Cisplatin treatment of primary and metastatic epithelial ovarian carcinomas generates residual cells with mesenchymal stem cell-like profile. - Wikidata - wikimedia - TriplyDB

Cisplatin treatment of primary and metastatic epithelial ovarian carcinomas generates residual cells with mesenchymal stem cell-like profile.

Cisplatin treatment of primary and metastatic epithelial ovarian carcinomas generates residual cells with mesenchymal stem cell-like profile.

http://www.wikidata.org/entity/Q39535066

about

A fresh look at zebrafish from the perspective of cancer research Chemotherapy-enhanced inflammation may lead to the failure of therapy and metastasis Emergence of zebrafish models in oncology for validating novel anticancer drug targets and nanomaterials The Role of TLR4 in Chemotherapy-Driven Metastasis Cancer stem cells, epithelial-mesenchymal transition, and drug resistance in high-grade ovarian serous carcinoma Hooking the big one: the potential of zebrafish xenotransplantation to reform cancer drug screening in the genomic era Epimorphin-induced MET sensitizes ovarian cancer cells to platinum Resveratrol inhibits cisplatin-induced epithelial-to-mesenchymal transition in ovarian cancer cell lines Ovarian cancer cell line panel (OCCP): clinical importance of in vitro morphological subtypes Nanog and β-catenin: a new convergence point in EpSC proliferation and differentiation Inhibition of the JAK2/STAT3 pathway in ovarian cancer results in the loss of cancer stem cell-like characteristics and a reduced tumor burden HIF-1α binding to AEG-1 promoter induced upregulated AEG-1 expression associated with metastasis in ovarian cancer.Epithelial-to-Mesenchymal Transition in the Female Reproductive Tract: From Normal Functioning to Disease Pathology.Metformin against cancer stem cells through the modulation of energy metabolism: special considerations on ovarian cancer.Antagonism of miR-21 reverses epithelial-mesenchymal transition and cancer stem cell phenotype through AKT/ERK1/2 inactivation by targeting PTEN TWIST and ovarian cancer stem cells: implications for chemoresistance and metastasis Isolation and characterization of tumor cells from the ascites of ovarian cancer patients: molecular phenotype of chemoresistant ovarian tumors.ARID3B increases ovarian tumor burden and is associated with a cancer stem cell gene signature Transforming growth factor-β signalling controls human breast cancer metastasis in a zebrafish xenograft model.Cisplatin-selected resistance is associated with increased motility and stem-like properties via activation of STAT3/Snail axis in atypical teratoid/rhabdoid tumor cells Preclinical safety and efficacy studies with an affinity-enhanced epithelial junction opener and PEGylated liposomal doxorubicin.Acquisition of resistance to trastuzumab in gastric cancer cells is associated with activation of IL-6/STAT3/Jagged-1/Notch positive feedback loop Down-regulation of ARNT promotes cancer metastasis by activating the fibronectin/integrin β1/FAK axis.Overexpression of the RNA-binding proteins Lin28B and IGF2BP3 (IMP3) is associated with chemoresistance and poor disease outcome in ovarian cancer.A critical role of Oct4A in mediating metastasis and disease-free survival in a mouse model of ovarian cancer.Epithelial-mesenchymal transition in ovarian carcinoma Lysophosphatidic Acid Initiates Epithelial to Mesenchymal Transition and Induces β-Catenin-mediated Transcription in Epithelial Ovarian Carcinoma.Coalition of Oct4A and β1 integrins in facilitating metastasis in ovarian cancer.Sensitizing ovarian cancer cells to chemotherapy by interfering with pathways that are involved in the formation of cancer stem cells.The lncRNA H19 promotes epithelial to mesenchymal transition by functioning as miRNA sponges in colorectal cancer.Role of intratumoural heterogeneity in cancer drug resistance: molecular and clinical perspectives Critical role of Wnt/β-catenin signaling in driving epithelial ovarian cancer platinum resistance.The emerging roles of Oct4 in tumor-initiating cells.Attributes of Oct4 in stem cell biology: perspectives on cancer stem cells of the ovary.MicroRNA profiling of cisplatin-resistant oral squamous cell carcinoma cell lines enriched with cancer-stem-cell-like and epithelial-mesenchymal transition-type features.Short-term single treatment of chemotherapy results in the enrichment of ovarian cancer stem cell-like cells leading to an increased tumor burden Strong synergy with APR-246 and DNA-damaging drugs in primary cancer cells from patients with TP53 mutant High-Grade Serous ovarian cancer Low-dose cisplatin-induced CXCR4 expression promotes proliferation of ovarian cancer stem-like cells.Inhibition of endotrophin, a cleavage product of collagen VI, confers cisplatin sensitivity to tumours.Getting to know ovarian cancer ascites: opportunities for targeted therapy-based translational research.

