Kinetic analysis of the effects of H+ or Ni2+ on Kv1.5 current shows that both ions enhance slow inactivation and induce resting inactivation.
about
SV31 is a Zn2+-binding synaptic vesicle proteinCa(V)3.2 channels and the induction of negative feedback in cerebral arteries.Genetic ablation of CaV3.2 channels enhances the arterial myogenic response by modulating the RyR-BKCa axis.Nickel inhibits β-1 adrenoceptor mediated activation of cardiac CFTR chloride channels.Molecular determinants of U-type inactivation in Kv2.1 channels.External Ba(2+) block of Kv4.2 channels is enhanced in the closed-inactivated state.ShakerIR and Kv1.5 mutant channels with enhanced slow inactivation also exhibit K⁺ o-dependent resting inactivation.Proton block of the pore underlies the inhibition of hERG cardiac K+ channels during acidosis.Regulation of human cardiac Kv1.5 channels by extracellular acidification.
P2860
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P2860
Kinetic analysis of the effects of H+ or Ni2+ on Kv1.5 current shows that both ions enhance slow inactivation and induce resting inactivation.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
Kinetic analysis of the effect ...... d induce resting inactivation.
@en
Kinetic analysis of the effect ...... d induce resting inactivation.
@nl
type
label
Kinetic analysis of the effect ...... d induce resting inactivation.
@en
Kinetic analysis of the effect ...... d induce resting inactivation.
@nl
prefLabel
Kinetic analysis of the effect ...... d induce resting inactivation.
@en
Kinetic analysis of the effect ...... d induce resting inactivation.
@nl
P2860
P1476
Kinetic analysis of the effect ...... nd induce resting inactivation
@en
P2093
David Fedida
Yen May Cheng
P2860
P304
P356
10.1113/JPHYSIOL.2010.191544
P407
P577
2010-06-25T00:00:00Z