Prolyl hydroxylase inhibition attenuates post-ischemic cardiac injury via induction of endoplasmic reticulum stress genes.
about
HIF hydroxylase pathways in cardiovascular physiology and medicineImpact of the phosphatidylinositide 3-kinase signaling pathway on the cardioprotection induced by intermittent hypoxiaDownregulation of miR-205 modulates cell susceptibility to oxidative and endoplasmic reticulum stresses in renal tubular cells.Fine tuning of the UPR by the ubiquitin ligases Siah1/2.Sex differences in the acute in vivo effects of different human SP-A variants on the mouse alveolar macrophage proteome.Atorvastatin ameliorates myocardial ischemia/reperfusion injury through attenuation of endoplasmic reticulum stress-induced apoptosis.In vivo rescue of alveolar macrophages from SP-A knockout mice with exogenous SP-A nearly restores a wild type intracellular proteome; actin involvement.Hydrogen sulfide preconditioning protects against myocardial ischemia/reperfusion injury in rats through inhibition of endo/sarcoplasmic reticulum stressSex differences in the response of the alveolar macrophage proteome to treatment with exogenous surfactant protein-A.Characterization of eicosanoid synthesis in a genetic ablation model of ceramide kinaseActivation of NADPH oxidase 4 in the endoplasmic reticulum promotes cardiomyocyte autophagy and survival during energy stress through the protein kinase RNA-activated-like endoplasmic reticulum kinase/eukaryotic initiation factor 2α/activating transHypoxia inducible factor 1 (HIF-1) and cardioprotection.Endoplasmic reticulum and the unfolded protein response: dynamics and metabolic integrationProlyl-hydroxylase inhibition induces SDF-1 associated with increased CXCR4+/CD11b+ subpopulations and cardiac repairEarly upregulation of myocardial CXCR4 expression is critical for dimethyloxalylglycine-induced cardiac improvement in acute myocardial infarction.The integrated stress response.HIF prolyl hydroxylase inhibition prior to transient focal cerebral ischaemia is neuroprotective in mice.Neuroprotection by dimethyloxalylglycine following permanent and transient focal cerebral ischemia in rats.Endoplasmic reticulum stress-related inflammation and cardiovascular diseases.Ω3 Supplementation and intermittent hypobaric hypoxia induce cardioprotection enhancing antioxidant mechanisms in adult rats.Hypoxic preconditioning protects microvascular endothelial cells against hypoxia/reoxygenation injury by attenuating endoplasmic reticulum stress.Role of NADPH oxidase in the regulation of autophagy in cardiomyocytes.Hsp90 Co-chaperone p23 contributes to dopaminergic mitochondrial stress via stabilization of PHD2: Implications for Parkinson's disease.
P2860
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P2860
Prolyl hydroxylase inhibition attenuates post-ischemic cardiac injury via induction of endoplasmic reticulum stress genes.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
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2009年學術文章
@zh-hant
name
Prolyl hydroxylase inhibition ...... lasmic reticulum stress genes.
@en
Prolyl hydroxylase inhibition ...... lasmic reticulum stress genes.
@nl
type
label
Prolyl hydroxylase inhibition ...... lasmic reticulum stress genes.
@en
Prolyl hydroxylase inhibition ...... lasmic reticulum stress genes.
@nl
prefLabel
Prolyl hydroxylase inhibition ...... lasmic reticulum stress genes.
@en
Prolyl hydroxylase inhibition ...... lasmic reticulum stress genes.
@nl
P2093
P1476
Prolyl hydroxylase inhibition ...... plasmic reticulum stress genes
@en
P2093
Alpha A Fowler
Bernard J Fisher
Fadi N Salloum
Jorge Almenara
Lisa Smithson
P304
P356
10.1016/J.VPH.2009.05.007
P577
2009-06-11T00:00:00Z