P2860

Q26799700-BB546C5E-E556-496C-B120-85CC262BEDD6 Q26858839-D4C4F792-278C-4A9F-AA9F-F93C079F7318 Q26860518-D3F7E882-CDFB-47DC-9D34-3E267E962957 Q26861495-A53F4A48-C8A5-4AC7-B559-BE2BEB478EE5 Q26998068-A0B6A62F-A039-472C-87D1-468B891F6B9D Q27024966-DE0D50CF-4EAA-4E8B-A8AE-65291A6A7927 Q28536991-1EFA7603-6BB8-42E2-BF38-994E1EF923B2 Q28539044-EEF66F57-94E8-409C-83DB-0F0D9262ECF2 Q28543021-97FD94BA-D099-44BB-88D7-D49C2E5EC055 Q28570720-0B2F57A8-1A6F-4B68-AE80-9E668B848C5B Q33628532-ECC94A52-E6A4-4940-A304-A78D35F9E110 Q33679589-80A227AB-E6C8-4226-A14E-B238410AA253 Q33872622-53B70647-7023-4177-8A5A-DFEC587B76F3 Q33890763-9010E0A8-41CD-4BC4-B4E0-3B1314B81C42 Q34326206-9F197093-EC62-4D9A-880D-6F96334DDD5E Q34365639-790884E0-92A5-473C-8870-A73ED477C20F Q34442318-5BFA6CDD-AED6-470B-A2A8-6C67F1CBC6D4 Q34483574-6B69171C-38B8-41B5-82ED-9493B4994453 Q35033110-80262D08-C256-408F-B093-8EC7DC76F42D Q35176432-CE476B0A-92A1-413D-8A9C-E5EB634555D7 Q35651085-7749B2B6-A100-4E53-9E0D-C717DA4AEDCE Q35741610-40F2B9B0-25B8-4A3B-9FA2-6E109920ABCB Q35793846-1B1C8060-3FCA-428B-A9B9-1D8225CE669A Q35909674-FB6F2F78-1E58-441B-90AA-DD0EE73CC4AA Q35936614-BC420950-F08F-4204-8F0B-AA6774630754 Q35971814-3BD9F54D-7F6B-4594-8FFA-B6288086EF3A Q36065252-1E3D32A4-FCDA-4789-93FA-DBAF81F69FFB Q36070842-0AF24907-7374-4A63-A34B-27052ABDD381 Q36116632-E8843013-DC24-4A16-9649-CF19DB0AD77F Q36356007-0154F5D7-DDB4-4C42-A9A9-D1A2996C5845 Q36386428-588E853F-EB75-409E-9C2D-B762DE16C9CA Q36414274-7AE70D94-D8DD-47CD-965C-4ED91FB1BFB5 Q36498067-B70EA8F6-8D6E-4BE9-ABB7-31F70B9BFEED Q36507476-235DB181-E763-41B8-938E-03E0E9CA75A9 Q36764848-BBFB431A-D8C2-455D-8875-94C443675641 Q36891437-D7B06F21-58B1-4E8E-8ED8-57B61DB2333A Q36904012-83D3448E-50D0-401A-BB9E-8E9061E3C555 Q36947111-37ECE5B3-1CC6-4A00-859C-306C4821C29B Q37188588-1928A6F6-ADCA-419D-80E8-605302585C52 Q37196532-E08AC2A0-7D99-4C93-A831-B391CDBDBE66

P2860

Cisplatin treatment of primary and metastatic epithelial ovarian carcinomas generates residual cells with mesenchymal stem cell-like profile.

http://www.wikidata.org/entity/Q39535066

description

2011 nî lūn-bûn

@nan

2011年の論文

@ja

2011年論文

@yue

2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

@wuu

2011年论文

@zh

2011年论文

@zh-cn

name

Cisplatin treatment of primary ...... chymal stem cell-like profile.

@en

Cisplatin treatment of primary ...... chymal stem cell-like profile.

@nl

type

label

Cisplatin treatment of primary ...... chymal stem cell-like profile.

@en

Cisplatin treatment of primary ...... chymal stem cell-like profile.

@nl

prefLabel

Cisplatin treatment of primary ...... chymal stem cell-like profile.

@en

Cisplatin treatment of primary ...... chymal stem cell-like profile.

