Microbial biotransformation of DON: molecular basis for reduced toxicity.
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Co-exposure to low doses of the food contaminants deoxynivalenol and nivalenol has a synergistic inflammatory effect on intestinal explants.Mycotoxin Biotransformation by Native and Commercial Enzymes: Present and Future PerspectivesStrategies and Methodologies for Developing Microbial Detoxification Systems to Mitigate MycotoxinsAerobic De-Epoxydation of Trichothecene Mycotoxins by a Soil Bacterial Consortium Isolated Using In Situ Soil Enrichment.The Food Contaminant Deoxynivalenol Exacerbates the Genotoxicity of Gut Microbiota.Intestinal toxicity of the type B trichothecene mycotoxin fusarenon-X: whole transcriptome profiling reveals new signaling pathwaysDeoxynivalenol and its metabolite deepoxy-deoxynivalenol: multi-parameter analysis for the evaluation of cytotoxicity and cellular effects.Impact of mycotoxins on the intestine: are mucus and microbiota new targets?Identification of Signaling Pathways Targeted by the Food Contaminant FB1: Transcriptome and Kinome Analysis of Samples from Pig Liver and Intestine.An aldo-keto reductase is responsible for Fusarium toxin-degrading activity in a soil Sphingomonas strain.Beyond Ribosomal Binding: The Increased Polarity and Aberrant Molecular Interactions of 3-epi-deoxynivalenol.Early Activation of MAPK p44/42 Is Partially Involved in DON-Induced Disruption of the Intestinal Barrier Function and Tight Junction Network.The enzymatic epimerization of deoxynivalenol by Devosia mutans proceeds through the formation of 3-keto-DON intermediate.Microbial Detoxification of Deoxynivalenol (DON), Assessed via a Lemna minor L. Bioassay, through Biotransformation to 3-epi-DON and 3-epi-DOM-1.Microbial Inhibition of Fusarium Pathogens and Biological Modification of Trichothecenes in Cereal Grains.Risks to human and animal health related to the presence of deoxynivalenol and its acetylated and modified forms in food and feedTrichothecenes: immunomodulatory effects, mechanisms, and anti-cancer potential.The mycotoxin metabolite deepoxy- deoxynivalenol increases apoptosis and decreases steroidogenesis in bovine ovarian theca cells.Toxicodynamics of Mycotoxins in the Framework of Food Risk Assessment-An In Silico Perspective.Intestinal toxicity of deoxynivalenol is limited by Lactobacillus rhamnosus RC007 in pig jejunum explants.Porcine Small and Large Intestinal Microbiota Rapidly Hydrolyze the Masked Mycotoxin Deoxynivalenol-3-Glucoside and Release Deoxynivalenol in Spiked Batch Cultures In Vitro.Bovine Peripheral Blood Mononuclear Cells Are More Sensitive to Deoxynivalenol Than Those Derived from Poultry and Swine.Impact of two mycotoxins deoxynivalenol and fumonisin on pig intestinal health.Saccharomyces cerevisiae Boulardii Reduces the Deoxynivalenol-Induced Alteration of the Intestinal Transcriptome.Ergot Alkaloids at Doses Close to EU Regulatory Limits Induce Alterations of the Liver and Intestine.
P2860
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P2860
Microbial biotransformation of DON: molecular basis for reduced toxicity.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Microbial biotransformation of DON: molecular basis for reduced toxicity.
@en
Microbial biotransformation of DON: molecular basis for reduced toxicity.
@nl
type
label
Microbial biotransformation of DON: molecular basis for reduced toxicity.
@en
Microbial biotransformation of DON: molecular basis for reduced toxicity.
@nl
prefLabel
Microbial biotransformation of DON: molecular basis for reduced toxicity.
@en
Microbial biotransformation of DON: molecular basis for reduced toxicity.
@nl
P2093
P2860
P50
P356
P1433
P1476
Microbial biotransformation of DON: molecular basis for reduced toxicity.
@en
P2093
Alix Pierron
Ana-Paula F L Bracarense
Gerd Schatzmayr
Jian Wei He
Leticia S Murate
Sabria Mimoun
P2860
P2888
P356
10.1038/SREP29105
P407
P577
2016-07-06T00:00:00Z
P6179
1002316510