Hypoxia activates the notch signaling pathway in cells of the intervertebral disc: implications in degenerative disc disease.
about
Disc in flames: Roles of TNF-α and IL-1β in intervertebral disc degenerationDiversity of intervertebral disc cells: phenotype and functionRole of cytokines in intervertebral disc degeneration: pain and disc contentTumor necrosis factor alpha promotes the proliferation of human nucleus pulposus cells via nuclear factor-κB, c-Jun N-terminal kinase, and p38 mitogen-activated protein kinaseInflammatory cytokines induce NOTCH signaling in nucleus pulposus cells: implications in intervertebral disc degenerationGlobal gene expression changes induced by prolonged cold ischemic stress and preservation method of breast cancer tissue.FIH-1-Mint3 axis does not control HIF-1 transcriptional activity in nucleus pulposus cells.Hypoxia-induced down-regulation of microRNA-34a promotes EMT by targeting the Notch signaling pathway in tubular epithelial cells.Notochordal cells in the adult intervertebral disc: new perspective on an old question.Understanding nucleus pulposus cell phenotype: a prerequisite for stem cell based therapies to treat intervertebral disc degenerationTranscription Factor KLF2 in Dendritic Cells Downregulates Th2 Programming via the HIF-1α/Jagged2/Notch Axis.Cell signaling pathways related to pain receptors in the degenerated disk.Notch1 controls development of the extravillous trophoblast lineage in the human placenta.Reconstruction of an in vitro niche for the transition from intervertebral disc development to nucleus pulposus regeneration.Understanding the native nucleus pulposus cell phenotype has important implications for intervertebral disc regeneration strategies.Molecular mechanisms of cell death in intervertebral disc degeneration (Review).Tumor necrosis factor-α: a key contributor to intervertebral disc degeneration.Circadian factors BMAL1 and RORα control HIF-1α transcriptional activity in nucleus pulposus cells: implications in maintenance of intervertebral disc healthUse of Hes1-GFP reporter mice to assess activity of the Hes1 promoter in bone cells under chronic inflammation.Pramlintide regulation of extracellular matrix (ECM) and apoptosis through mitochondrial-dependent pathways in human nucleus pulposus cells.The E3 ubiquitin ligase SCFFBXL14 complex stimulates neuronal differentiation by targeting the Notch signaling factor HES1 for proteolysis.Circular RNA VMA21 protects against intervertebral disc degeneration through targeting miR-200c and X linked inhibitor-of-apoptosis protein.TGF-β1 induces a nucleus pulposus-like phenotype in Notch 1 knockdown rabbit bone marrow mesenchymal stem cells.Effect of young extrinsic environment stimulated by hypoxia on the function of aged tendon stem cell.
P2860
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P2860
Hypoxia activates the notch signaling pathway in cells of the intervertebral disc: implications in degenerative disc disease.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Hypoxia activates the notch si ...... in degenerative disc disease.
@en
Hypoxia activates the notch si ...... in degenerative disc disease.
@nl
type
label
Hypoxia activates the notch si ...... in degenerative disc disease.
@en
Hypoxia activates the notch si ...... in degenerative disc disease.
@nl
prefLabel
Hypoxia activates the notch si ...... in degenerative disc disease.
@en
Hypoxia activates the notch si ...... in degenerative disc disease.
@nl
P2093
P2860
P356
P1476
Hypoxia activates the notch si ...... in degenerative disc disease.
@en
P2093
Akihiko Hiyama
D Greg Anderson
Daisuke Sakai
Dessislava Markova
Irving M Shapiro
Joji Mochida
Makarand V Risbud
Renata Skubutyte
Sanjay Yadla
Todd J Albert
P2860
P304
P356
10.1002/ART.30246
P577
2011-05-01T00:00:00Z