Mitotic catastrophe is the predominant response to histone acetyltransferase depletion.
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Acetyltransferase p300 regulates NBS1-mediated DNA damage responseATRX ADD domain links an atypical histone methylation recognition mechanism to human mental-retardation syndromeDynamical modelling of phenotypes in a genome-wide RNAi live-cell imaging assayCaspase-3-mediated degradation of condensin Cap-H regulates mitotic cell deathMutation frequency dynamics in HPRT locus in culture-adapted human embryonic stem cells and induced pluripotent stem cells correspond to their differentiated counterpartsPerturbation of mitosis through inhibition of histone acetyltransferases: the key to ochratoxin a toxicity and carcinogenicity?Epigenetic displacement of HP1 from heterochromatin by HIV-1 Vpr causes premature sister chromatid separation.Targeted inhibition of Polo-like kinase 1 by a novel small-molecule inhibitor induces mitotic catastrophe and apoptosis in human bladder cancer cells.Ochratoxin a and mitotic disruption: mode of action analysis of renal tumor formation by ochratoxin A.14-3-3 eta depletion sensitizes glioblastoma cells to irradiation due to enhanced mitotic cell death.Therapeutic potential of mitotic interaction between the nucleoporin Tpr and aurora kinase A.Beta human papillomavirus E6 expression inhibits stabilization of p53 and increases tolerance of genomic instability.Premature chromosome condensation induced by caffeine, 2-aminopurine, staurosporine and sodium metavanadate in S-phase arrested HeLa cells is associated with a decrease in Chk1 phosphorylation, formation of phospho-H2AX and minor cytoskeletal rearraRoles of 14-3-3η in mitotic progression and its potential use as a therapeutic target for cancers.Exposure to reversine affects the chondrocyte morphology and phenotype in vitro.Linking site-specific loss of histone acetylation to repression of gene expression by the mycotoxin ochratoxin A.Chromosomal breaks during mitotic catastrophe trigger γH2AX-ATM-p53-mediated apoptosis.STK3 is a therapeutic target for a subset of acute myeloid leukemias.Cyclizing-berberine A35 induces G2/M arrest and apoptosis by activating YAP phosphorylation (Ser127).CBP and P300 regulate distinct gene networks required for human primary myoblast differentiation and muscle integrity
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P2860
Mitotic catastrophe is the predominant response to histone acetyltransferase depletion.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
Mitotic catastrophe is the predominant response to histone acetyltransferase depletion.
@en
Mitotic catastrophe is the predominant response to histone acetyltransferase depletion.
@nl
type
label
Mitotic catastrophe is the predominant response to histone acetyltransferase depletion.
@en
Mitotic catastrophe is the predominant response to histone acetyltransferase depletion.
@nl
prefLabel
Mitotic catastrophe is the predominant response to histone acetyltransferase depletion.
@en
Mitotic catastrophe is the predominant response to histone acetyltransferase depletion.
@nl
P2093
P2860
P356
P1476
Mitotic catastrophe is the predominant response to histone acetyltransferase depletion.
@en
P2093
P2860
P2888
P304
P356
10.1038/CDD.2008.182
P577
2008-12-19T00:00:00Z
P5875
P6179
1008165033