Nonpeptidic lysosomal modulators derived from z-phe-ala-diazomethylketone for treating protein accumulation diseases.
about
Cathepsin B degrades amyloid-β in mice expressing wild-type human amyloid precursor protein.Prion degradation pathways: Potential for therapeutic intervention.Aβ42-mediated proteasome inhibition and associated tau pathology in hippocampus are governed by a lysosomal response involving cathepsin B: Evidence for protective crosstalk between protein clearance pathways.Potential Alzheimer's Disease Therapeutics Among Weak Cysteine Protease Inhibitors Exhibit Mechanistic Differences Regarding Extent of Cathepsin B Up-Regulation and Ability to Block Calpain.
P2860
Nonpeptidic lysosomal modulators derived from z-phe-ala-diazomethylketone for treating protein accumulation diseases.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
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2012年學術文章
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2012年學術文章
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name
Nonpeptidic lysosomal modulato ...... protein accumulation diseases.
@en
Nonpeptidic lysosomal modulato ...... protein accumulation diseases.
@nl
type
label
Nonpeptidic lysosomal modulato ...... protein accumulation diseases.
@en
Nonpeptidic lysosomal modulato ...... protein accumulation diseases.
@nl
prefLabel
Nonpeptidic lysosomal modulato ...... protein accumulation diseases.
@en
Nonpeptidic lysosomal modulato ...... protein accumulation diseases.
@nl
P2093
P2860
P356
P1476
Nonpeptidic lysosomal modulato ...... protein accumulation diseases
@en
P2093
Ben A Bahr
Dennis J Hoover
Jeannie Hwang
Kishore Viswanathan
Meagan L Wisniewski
Uzoma S Ikonne
P2860
P304
P356
10.1021/ML300197H
P577
2012-09-09T00:00:00Z