Chronic activation of wild-type epidermal growth factor receptor and loss of Cdkn2a cause mouse glioblastoma formation. - Wikidata - wikimedia - TriplyDB

Chronic activation of wild-type epidermal growth factor receptor and loss of Cdkn2a cause mouse glioblastoma formation.

Chronic activation of wild-type epidermal growth factor receptor and loss of Cdkn2a cause mouse glioblastoma formation.

http://www.wikidata.org/entity/Q39539196

about

Constitutive and ligand-induced EGFR signalling triggers distinct and mutually exclusive downstream signalling networks.Deregulated proliferation and differentiation in brain tumors.Pre-diagnostic serum levels of EGFR and ErbB2 and genetic glioma risk variants: a nested case-control study.Driver or passenger effects of augmented c-Myc and Cdc20 in gliomagenesis.An EGFR wild type-EGFRvIII-HB-EGF feed-forward loop regulates the activation of EGFRvIII.What underlies the diversity of brain tumors?EGFR-dependent mechanisms in glioblastoma: towards a better therapeutic strategy.Immune Checkpoint Blockade Biology in Mouse Models of Glioblastoma.Inhibition of EGFR induces a c-MET-driven stem cell population in glioblastoma.HBEGF promotes gliomagenesis in the context of Ink4a/Arf and Pten loss.A TNF-JNK-Axl-ERK signaling axis mediates primary resistance to EGFR inhibition in glioblastoma.Immunotherapies for malignant glioma.Analysis of Constitutive EGFR Signaling Regulating IRF3 Transcriptional Activity in Cancer Cells.A PDGFRα-driven mouse model of glioblastoma reveals a stathmin1-mediated mechanism of sensitivity to vinblastine

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Chronic activation of wild-type epidermal growth factor receptor and loss of Cdkn2a cause mouse glioblastoma formation.

http://www.wikidata.org/entity/Q39539196

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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2011年论文

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2011年论文

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name

Chronic activation of wild-typ ...... mouse glioblastoma formation.

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Chronic activation of wild-typ ...... mouse glioblastoma formation.

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type

label

Chronic activation of wild-typ ...... mouse glioblastoma formation.

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Chronic activation of wild-typ ...... mouse glioblastoma formation.

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prefLabel

Chronic activation of wild-typ ...... mouse glioblastoma formation.

@en

Chronic activation of wild-typ ...... mouse glioblastoma formation.

@nl

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0008-5472.CAN-11-1514

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Cancer Research

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Chronic activation of wild-typ ...... mouse glioblastoma formation.

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P2093

Abraham Boskovitz

http://www.w3.org/2001/XMLSchema#string

Al Charest

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Ami Raval

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Charles A Whittaker

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David E Housman

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Haihao Zhu

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Hyun Jung Jun

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Jaime Acquaviva

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Julie Lessard

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Melissa Donovan

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P2860

ERK1/2-dependent phosphorylation of BimEL promotes its rapid dissociation from Mcl-1 and Bcl-xL

Epidermal growth factor receptor mediates increased cell proliferation, migration, and aggregation in esophageal keratinocytes in vitro and in vivo

Integrated genomic analysis identifies clinically relevant subtypes of glioblastoma characterized by abnormalities in PDGFRA, IDH1, EGFR, and NF1

Comprehensive genomic characterization defines human glioblastoma genes and core pathways

A comprehensive pathway map of epidermal growth factor receptor signaling

Molecular determinants of the response of glioblastomas to EGFR kinase inhibitors.

Mechanism of epidermal growth factor regulation of Vav2, a guanine nucleotide exchange factor for Rac

Codon-improved Cre recombinase (iCre) expression in the mouse

Molecular mechanism for a role of SHP2 in epidermal growth factor receptor signaling

BIM mediates EGFR tyrosine kinase inhibitor-induced apoptosis in lung cancers with oncogenic EGFR mutations

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7198-7206

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3228869

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