Pressure-independent cardiac hypertrophy in mice with cardiomyocyte-restricted inactivation of the atrial natriuretic peptide receptor guanylyl cyclase-A.
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A friend within the heart: natriuretic peptide receptor signalingSildenafil citrate increases myocardial cGMP content in rat heart, decreases its hypertrophic response to isoproterenol and decreases myocardial leak of creatine kinase and troponin TNatriuretic Peptides in the Regulation of Cardiovascular Physiology and Metabolic EventsNatriuretic Peptides and Cardiometabolic HealthPhosphodiesterases and cardiac cGMP: evolving roles and controversiesAtrial natriuretic peptide in cardiovascular biology and disease (NPPA)Identification of an orphan guanylate cyclase receptor selectively expressed in mouse testisHearts of dystonia musculorum mice display normal morphological and histological features but show signs of cardiac stressHypertension in mice lacking the proatrial natriuretic peptide convertase corinAlternative splicing of the guanylyl cyclase-A receptor modulates atrial natriuretic peptide signalingCentral role of guanylyl cyclase in natriuretic peptide signaling in hypertension and metabolic syndrome.Stress-dependent dilated cardiomyopathy in mice with cardiomyocyte-restricted inactivation of cyclic GMP-dependent protein kinase INatriuretic peptides and the genomics of left-ventricular hypertrophy.Brain natriuretic peptide in pulmonary arterial hypertension: biomarker and potential therapeutic agentA human atrial natriuretic peptide gene mutation reveals a novel peptide with enhanced blood pressure-lowering, renal-enhancing, and aldosterone-suppressing actions.Role of Ca2+/calmodulin-stimulated cyclic nucleotide phosphodiesterase 1 in mediating cardiomyocyte hypertrophy.Natriuretic Peptide Signaling via Guanylyl Cyclase (GC)-A: An Endogenous Protective Mechanism of the Heart.Cardiac hypertrophy is not amplified by deletion of cGMP-dependent protein kinase I in cardiomyocytes.Cardiomyocyte-specific Estrogen Receptor Alpha Increases Angiogenesis, Lymphangiogenesis and Reduces Fibrosis in the Female Mouse Heart Post-Myocardial Infarction.Vascular endothelium is critically involved in the hypotensive and hypovolemic actions of atrial natriuretic peptidePlasma soluble corin in patients with heart failure.Novel insights into the mechanisms mediating the local antihypertrophic effects of cardiac atrial natriuretic peptide: role of cGMP-dependent protein kinase and RGS2.Class IA phosphoinositide 3-kinase regulates heart size and physiological cardiac hypertrophy.Cardiomyocyte-specific deletion of the vitamin D receptor gene results in cardiac hypertrophyBNP controls early load-dependent regulation of SERCA through calcineurin.Salt-sensitive hypertension and cardiac hypertrophy in transgenic mice expressing a corin variant identified in blacksSildenafil stops progressive chamber, cellular, and molecular remodeling and improves calcium handling and function in hearts with pre-existing advanced hypertrophy caused by pressure overload.Protein kinase G I and heart failure: Shifting focus from vascular unloading to direct myocardial antiremodeling effects.Identification of temporal and region-specific myocardial gene expression patterns in response to infarction in swine.Pressure overload-induced cardiac hypertrophy response requires janus kinase 2-histone deacetylase 2 signaling.Corin variant associated with hypertension and cardiac hypertrophy exhibits impaired zymogen activation and natriuretic peptide processing activityNatriuretic peptides: their structures, receptors, physiologic functions and therapeutic applicationsIncreased effects of C-type natriuretic peptide on contractility and calcium regulation in murine hearts overexpressing cyclic GMP-dependent protein kinase I.Regulated inositol-requiring protein 1-dependent decay as a mechanism of corin RNA and protein deficiency in advanced human systolic heart failure.A cardiac pathway of cyclic GMP-independent signaling of guanylyl cyclase A, the receptor for atrial natriuretic peptideIQGAP1 regulates ERK1/2 and AKT signalling in the heart and sustains functional remodelling upon pressure overload.Guanylyl cyclase / atrial natriuretic peptide receptor-A: role in the pathophysiology of cardiovascular regulation.Propranolol causes a paradoxical enhancement of cardiomyocyte foetal gene response to hypertrophic stimuli.Cardioprotective actions of cyclic GMP: lessons from genetic animal models.Pathological cardiac hypertrophy alters intracellular targeting of phosphodiesterase type 5 from nitric oxide synthase-3 to natriuretic peptide signaling.
P2860
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P2860
Pressure-independent cardiac hypertrophy in mice with cardiomyocyte-restricted inactivation of the atrial natriuretic peptide receptor guanylyl cyclase-A.
description
2003 nî lūn-bûn
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2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
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name
Pressure-independent cardiac h ...... e receptor guanylyl cyclase-A.
@en
Pressure-independent cardiac h ...... e receptor guanylyl cyclase-A.
@nl
type
label
Pressure-independent cardiac h ...... e receptor guanylyl cyclase-A.
@en
Pressure-independent cardiac h ...... e receptor guanylyl cyclase-A.
@nl
prefLabel
Pressure-independent cardiac h ...... e receptor guanylyl cyclase-A.
@en
Pressure-independent cardiac h ...... e receptor guanylyl cyclase-A.
@nl
P2093
P2860
P1476
Pressure-independent cardiac h ...... de receptor guanylyl cyclase-A
@en
P2093
Alexander Bubikat
Boris V Skryabin
David L Garbers
Frank Begrow
Hideo A Baba
Martin van Eickels
Michaela Kuhn
Rita Holtwick
P2860
P304
P356
10.1172/JCI17061
10.1172/JCI200317061
P407
P577
2003-05-01T00:00:00Z