@nl

P1433

Q39535066-632A4786-D14B-4C7A-861D-D6E6AF106F07

P1476

Q39535066-FC2C5FD1-9BBF-46D8-96CC-F1A2A2407350

P2093

Q39535066-5470DFAF-5932-44E8-BB38-711B3413FCA0

Q39535066-659505B5-B4DD-4431-81F0-97CFC183D940

Q39535066-68504724-89E6-4E17-8803-F74EE1574AD0

Q39535066-A7E30C0A-CD07-4531-9AA1-E0233E64E338

Q39535066-C09935BA-CCF4-47DB-BAEA-58A6001E7FCF

Q39535066-CD36EF55-89F7-4E3C-AD9A-BB3700BB22ED

Q39535066-D4205EF7-565A-4EC8-9035-8CAF37416205

Q39535066-EF8E3F24-F4F9-4AC6-80D8-DFB1BF92774A

P2860

Q39535066-082A78BC-1BC0-45C7-A245-991091062DE7

Q39535066-11FE4E41-3CB8-417E-A87D-302D9F34A9AB

Q39535066-185DD19A-224E-49F4-938F-704250B87EBB

Q39535066-1E07E524-26CD-4262-A3DE-506FAFE4EB9B

Q39535066-23478075-ADAD-4D57-913B-BFB6BD421B4F

Q39535066-24FE9996-16BD-4FC5-B0CF-A8D574139F7D

Q39535066-25B980CE-08A1-4819-827F-F7D1BD219164

Q39535066-32A3828C-0DC4-4C5C-AB57-639352E83148

Q39535066-379AD951-1425-4507-B657-4636A821FC50

Q39535066-397ADAB6-879E-472A-A615-957C7827DDD5

P304

Q39535066-FFB7B0AA-57E5-4DFD-A33F-0775FB7CCE29

P31

Q39535066-EA584A8B-FCEB-4947-88A6-BE982122D817

P356

Q39535066-0EC701A0-6455-44F7-B55F-9247069EE642

P433

Q39535066-FB8B308E-3B1B-4480-B181-7B3BD4A1068A

P478

Q39535066-A87D87E1-042C-47EA-9C3E-4654047509F7

P50

Q39535066-42983BD0-6823-45AC-BDA9-8341E49BE8EF

Q39535066-C1FCFC29-428E-4595-8B67-10CC6115759C

Q39535066-E9C67444-2170-41D7-93D6-4B785EDA4B84

P577

Q39535066-0622C941-ACB4-4EF4-B529-D9022EA6FC1D

P5875

Q39535066-A89AFFF1-7117-4717-904B-EBB51F8AD663

P698

Q39535066-78D78660-2252-49A4-B632-FDE681224F8B

P356

P1433

Journal of Cellular Biochemistry

P1476

Cisplatin treatment of primary ...... nchymal stem cell-like profile

@en

P2093

Ardian Latifi

http://www.w3.org/2001/XMLSchema#string

Clifford Liongue

http://www.w3.org/2001/XMLSchema#string

Francoise Dobill

http://www.w3.org/2001/XMLSchema#string

Janani Kumar

http://www.w3.org/2001/XMLSchema#string

Jock K Findlay

http://www.w3.org/2001/XMLSchema#string

Khalid Abubaker

http://www.w3.org/2001/XMLSchema#string

Natalie Castrechini

http://www.w3.org/2001/XMLSchema#string

Nuzhat Ahmed

http://www.w3.org/2001/XMLSchema#string

P2860

Aberrant expression of the transcription factors snail and slug alters the response to genotoxic stress

Epithelial-mesenchymal transition in cancer: parallels between normal development and tumor progression

The epithelial-mesenchymal transition generates cells with properties of stem cells

EpCAM (CD326) finding its role in cancer

Prospective identification of tumorigenic breast cancer cells

Overexpression of alpha(v)beta6 integrin in serous epithelial ovarian cancer regulates extracellular matrix degradation via the plasminogen activation cascade

Snail, Zeb and bHLH factors in tumour progression: an alliance against the epithelial phenotype?

New functions for the matrix metalloproteinases in cancer progression

Metastatic behaviour of primary human tumours in a zebrafish xenotransplantation model

NPRL2 sensitizes human non-small cell lung cancer (NSCLC) cells to cisplatin treatment by regulating key components in the DNA repair pathway.

P304

2850-2864

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1002/JCB.23199

http://www.w3.org/2001/XMLSchema#string

P433

10

http://www.w3.org/2001/XMLSchema#string

P478

112

http://www.w3.org/2001/XMLSchema#string

P50

Alister C. Ward

Erik W. Thompson

Michael A. Quinn

P577

2011-10-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

51170922

http://www.w3.org/2001/XMLSchema#string

P698

21618587

http://www.w3.org/2001/XMLSchema#string