Herpes simplex virus gE/gI sorts nascent virions to epithelial cell junctions, promoting virus spread.
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Cellular localization of nectin-1 and glycoprotein D during herpes simplex virus infectionMacrophages and cytokines in the early defence against herpes simplex virusHerpes simplex virus dances with amyloid precursor protein while exiting the cellHerpes Simplex Virus Type 2 Glycoprotein G Is Targeted by the Sulfated Oligo- and Polysaccharide Inhibitors of Virus Attachment to CellsReplication of herpes simplex virus: egress of progeny virus at specialized cell membrane sites.Reconstitution of herpes simplex virus microtubule-dependent trafficking in vitro.Herpes simplex virus membrane proteins gE/gI and US9 act cooperatively to promote transport of capsids and glycoproteins from neuron cell bodies into initial axon segments.Polarized cell migration during cell-to-cell transmission of herpes simplex virus in human skin keratinocytes.Canine distemper virus persistence in demyelinating encephalitis by swift intracellular cell-to-cell spread in astrocytes is controlled by the viral attachment protein.Herpes simplex virus type 1 enters human epidermal keratinocytes, but not neurons, via a pH-dependent endocytic pathway.Herpes simplex virus with highly reduced gD levels can efficiently enter and spread between human keratinocytes.Cytoplasmic domain of herpes simplex virus gE causes accumulation in the trans-Golgi network, a site of virus envelopment and sorting of virions to cell junctions.Efficient axonal localization of alphaherpesvirus structural proteins in cultured sympathetic neurons requires viral glycoprotein E.The extracellular domain of herpes simplex virus gE is indispensable for efficient cell-to-cell spread: evidence for gE/gI receptorsCell-to-cell movement and assembly of a plant closterovirus: roles for the capsid proteins and Hsp70 homologHerpes simplex virus gE/gI extracellular domains promote axonal transport and spread from neurons to epithelial cells.Human cytomegalovirus entry into epithelial and endothelial cells depends on genes UL128 to UL150 and occurs by endocytosis and low-pH fusion.Directed egress of animal viruses promotes cell-to-cell spreadHerpes simplex virus type 1 evades the effects of antibody and complement in vivoPhosphorylation by the varicella-zoster virus ORF47 protein serine kinase determines whether endocytosed viral gE traffics to the trans-Golgi network or recycles to the cell membrane.Human cytomegalovirus US7, US8, US9, and US10 are cytoplasmic glycoproteins, not found at cell surfaces, and US9 does not mediate cell-to-cell spread.Rho-ROCK and Rac-PAK signaling pathways have opposing effects on the cell-to-cell spread of Marek's Disease VirusProspects and perspectives for development of a vaccine against herpes simplex virus infections.Comparison of virus transcription during lytic infection of the Oka parental and vaccine strains of Varicella-Zoster virusHerpesvirus assembly and egressHerpes simplex virus gE/gI must accumulate in the trans-Golgi network at early times and then redistribute to cell junctions to promote cell-cell spread.Physical interaction between envelope glycoproteins E and M of pseudorabies virus and the major tegument protein UL49.Expression and characterization of duck enteritis virus gI geneHerpes simplex virus glycoproteins gB and gD function in a redundant fashion to promote secondary envelopmentThe unusual fold of herpes simplex virus 1 UL21, a multifunctional tegument protein.Essential functions of the unique N-terminal region of the varicella-zoster virus glycoprotein E ectodomain in viral replication and in the pathogenesis of skin infectionHerpes simplex virus glycoproteins gD and gE/gI serve essential but redundant functions during acquisition of the virion envelope in the cytoplasm.Direct and specific binding of the UL16 tegument protein of herpes simplex virus to the cytoplasmic tail of glycoprotein E.Herpesvirus entry: an updateEvasion of early antiviral responses by herpes simplex viruses.Non-redundant and redundant roles of cytomegalovirus gH/gL complexes in host organ entry and intra-tissue spread.CD2v Interacts with Adaptor Protein AP-1 during African Swine Fever Infection.Cytoplasmic residues of herpes simplex virus glycoprotein gE required for secondary envelopment and binding of tegument proteins VP22 and UL11 to gE and gDBcl-2 blocks accretion or depletion of stored calcium but has no effect on the redistribution of IP3 receptor I mediated by glycoprotein E of herpes simplex virus 1.Virulence genes in herpes simplex virus type 1 corneal infection.
P2860
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P2860
Herpes simplex virus gE/gI sorts nascent virions to epithelial cell junctions, promoting virus spread.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
2001年论文
@zh
2001年论文
@zh-cn
name
Herpes simplex virus gE/gI sor ...... tions, promoting virus spread.
@en
Herpes simplex virus gE/gI sor ...... tions, promoting virus spread.
@nl
type
label
Herpes simplex virus gE/gI sor ...... tions, promoting virus spread.
@en
Herpes simplex virus gE/gI sor ...... tions, promoting virus spread.
@nl
prefLabel
Herpes simplex virus gE/gI sor ...... tions, promoting virus spread.
@en
Herpes simplex virus gE/gI sor ...... tions, promoting virus spread.
@nl
P2093
P2860
P1433
P1476
Herpes simplex virus gE/gI sor ...... tions, promoting virus spread.
@en
P2093
P2860
P304
P356
10.1128/JVI.75.2.821-833.2001
P407
P577
2001-01-01T00:00:00Z