Discovery of TBC1D1 as an insulin-, AICAR-, and contraction-stimulated signaling nexus in mouse skeletal muscle.
about
Regulation and function of AMPK in physiology and diseasesInsulin receptor signaling in normal and insulin-resistant statesAMPK as a potential anticancer target - friend or foe?General aspects of muscle glucose uptakeExercise and Regulation of Carbohydrate MetabolismContraction regulates site-specific phosphorylation of TBC1D1 in skeletal muscleMuscle cells engage Rab8A and myosin Vb in insulin-dependent GLUT4 translocationGenetic disruption of AMPK signaling abolishes both contraction- and insulin-stimulated TBC1D1 phosphorylation and 14-3-3 binding in mouse skeletal muscleInsulin-stimulated phosphorylation of the Rab GTPase-activating protein TBC1D1 regulates GLUT4 translocationRab GAPs AS160 and Tbc1d1 play nonredundant roles in the regulation of glucose and energy homeostasis in miceDeletion of the Rab GAP Tbc1d1 modifies glucose, lipid, and energy homeostasis in miceExercise and type 2 diabetes: molecular mechanisms regulating glucose uptake in skeletal muscle.Deletion of Rab GAP AS160 modifies glucose uptake and GLUT4 translocation in primary skeletal muscles and adipocytes and impairs glucose homeostasis.AMPK alpha1 activation is required for stimulation of glucose uptake by twitch contraction, but not by H2O2, in mouse skeletal muscleExercise and Glycemic Control: Focus on Redox Homeostasis and Redox-Sensitive Protein Signaling.Identification of a novel phosphorylation site on TBC1D4 regulated by AMP-activated protein kinase in skeletal muscleEffects of excess corticosterone on LKB1 and AMPK signaling in rat skeletal muscle.In vivo exercise followed by in vitro contraction additively elevates subsequent insulin-stimulated glucose transport by rat skeletal muscle.Insulin signaling, resistance, and the metabolic syndrome: insights from mouse models into disease mechanisms.TBC1D1 regulates insulin- and contraction-induced glucose transport in mouse skeletal muscle.Role of ataxia telangiectasia mutated in insulin signalling of muscle-derived cell lines and mouse soleus.A link between hepatic glucose production and peripheral energy metabolism via hepatokinesBeneficial effects of resistance exercise on glycemic control are not further improved by protein ingestionMetabolic syndrome and insulin resistance: underlying causes and modification by exercise training.Leptin reduces the expression and increases the phosphorylation of the negative regulators of GLUT4 traffic TBC1D1 and TBC1D4 in muscle of ob/ob mice.Overexpression of TRB3 in muscle alters muscle fiber type and improves exercise capacity in mice.EMG-normalised kinase activation during exercise is higher in human gastrocnemius compared to soleus muscle.Electrical vs manual acupuncture stimulation in a rat model of polycystic ovary syndrome: different effects on muscle and fat tissue insulin signaling.Myo1c regulates glucose uptake in mouse skeletal muscle.Roles of TBC1D1 and TBC1D4 in insulin- and exercise-stimulated glucose transport of skeletal muscleNonalcoholic fatty liver disease: molecular pathways and therapeutic strategiesStructural and population-based evaluations of TBC1D1 p.Arg125TrpEmerging role for AS160/TBC1D4 and TBC1D1 in the regulation of GLUT4 trafficMice with AS160/TBC1D4-Thr649Ala knockin mutation are glucose intolerant with reduced insulin sensitivity and altered GLUT4 trafficking.Exercise increases TBC1D1 phosphorylation in human skeletal muscle.A Potent and Selective AMPK Activator That Inhibits de Novo Lipogenesis.Clustering of GLUT4, TUG, and RUVBL2 protein levels correlate with myosin heavy chain isoform pattern in skeletal muscles, but AS160 and TBC1D1 levels do notDifferential Role of Insulin/IGF-1 Receptor Signaling in Muscle Growth and Glucose HomeostasisProtein kinase D1 is essential for contraction-induced glucose uptake but is not involved in fatty acid uptake into cardiomyocytes.Interactions between mitochondrial reactive oxygen species and cellular glucose metabolism.
P2860
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P2860
Discovery of TBC1D1 as an insulin-, AICAR-, and contraction-stimulated signaling nexus in mouse skeletal muscle.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Discovery of TBC1D1 as an insu ...... exus in mouse skeletal muscle.
@en
Discovery of TBC1D1 as an insu ...... exus in mouse skeletal muscle.
@nl
type
label
Discovery of TBC1D1 as an insu ...... exus in mouse skeletal muscle.
@en
Discovery of TBC1D1 as an insu ...... exus in mouse skeletal muscle.
@nl
prefLabel
Discovery of TBC1D1 as an insu ...... exus in mouse skeletal muscle.
@en
Discovery of TBC1D1 as an insu ...... exus in mouse skeletal muscle.
@nl
P2093
P2860
P356
P1476
Discovery of TBC1D1 as an insu ...... nexus in mouse skeletal muscle
@en
P2093
Edward P Feener
Henning F Kramer
Jianxin Xie
Katja S C Roeckl
Laurie J Goodyear
Michael F Hirshman
Nicole Bowles
Nobuharu L Fujii
P2860
P304
P356
10.1074/JBC.M708839200
P407
P577
2008-02-13T00:00:00Z