Usp18 driven enforced viral replication in dendritic cells contributes to break of immunological tolerance in autoimmune diabetes.CEACAM1 induces B-cell survival and is essential for protective antiviral antibody production Tumor necrosis factor superfamily in innate immunity and inflammation.Deficiency of the B cell-activating factor receptor results in limited CD169+ macrophage function during viral infection CD169+ macrophages are sufficient for priming of CTLs with specificities left out by cross-priming dendritic cells.Much More than M1 and M2 Macrophages, There are also CD169(+) and TCR(+) Macrophages.Lymph Node Macrophages Restrict Murine Cytomegalovirus Dissemination Antiviral Protection via RdRP-Mediated Stable Activation of Innate Immunity.Stat1-Deficient Mice Are Not an Appropriate Model for Efficacy Testing of Recombinant Vesicular Stomatitis Virus-Based Filovirus Vaccines Type 1 Interferons and NK Cells Limit Murine Cytomegalovirus Escape from the Lymph Node Subcapsular Sinus.Global survey of the immunomodulatory potential of common drugs.Subcapsular sinus macrophages limit acute gammaherpesvirus dissemination Virus-specific antibodies allow viral replication in the marginal zone, thereby promoting CD8(+) T-cell priming and viral control Type I interferons in viral control and immune regulation Lymphotoxin network pathways shape the tumor microenvironment Lymphoid-tissue stromal cells coordinate innate defense to cytomegalovirus.Lipopolysaccharide and Tumor Necrosis Factor Alpha Inhibit Interferon Signaling in Hepatocytes by Increasing Ubiquitin-Like Protease 18 (USP18) Expression.Rapid early innate control of hepatitis C virus during IFN-α treatment compromises adaptive CD4+ T-cell immunity.Type I Interferons and NK Cells Restrict Gammaherpesvirus Lymph Node Infection.Immunoactivation induced by chronic viral infection inhibits viral replication and drives immunosuppression through sustained IFN-I responses.CD169+ macrophages regulate PD-L1 expression via type I interferon and thereby prevent severe immunopathology after LCMV infection.Presentation of Autoantigen in Peripheral Lymph Nodes Is Sufficient for Priming Autoreactive CD8+ T Cells.

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P2860

Enforced viral replication activates adaptive immunity and is essential for the control of a cytopathic virus.

http://www.wikidata.org/entity/Q39439883

description

2011 nî lūn-bûn

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2011年の論文

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年學術文章

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2011年學術文章

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2011年學術文章

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Enforced viral replication act ...... control of a cytopathic virus.

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Enforced viral replication act ...... control of a cytopathic virus.

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Enforced viral replication act ...... control of a cytopathic virus.

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Enforced viral replication act ...... control of a cytopathic virus.

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Enforced viral replication act ...... control of a cytopathic virus.

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Enforced viral replication act ...... control of a cytopathic virus.

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Nature Immunology

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Enforced viral replication act ...... control of a cytopathic virus.

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P2093

Christoph Burkart

http://www.w3.org/2001/XMLSchema#string

Dong-Er Zhang

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Giuseppe Cadeddu

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Hartmut Hengel

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Karl S Lang

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Masato Tanaka

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Max Löhning

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Melanie Grusdat

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Mike Recher

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Nadine Honke

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P2860

Functional role of type I and type II interferons in antiviral defense

Immunoproteasomes preserve protein homeostasis upon interferon-induced oxidative stress

UBP43 is a novel regulator of interferon signaling independent of its ISG15 isopeptidase activity

The alpha/beta interferon response controls tissue tropism and pathogenicity of poliovirus.

Interferon-inducible antiviral effectors

Mechanisms of type-I- and type-II-interferon-mediated signalling

Identification of genes differentially regulated by interferon alpha, beta, or gamma using oligonucleotide arrays

Localization dose and time of antigens determine immune reactivity.

Antigen decoding by T lymphocytes: from synapses to fate determination.

Critical role of macrophages in the marginal zone in the suppression of immune responses to apoptotic cell-associated antigens.

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51-57

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10.1038/NI.2169

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1

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13

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Dieter Häussinger

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Reinhard Kandolf

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2011-11-20T00:00:00Z

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51815936

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22101728

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