Local chemokine paralysis, a novel pathogenic mechanism for Porphyromonas gingivalis.
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Altered T-cell responses by the periodontal pathogen Porphyromonas gingivalisArginine-specific protease from Porphyromonas gingivalis activates protease-activated receptors on human oral epithelial cells and induces interleukin-6 secretionLow-abundance biofilm species orchestrates inflammatory periodontal disease through the commensal microbiota and complementPorphyromonas gingivalis as a potential community activist for diseasePorphyromonas gingivalis-host interactions: open war or intelligent guerilla tactics?Historical and contemporary hypotheses on the development of oral diseases: are we there yet?Porphyromonas gingivalis suppresses adaptive immunity in periodontitis, atherosclerosis, and Alzheimer's diseasePorphyromonas gingivalis: An Overview of Periodontopathic Pathogen below the Gum LineThe enduring importance of animal models in understanding periodontal diseaseBacterial challenge stimulates formation of arachidonic acid metabolites by human keratinocytes and neutrophils in vitro.Porphyromonas gingivalis mediates inflammasome repression in polymicrobial cultures through a novel mechanism involving reduced endocytosisIdentification of Small-Molecule Inhibitors against Meso-2, 6-Diaminopimelate Dehydrogenase from Porphyromonas gingivalisIdentification and characterization of a novel adhesin unique to oral fusobacteria.Differential and coordinated expression of defensins and cytokines by gingival epithelial cells and dendritic cells in response to oral bacteria.Fusobacterium nucleatum-associated beta-defensin inducer (FAD-I): identification, isolation, and functional evaluationManipulation of Neutrophils by Porphyromonas gingivalis in the Development of Periodontitis.Invasion of aortic and heart endothelial cells by Porphyromonas gingivalisMouse paneth cell secretory responses to cell surface glycolipids of virulent and attenuated pathogenic bacteria.Cytokine profiling of macrophages exposed to Porphyromonas gingivalis, its lipopolysaccharide, or its FimA protein.Inducible expression of human beta-defensin 2 by Fusobacterium nucleatum in oral epithelial cells: multiple signaling pathways and role of commensal bacteria in innate immunity and the epithelial barrier.Interactions between periodontal bacteria and human oral epithelial cells: Fusobacterium nucleatum adheres to and invades epithelial cells.Porphyromonas gingivalis infection increases osteoclastic bone resorption and osteoblastic bone formation in a periodontitis mouse model.Porphyromonas gingivalis gingipain-R enhances interleukin-8 but decreases gamma interferon-inducible protein 10 production by human gingival fibroblasts in response to T-cell contact.Life below the gum line: pathogenic mechanisms of Porphyromonas gingivalis.Relationships between subgingival microbiota and GCF biomarkers in generalized aggressive periodontitis.Changes in gingival crevicular fluid inflammatory mediator levels during the induction and resolution of experimental gingivitis in humans.Role for fimbriae and lysine-specific cysteine proteinase gingipain K in expression of interleukin-8 and monocyte chemoattractant protein in Porphyromonas gingivalis-infected endothelial cells.Porphyromonas gingivalis lipopolysaccharide is both agonist and antagonist for p38 mitogen-activated protein kinase activation.Role of Porphyromonas gingivalis phosphoserine phosphatase enzyme SerB in inflammation, immune response, and induction of alveolar bone resorption in ratsThe keystone-pathogen hypothesis.Proteomics of Streptococcus gordonii within a model developing oral microbial community.Beyond the red complex and into more complexity: the polymicrobial synergy and dysbiosis (PSD) model of periodontal disease etiology.Tetra- and penta-acylated lipid A structures of Porphyromonas gingivalis LPS differentially activate TLR4-mediated NF-κB signal transduction cascade and immuno-inflammatory response in human gingival fibroblastsThe serine phosphatase SerB of Porphyromonas gingivalis suppresses IL-8 production by dephosphorylation of NF-κB RelA/p65Activation of the phosphatidylinositol 3-kinase/Akt pathway contributes to survival of primary epithelial cells infected with the periodontal pathogen Porphyromonas gingivalisIdentification of Porphyromonas gingivalis genes specifically expressed in human gingival epithelial cells by using differential display reverse transcription-PCRPorphyromonas gingivalis lipopolysaccharide antagonizes Escherichia coli lipopolysaccharide at toll-like receptor 4 in human endothelial cells.P. gingivalis modulates keratinocytes through FOXO transcription factors.Interleukin-8 responses of multi-layer gingival epithelia to subgingival biofilms: role of the "red complex" speciesGenetic exchange of fimbrial alleles exemplifies the adaptive virulence strategy of Porphyromonas gingivalis.
P2860
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P2860
Local chemokine paralysis, a novel pathogenic mechanism for Porphyromonas gingivalis.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
1998年论文
@zh
1998年论文
@zh-cn
name
Local chemokine paralysis, a novel pathogenic mechanism for Porphyromonas gingivalis.
@en
Local chemokine paralysis, a novel pathogenic mechanism for Porphyromonas gingivalis.
@nl
type
label
Local chemokine paralysis, a novel pathogenic mechanism for Porphyromonas gingivalis.
@en
Local chemokine paralysis, a novel pathogenic mechanism for Porphyromonas gingivalis.
@nl
prefLabel
Local chemokine paralysis, a novel pathogenic mechanism for Porphyromonas gingivalis.
@en
Local chemokine paralysis, a novel pathogenic mechanism for Porphyromonas gingivalis.
@nl
P2093
P2860
P1476
Local chemokine paralysis, a novel pathogenic mechanism for Porphyromonas gingivalis
@en
P2093
C M Belton
R J Lamont
R P Darveau
P2860
P304
P407
P577
1998-04-01T00:00:00Z