Ca2+ cycling and new therapeutic approaches for heart failure. - Wikidata - wikimedia - TriplyDB

Ca2+ cycling and new therapeutic approaches for heart failure.

Ca2+ cycling and new therapeutic approaches for heart failure.

http://www.wikidata.org/entity/Q39493909

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Dysfunctional ryanodine receptors in the heart: new insights into complex cardiovascular diseases Imaging alterations of cardiomyocyte cAMP microdomains in disease Adeno-associated virus-mediated gene therapy in cardiovascular disease S100A1 in human heart failure: lack of recovery following left ventricular assist device support Molecular targets in heart failure gene therapy: current controversies and translational perspectives Expression of sarco (endo) plasmic reticulum calcium ATPase (SERCA) system in normal mouse cardiovascular tissues, heart failure and atherosclerosis Differential effects of S100 proteins A2 and A6 on cardiac Ca(2+) cycling and contractile performance.Mechanisms of Ca²+ handling in zebrafish ventricular myocytes (Pro)renin receptor triggers distinct angiotensin II-independent extracellular matrix remodeling and deterioration of cardiac function.High-throughput FRET assay yields allosteric SERCA activators Different roles for contracture and calpain in calcium paradox-induced heart injury.Fetal cardiac troponin isoforms rescue the increased Ca2+ sensitivity produced by a novel double deletion in cardiac troponin T linked to restrictive cardiomyopathy: a clinical, genetic, and functional approach A novel artificial microRNA expressing AAV vector for phospholamban silencing in cardiomyocytes improves Ca2+ uptake into the sarcoplasmic reticulum.Dysfunctional conformational dynamics of protein kinase A induced by a lethal mutant of phospholamban hinder phosphorylation.Mice lacking functional TRPV1 are protected from pressure overload cardiac hypertrophy In search of new therapeutic targets and strategies for heart failure: recent advances in basic science Calcium cycling proteins and heart failure: mechanisms and therapeutics In vivo and in vitro cardiac responses to beta-adrenergic stimulation in volume-overload heart failure Short-term mechanical unloading with left ventricular assist devices after acute myocardial infarction conserves calcium cycling and improves heart function.Inducible overexpression of GLUT1 prevents mitochondrial dysfunction and attenuates structural remodeling in pressure overload but does not prevent left ventricular dysfunction.Interactions of Calcium Fluctuations during Cardiomyocyte Contraction with Real-Time cAMP Dynamics Detected by FRET.Ca handling during excitation-contraction coupling in heart failure.Novel therapeutic targets for the treatment of heart failure.Calcium cycling proteins and their association with heart failure.Molecular targets of current and prospective heart failure therapies.Compartmentation of cAMP signalling in cardiomyocytes in health and disease.Biophysical techniques for detection of cAMP and cGMP in living cells.Heart failure with preserved ejection fraction: a clinical dilemma.Myocardial recovery: a focus on the impact of left ventricular assist devices.Gene therapy to treat cardiac arrhythmias.Heart Failure with Preserved Ejection Fraction and Future Pharmacological Strategies: a Glance in the Crystal Ball.Pathophysiological mechanism and therapeutic role of S100 proteins in cardiac failure: a systematic review.Cardiac gene therapy: are we there yet?Functional Impact of Ryanodine Receptor Oxidation on Intracellular Calcium Regulation in the Heart.Post-extrasystolic Potentiation: Link between Ca(2+) Homeostasis and Heart Failure?Concise Review: Mending a Broken Heart: The Evolution of Biological Therapeutics.Raf kinase inhibitor protein: lessons of a better way for β-adrenergic receptor activation in the heart.Prevention of Atrial Fibrillation by Using Sarcoplasmic Reticulum Calcium ATPase Pump Overexpression in a Rabbit Model of Rapid Atrial Pacing.A calcium-sensitive promoter construct for gene therapy.In vivo model with targeted cAMP biosensor reveals changes in receptor-microdomain communication in cardiac disease.

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Ca2+ cycling and new therapeutic approaches for heart failure.

http://www.wikidata.org/entity/Q39493909

description

2010 nî lūn-bûn

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2010年の論文

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年學術文章

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2010年學術文章

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2010年學術文章

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name

Ca2+ cycling and new therapeutic approaches for heart failure.

@en

Ca2+ cycling and new therapeutic approaches for heart failure.

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type

label

Ca2+ cycling and new therapeutic approaches for heart failure.

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Ca2+ cycling and new therapeutic approaches for heart failure.

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prefLabel

Ca2+ cycling and new therapeutic approaches for heart failure.

@en

Ca2+ cycling and new therapeutic approaches for heart failure.

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Ca2+ cycling and new therapeutic approaches for heart failure.

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Andrew R Marks

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Anne-Marie Lompré

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Evangelia G Kranias

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Martin J Lohse

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Roger J Hajjar

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Sian E Harding

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P2860

Restoration of contractile function in isolated cardiomyocytes from failing human hearts by gene transfer of SERCA2a.

PKA phosphorylation dissociates FKBP12.6 from the calcium release channel (ryanodine receptor): defective regulation in failing hearts

Sudden death in familial polymorphic ventricular tachycardia associated with calcium release channel (ryanodine receptor) leak

Clinical phenotype and functional characterization of CASQ2 mutations associated with catecholaminergic polymorphic ventricular tachycardia

On the role of junctin in cardiac Ca2+ handling, contractility, and heart failure

Histidine-rich Ca-binding protein interacts with sarcoplasmic reticulum Ca-ATPase

Hypernitrosylated ryanodine receptor calcium release channels are leaky in dystrophic muscle

The Ser96Ala variant in histidine-rich calcium-binding protein is associated with life-threatening ventricular arrhythmias in idiopathic dilated cardiomyopathy

Phosphodiesterase 4D deficiency in the ryanodine-receptor complex promotes heart failure and arrhythmias

Multidrug resistance-associated protein 4 regulates cAMP-dependent signaling pathways and controls human and rat SMC proliferation

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20124124

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