about
Splenic red pulp macrophages produce type I interferons as early sentinels of malaria infection but are dispensable for controlThe role of platelet MyD88 in host response during gram-negative sepsis.Toll-like receptors activate innate and adaptive immunity by using dendritic cell-intrinsic and -extrinsic mechanismsRequirement for MyD88 signaling in B cells and dendritic cells for germinal center anti-nuclear antibody production in Lyn-deficient miceCritical coordination of innate immune defense against Toxoplasma gondii by dendritic cells responding via their Toll-like receptorsPolysaccharide from Lentinus edodes inhibits the immunosuppressive function of myeloid-derived suppressor cells.Antiviral memory CD8 T-cell differentiation, maintenance, and secondary expansion occur independently of MyD88.B cell-intrinsic MyD88 signaling prevents the lethal dissemination of commensal bacteria during colonic damage.Toll-Like Receptor Signalling Is Not Involved in Platelet Response to Streptococcus pneumoniae In Vitro or In Vivo.Expression of A20 by dendritic cells preserves immune homeostasis and prevents colitis and spondyloarthritis.B cell IFN-γ receptor signaling promotes autoimmune germinal centers via cell-intrinsic induction of BCL-6.TLR signaling in B-cell development and activation.Tumor necrosis factor alpha activates the human plasminogen activator inhibitor-1 gene through a distal nuclear factor kappaB site.Lung epithelial MyD88 drives early pulmonary clearance of Pseudomonas aeruginosa by a flagellin dependent mechanism.Characterization of T-Dependent and T-Independent B Cell Responses to a Virus-like Particle.Epithelial Myeloid-Differentiation Factor 88 Is Dispensable during Klebsiella Pneumonia.General, but not myeloid or type II lung epithelial cell, myeloid differentiation factor 88 deficiency abrogates house dust mite induced allergic lung inflammation.Selective utilization of Toll-like receptor and MyD88 signaling in B cells for enhancement of the antiviral germinal center response.Normal development and activation but altered cytokine production of Fyn-deficient CD4+ T cells.B Cell-Intrinsic MyD88 Signaling Promotes Initial Cell Proliferation and Differentiation To Enhance the Germinal Center Response to a Virus-like Particle.Functional Characterization of CD11c+ Age-Associated B Cells as Memory B CellsSerine Phosphorylation of the STAT1 Transactivation Domain Promotes Autoreactive B Cell and Systemic Autoimmunity DevelopmentThe role of B cell antigen presentation in the initiation of CD4+ T cell response
P50
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P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Baidong Hou
@ast
Baidong Hou
@en
Baidong Hou
@es
Baidong Hou
@nl
Baidong Hou
@sl
type
label
Baidong Hou
@ast
Baidong Hou
@en
Baidong Hou
@es
Baidong Hou
@nl
Baidong Hou
@sl
prefLabel
Baidong Hou
@ast
Baidong Hou
@en
Baidong Hou
@es
Baidong Hou
@nl
Baidong Hou
@sl
P106
P31
P496
0000-0002-0892-0181