Adenovirus exploits the cellular aggresome response to accelerate inactivation of the MRN complex.
about
Valosin-containing protein (VCP/p97) is required for poliovirus replication and is involved in cellular protein secretion pathway in poliovirus infectionThe DNA damage response induced by infection with human cytomegalovirus and other virusesRecruitment of the oncoprotein v-ErbA to aggresomes.An intrinsically disordered region of the acetyltransferase p300 with similarity to prion-like domains plays a role in aggregationAdenovirus Early Proteins and Host SumoylationAdenovirus type 5 early region 1B 55-kDa oncoprotein can promote cell transformation by a mechanism independent from blocking p53-activated transcriptionLocalization and importance of the adenovirus E4orf4 protein during lytic infectionHerpes simplex virus type I disrupts the ATR-dependent DNA-damage response during lytic infection.Mislocalization of the MRN complex prevents ATR signaling during adenovirus infection.Deletion of the B-B' and C-C' regions of inverted terminal repeats reduces rAAV productivity but increases transgene expressionAdenovirus 12 E4orf6 inhibits ATR activation by promoting TOPBP1 degradationAdenovirus E1B 55-kilodalton protein is a p53-SUMO1 E3 ligase that represses p53 and stimulates its nuclear export through interactions with promyelocytic leukemia nuclear bodies.The adenovirus E4 11 k protein binds and relocalizes the cytoplasmic P-body component Ddx6 to aggresomes.Adenovirus replaces mitotic checkpoint controls.Adenovirus ubiquitin-protein ligase stimulates viral late mRNA nuclear export.Intranuclear targeting and nuclear export of the adenovirus E1B-55K protein are regulated by SUMO1 conjugation.Timely synthesis of the adenovirus type 5 E1B 55-kilodalton protein is required for efficient genome replication in normal human cells.What's the damage? The impact of pathogens on pathways that maintain host genome integrityChanging the ubiquitin landscape during viral manipulation of the DNA damage response.Role of the RNA recognition motif of the E1B 55 kDa protein in the adenovirus type 5 infectious cycle.Biophysical and functional analyses suggest that adenovirus E4-ORF3 protein requires higher-order multimerization to function against promyelocytic leukemia protein nuclear bodiesCharacterization of aggregate/aggresome structures formed by polyhedrin of Bombyx mori nucleopolyhedrovirusControl of mRNA export by adenovirus E4orf6 and E1B55K proteins during productive infection requires E4orf6 ubiquitin ligase activityRUNX1 permits E4orf6-directed nuclear localization of the adenovirus E1B-55K protein and associates with centers of viral DNA and RNA synthesisAggresome formation by the adenoviral protein E1B55K is not conserved among adenovirus species and is not required for efficient degradation of nuclear substratesIdentification of integrin alpha3 as a new substrate of the adenovirus E4orf6/E1B 55-kilodalton E3 ubiquitin ligase complexWidespread phosphorylation of histone H2AX by species C adenovirus infection requires viral DNA replication.Viral and Cellular Components of AAV2 Replication Compartments.Viral manipulation of DNA repair and cell cycle checkpointsAtaxia telangiectasia-mutated-Rad3-related DNA damage checkpoint signaling pathway triggered by hepatitis B virus infection.Adenovirus E1B 55-kilodalton protein: multiple roles in viral infection and cell transformation.Efficient Transformation of Primary Human Mesenchymal Stromal Cells by Adenovirus Early Region 1 Oncogenes.The critical protein interactions and structures that elicit growth deregulation in cancer and viral replication.Design stars: how small DNA viruses remodel the host nucleus.Viral manipulation of cellular protein conjugation pathways: The SUMO lesson.Ataxia telangiectasia-mutated damage-signaling kinase- and proteasome-dependent destruction of Mre11-Rad50-Nbs1 subunits in Simian virus 40-infected primate cellsCombining Oncolytic Adenovirus with Radiation-A Paradigm for the Future of Radiosensitization.Localization of Double-Strand Break Repair Proteins to Viral Replication Compartments following Lytic Reactivation of Kaposi's Sarcoma-Associated Herpesvirus.Comparison of protein expression during wild-type, and E1B-55k-deletion, adenovirus infection using quantitative time-course proteomics.Role of E1B55K in E4orf6/E1B55K E3 ligase complexes formed by different human adenovirus serotypes.
P2860
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P2860
Adenovirus exploits the cellular aggresome response to accelerate inactivation of the MRN complex.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Adenovirus exploits the cellul ...... activation of the MRN complex.
@en
Adenovirus exploits the cellul ...... activation of the MRN complex.
@nl
type
label
Adenovirus exploits the cellul ...... activation of the MRN complex.
@en
Adenovirus exploits the cellul ...... activation of the MRN complex.
@nl
prefLabel
Adenovirus exploits the cellul ...... activation of the MRN complex.
@en
Adenovirus exploits the cellul ...... activation of the MRN complex.
@nl
P2093
P2860
P1433
P1476
Adenovirus exploits the cellul ...... activation of the MRN complex.
@en
P2093
Andrej Shevchenko
Anna Shevchenko
Arnold J Berk
P2860
P304
14004-14016
P356
10.1128/JVI.79.22.14004-14016.2005
P407
P577
2005-11-01T00:00:00Z