Viral complementation allows HIV-1 replication without integration. - Wikidata - wikimedia - TriplyDB

Viral complementation allows HIV-1 replication without integration.

Viral complementation allows HIV-1 replication without integration.

http://www.wikidata.org/entity/Q39963401

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Increased burst size in multiply infected cells can alter basic virus dynamics Viral quasispecies evolution Contribution of HIV-1 genomes that do not integrate to the basic reproductive ratio of the virus.Fitness ranking of individual mutants drives patterns of epistatic interactions in HIV-1 Human immunodeficiency virus type 1 Vpu and cellular TASK proteins suppress transcription of unintegrated HIV-1 DNA.Multiploid inheritance of HIV-1 during cell-to-cell infection.Synaptic transmission and the susceptibility of HIV infection to anti-viral drugs Peptides derived from HIV-1 integrase that bind Rev stimulate viral genome integration.Uterine epithelial cell regulation of DC-SIGN expression inhibits transmitted/founder HIV-1 trans infection by immature dendritic cells Expression of Nef from unintegrated HIV-1 DNA downregulates cell surface CXCR4 and CCR5 on T-lymphocytes.Effect of synaptic transmission on viral fitness in HIV infection.Cell-to-Cell Transmission of HIV-1 Is Required to Trigger Pyroptotic Death of Lymphoid-Tissue-Derived CD4 T Cells HIV-1 pathogenicity and virion production are dependent on the metabolic phenotype of activated CD4+ T cells Specific eradication of HIV-1 from infected cultured cells Estradiol reduces susceptibility of CD4+ T cells and macrophages to HIV-infection.Mice transgenic for CD4-specific human CD4, CCR5 and cyclin T1 expression: a new model for investigating HIV-1 transmission and treatment efficacy.The role of unintegrated DNA in HIV infection Integrase inhibitor reversal dynamics indicate unintegrated HIV-1 dna initiate de novo integration.Suppression of Foxo1 activity and down-modulation of CD62L (L-selectin) in HIV-1 infected resting CD4 T cells.Inhibitory Effect of Individual or Combinations of Broadly Neutralizing Antibodies and Antiviral Reagents against Cell-Free and Cell-to-Cell HIV-1 Transmission.Mucosal stromal fibroblasts markedly enhance HIV infection of CD4+ T cells.HIV-1 latency and virus production from unintegrated genomes following direct infection of resting CD4 T cells.Opposite transcriptional regulation of integrated vs unintegrated HIV genomes by the NF-κB pathway.Binding of RNA by APOBEC3G controls deamination-independent restriction of retroviruses Quantitative analysis of the time-course of viral DNA forms during the HIV-1 life cycle The remarkable frequency of human immunodeficiency virus type 1 genetic recombination Measurement of human immunodeficiency virus type 1 preintegration transcription by using Rev-dependent Rev-CEM cells reveals a sizable transcribing DNA population comparable to that from proviral templates.An HIV-1 replication pathway utilizing reverse transcription products that fail to integrate.Epigenetic activation of unintegrated HIV-1 genomes by gut-associated short chain fatty acids and its implications for HIV infection.The use of immune complex vaccines to enhance antibody responses against neutralizing epitopes on HIV-1 envelope gp120.A systematic comparison reveals substantial differences in chromosomal versus episomal encoding of enhancer activity.Distribution and fate of HIV-1 unintegrated DNA species: a comprehensive update Effect of multiple infection of cells on the evolutionary dynamics of HIV in vivo: implications for host adaptation mechanisms.High-Throughput Humanized Mouse Models for Evaluation of HIV-1 Therapeutics and Pathogenesis.Total HIV-1 DNA, a Marker of Viral Reservoir Dynamics with Clinical Implications.Identification of highly conserved residues involved in inhibition of HIV-1 RNase H function by Diketo acid derivatives.Different Pathways Leading to Integrase Inhibitors Resistance Suboptimal provirus expression explains apparent nonrandom cell coinfection with HIV-1.Transcription of preintegrated HIV-1 cDNA modulates cell surface expression of major histocompatibility complex class I via Nef.Development of a nonintegrating Rev-dependent lentiviral vector carrying diphtheria toxin A chain and human TRAF6 to target HIV reservoirs.

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Viral complementation allows HIV-1 replication without integration.

http://www.wikidata.org/entity/Q39963401

description

2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年學術文章

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2008年學術文章

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2008年學術文章

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name

Viral complementation allows HIV-1 replication without integration.

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Viral complementation allows HIV-1 replication without integration.

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type

label

Viral complementation allows HIV-1 replication without integration.

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Viral complementation allows HIV-1 replication without integration.

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prefLabel

Viral complementation allows HIV-1 replication without integration.

@en

Viral complementation allows HIV-1 replication without integration.

@nl

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Viral complementation allows HIV-1 replication without integration.

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David N Levy

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Dimitrios N Vatakis

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Gregory C Bristol

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Sean A Burke

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Steven D Lawrie

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P2860

HIV-1 cell to cell transfer across an Env-induced, actin-dependent synapse

Active nuclear import of human immunodeficiency virus type 1 preintegration complexes

Gene transfer in humans using a conditionally replicating lentiviral vector

Broad antiretroviral defence by human APOBEC3G through lethal editing of nascent reverse transcripts

Inhibitors of strand transfer that prevent integration and inhibit HIV-1 replication in cells

Presence of an inducible HIV-1 latent reservoir during highly active antiretroviral therapy

HIV population dynamics in vivo: implications for genetic variation, pathogenesis, and therapy

Massive infection and loss of memory CD4+ T cells in multiple tissues during acute SIV infection

RNA virus mutations and fitness for survival

Quantification of latent tissue reservoirs and total body viral load in HIV-1 infection

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10.1186/1742-4690-5-60

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Huub C Gelderblom

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