Redirection of the respiro-fermentative flux distribution in Saccharomyces cerevisiae by overexpression of the transcription factor Hap4p
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Hap4p overexpression in glucose-grown Saccharomyces cerevisiae induces cells to enter a novel metabolic stateValidation and refinement of gene-regulatory pathways on a network of physical interactionsPhysiological adaptations of Saccharomyces cerevisiae evolved for improved butanol toleranceIncreased glycolytic flux as an outcome of whole-genome duplication in yeastFluxomics - connecting ‘omics analysis and phenotypesOsmotic stress signaling and osmoadaptation in yeasts.Integrative investigation of metabolic and transcriptomic dataIntegration of metabolic modeling and phenotypic data in evaluation and improvement of ethanol production using respiration-deficient mutants of Saccharomyces cerevisiae.Gene transcription analysis of Saccharomyces cerevisiae exposed to neocarzinostatin protein-chromophore complex reveals evidence of DNA damage, a potential mechanism of resistance, and consequences of prolonged exposureCalorie restriction extends yeast life span by lowering the level of NADH.Minimization of glycerol production during the high-performance fed-batch ethanolic fermentation process in Saccharomyces cerevisiae, using a metabolic model as a prediction toolFunctional study of the Hap4-like genes suggests that the key regulators of carbon metabolism HAP4 and oxidative stress response YAP1 in yeast diverged from a common ancestor.Mechanistic study on the nuclear modifier gene MSS1 mutation suppressing neomycin sensitivity of the mitochondrial 15S rRNA C1477G mutation in Saccharomyces cerevisiaeModular pathway rewiring of Saccharomyces cerevisiae enables high-level production of L-ornithine.Reduced Histone Expression or a Defect in Chromatin Assembly Induces Respiration.The S. Cerevisiae HAP complex, a key regulator of mitochondrial function, coordinates nuclear and mitochondrial gene expression.Overexpression of a Water-Forming NADH Oxidase Improves the Metabolism and Stress Tolerance of Saccharomyces cerevisiae in Aerobic Fermentation.GSF2 deletion increases lactic acid production by alleviating glucose repression in Saccharomyces cerevisiae.The transcription factors ADR1 or CAT8 are required for RTG pathway activation and evasion from yeast acetic acid-induced programmed cell death in raffinose.A flux-sensing mechanism could regulate the switch between respiration and fermentation.Improved properties of baker's yeast mutants resistant to 2-deoxy-D-glucose.Directed evolution of pyruvate decarboxylase-negative Saccharomyces cerevisiae, yielding a C2-independent, glucose-tolerant, and pyruvate-hyperproducing yeast.An internal deletion in MTH1 enables growth on glucose of pyruvate-decarboxylase negative, non-fermentative Saccharomyces cerevisiae.Shifting the fermentative/oxidative balance in Saccharomyces cerevisiae by transcriptional deregulation of Snf1 via overexpression of the upstream activating kinase Sak1pReactive oxygen species-mediated control of mitochondrial biogenesis.Switching the mode of sucrose utilization by Saccharomyces cerevisiae.Longevity: don't hold your breath.The yeast mitochondrial proteome, a study of fermentative and respiratory growth.Saccharomyces cerevisiae glutaredoxin 5-deficient cells subjected to continuous oxidizing conditions are affected in the expression of specific sets of genes.Dynamic effects related to steady-state multiplicity in continuous Saccharomyces cerevisiae cultivations.Fermentative metabolism impedes p53-dependent apoptosis in a Crabtree-positive but not in Crabtree-negative yeast.Increased heme synthesis in yeast induces a metabolic switch from fermentation to respiration even under conditions of glucose repression.Two-dimensional transcriptome analysis in chemostat cultures. Combinatorial effects of oxygen availability and macronutrient limitation in Saccharomyces cerevisiae.The impact of MIG1 and/or MIG2 disruption on aerobic metabolism of succinate dehydrogenase negative Saccharomyces cerevisiae.Stoichiometry and compartmentation of NADH metabolism inSaccharomyces cerevisiaeDissection of the promoter of theHAP4 gene inS. cerevisiae unveils a complex regulatory framework of transcriptional regulation
P2860
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P2860
Redirection of the respiro-fermentative flux distribution in Saccharomyces cerevisiae by overexpression of the transcription factor Hap4p
description
2000 nî lūn-bûn
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2000年の論文
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2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
2000年论文
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2000年论文
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name
Redirection of the respiro-fer ...... the transcription factor Hap4p
@en
Redirection of the respiro-fer ...... he transcription factor Hap4p.
@nl
type
label
Redirection of the respiro-fer ...... the transcription factor Hap4p
@en
Redirection of the respiro-fer ...... he transcription factor Hap4p.
@nl
prefLabel
Redirection of the respiro-fer ...... the transcription factor Hap4p
@en
Redirection of the respiro-fer ...... he transcription factor Hap4p.
@nl
P2093
P2860
P1476
Redirection of the respiro-fer ...... the transcription factor Hap4p
@en
P2093
L A Grivell
M J De Mattos
P2860
P304
P356
10.1128/AEM.66.5.1970-1973.2000
P407
P577
2000-05-01T00:00:00Z