Differential expression of tetrodotoxin-resistant sodium channels Nav1.8 and Nav1.9 in normal and inflamed rats.
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Conotoxins That Could Provide Analgesia through Voltage Gated Sodium Channel InhibitionTrafficking regulates the subcellular distribution of voltage-gated sodium channels in primary sensory neuronsNav1.9 channel contributes to mechanical and heat pain hypersensitivity induced by subacute and chronic inflammation.Pain related channels are differentially expressed in neuronal and non-neuronal cells of glabrous skin of fabry knockout male miceA-803467, a potent and selective Nav1.8 sodium channel blocker, attenuates neuropathic and inflammatory pain in the rat.Are voltage-gated sodium channels on the dorsal root ganglion involved in the development of neuropathic pain?muO-conotoxin MrVIB selectively blocks Nav1.8 sensory neuron specific sodium channels and chronic pain behavior without motor deficitsBeyond faithful conduction: short-term dynamics, neuromodulation, and long-term regulation of spike propagation in the axon.Site-specific mesenchymal control of inflammatory pain to yeast challenge in vulvodynia-afflicted and pain-free womenA novel selective and orally bioavailable Nav 1.8 channel blocker, PF-01247324, attenuates nociception and sensory neuron excitability.Betaine inhibits vascularization via suppression of Akt in the retinas of streptozotocin-induced hyperglycemic ratsA novel substituted aminoquinoline selectively targets voltage-sensitive sodium channel isoforms and NMDA receptor subtypes and alleviates chronic inflammatory and neuropathic pain.Antinociceptive activities of lidocaine and the nav1.8 blocker a803467 in diabetic rats.Post-translational modifications of voltage-gated sodium channels in chronic pain syndromesEfficacy of Anti-NaV1.7 Antibody on the Sensory Nervous System in a Rat Model of Lumbar Intervertebral Disc Injury.Protein kinase B/Akt is required for complete Freund's adjuvant-induced upregulation of Nav1.7 and Nav1.8 in primary sensory neurons.Sodium channel diversity in the vestibular ganglion: NaV1.5, NaV1.8, and tetrodotoxin-sensitive currents.CXCL13/CXCR5 enhances sodium channel Nav1.8 current density via p38 MAP kinase in primary sensory neurons following inflammatory painEvaluation of behavior and expression of NaV1.7 in dorsal root ganglia after sciatic nerve compression and application of nucleus pulposus in rats.Neurophysiology of arthritis pain.Functional up-regulation of Nav1.8 sodium channel in Aβ afferent fibers subjected to chronic peripheral inflammation.Osteoarthritis-dependent changes in antinociceptive action of Nav1.7 and Nav1.8 sodium channel blockers: An in vivo electrophysiological study in the rat.Regulation of the spontaneous augmentation of Na(V)1.9 in mouse dorsal root ganglion neurons: effect of PKA and PKC pathways.Inflammatory Pain Reduces C Fiber Activity-Dependent Slowing in a Sex-Dependent Manner, Amplifying Nociceptive Input to the Spinal Cord.Ambroxol for the treatment of fibromyalgia: science or fiction?Involvement of Nav 1.8 sodium ion channels in the transduction of mechanical pain in a rodent model of osteoarthritis.Antinociceptive effect of ambroxol in rats with neuropathic spinal cord injury pain.Nociceptors as chronic drivers of pain and hyperreflexia after spinal cord injury: an adaptive-maladaptive hyperfunctional state hypothesis.Tetrodotoxin-resistant fibres and spinal Fos expression: differences between input from muscle and skin.Influence of a chronic myositis on rat spinal field potentials evoked by TTX-resistant unmyelinated skin and muscle afferents.Phenotypic changes of morphologically identified guinea-pig myenteric neurons following intestinal inflammation.Defining a holistic pain-relieving approach to wound care via a drug free polymeric membrane dressing.Dissecting the role of sodium currents in visceral sensory neurons in a model of chronic hyperexcitability using Nav1.8 and Nav1.9 null mice.NaV 1.8, but not NaV 1.9, is upregulated in the inflamed dental pulp tissue of human primary teeth.
P2860
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P2860
Differential expression of tetrodotoxin-resistant sodium channels Nav1.8 and Nav1.9 in normal and inflamed rats.
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2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Differential expression of tet ...... 9 in normal and inflamed rats.
@en
Differential expression of tet ...... 9 in normal and inflamed rats.
@nl
type
label
Differential expression of tet ...... 9 in normal and inflamed rats.
@en
Differential expression of tet ...... 9 in normal and inflamed rats.
@nl
prefLabel
Differential expression of tet ...... 9 in normal and inflamed rats.
@en
Differential expression of tet ...... 9 in normal and inflamed rats.
@nl
P2093
P1433
P1476
Differential expression of tet ...... 9 in normal and inflamed rats.
@en
P2093
Richard E Coggeshall
Simon Tate
Susan M Carlton
P356
10.1016/J.NEULET.2003.10.023
P407
P577
2004-01-01T00:00:00Z