Transcription factor Fli1 regulates collagen fibrillogenesis in mouse skin.
about
Animal models of systemic sclerosis: their utility and limitations.Thrombocytopenia in mice lacking the carboxy-terminal regulatory domain of the Ets transcription factor Fli1.Endothelial Fli1 deficiency impairs vascular homeostasis: a role in scleroderma vasculopathy.Adenosine A(2A) receptors promote collagen production by a Fli1- and CTGF-mediated mechanism.Decreased interleukin-20 expression in scleroderma skin contributes to cutaneous fibrosisFLI1 expression is correlated with breast cancer cellular growth, migration, and invasion and altered gene expression.Simultaneous downregulation of KLF5 and Fli1 is a key feature underlying systemic sclerosis.Fibrosis, vascular activation, and immune abnormalities resembling systemic sclerosis in bleomycin-treated Fli-1-haploinsufficient miceA role for estrogen receptor-α and estrogen receptor-β in collagen biosynthesis in mouse skin.Update on macrophages and innate immunity in sclerodermaFli1 is a negative regulator of estrogen receptor α in dermal fibroblastsThe impact of Fli1 deficiency on the pathogenesis of systemic sclerosisMicroenvironmental reprogramming by three-dimensional culture enables dermal papilla cells to induce de novo human hair-follicle growthNoncanonical transforming growth factor beta signaling in scleroderma fibrosisCellular and molecular aspects of vascular dysfunction in systemic sclerosis.PAI-1 in tissue fibrosis.Animal models of scleroderma: current state and recent development.Double heterozygous mice for Klf5 and Fli1 genes: a new animal model of systemic sclerosis recapitulating its three cardinal pathological features.Fibrogenesis, novel lessons from animal models.Critical Appraisal of the Utility and Limitations of Animal Models of Scleroderma.Recent advances in animal models of systemic sclerosis.Ciprofloxacin has antifibrotic effects in scleroderma fibroblasts via downregulation of Dnmt1 and upregulation of Fli1.A possible contribution of endothelial CCN1 downregulation due to Fli1 deficiency to the development of digital ulcers in systemic sclerosis.Epithelial Fli1 deficiency drives systemic autoimmunity and fibrosis: Possible roles in scleroderma.Bosentan reverses the pro-fibrotic phenotype of systemic sclerosis dermal fibroblasts via increasing DNA binding ability of transcription factor Fli1.Phosphorylation of Fli1 at threonine 312 by protein kinase C delta promotes its interaction with p300/CREB-binding protein-associated factor and subsequent acetylation in response to transforming growth factor beta.Constitutive activation of c-Abl/protein kinase C-δ/Fli1 pathway in dermal fibroblasts derived from patients with localized scleroderma.Combined analysis of gene expression and genome binding profiles identified potential therapeutic targets of ciclopirox in Ewing sarcoma.
P2860
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P2860
Transcription factor Fli1 regulates collagen fibrillogenesis in mouse skin.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
Transcription factor Fli1 regulates collagen fibrillogenesis in mouse skin.
@en
Transcription factor Fli1 regulates collagen fibrillogenesis in mouse skin.
@nl
type
label
Transcription factor Fli1 regulates collagen fibrillogenesis in mouse skin.
@en
Transcription factor Fli1 regulates collagen fibrillogenesis in mouse skin.
@nl
prefLabel
Transcription factor Fli1 regulates collagen fibrillogenesis in mouse skin.
@en
Transcription factor Fli1 regulates collagen fibrillogenesis in mouse skin.
@nl
P2093
P2860
P356
P1476
Transcription factor Fli1 regulates collagen fibrillogenesis in mouse skin.
@en
P2093
Dennis K Watson
Gabor Szalai
Margaret Markiewicz
Masahide Kubo
Yoshihide Asano
P2860
P304
P356
10.1128/MCB.01278-08
P407
P577
2008-11-10T00:00:00Z