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alpha-Synuclein in filamentous inclusions of Lewy bodies from Parkinson's disease and dementia with lewy bodiesA specific clinical pattern of camptocormia in Parkinson's diseaseMetabolism and possible health effects of aluminumFormation of α-synuclein Lewy neurite-like aggregates in axons impedes the transport of distinct endosomes.Autonomic nervous system dysfunction in Parkinson's disease: relationships with age, medication, duration, and severity.Critical role of truncated α-synuclein and aggregates in Parkinson's disease and incidental Lewy body disease.Aggregation of α-synuclein in S. cerevisiae is associated with defects in endosomal trafficking and phospholipid biosynthesis.(123)I-metaiodobenzylguanidine myocardial scintigraphy in Parkinson's disease.Neuronal cell death in neurodegenerative diseases: recurring themes around protein handling.Suppression of α-synuclein toxicity and vesicle trafficking defects by phosphorylation at S129 in yeast depends on genetic context.The Parkinson's disease protein alpha-synuclein disrupts cellular Rab homeostasisChanges in the sympathetic innervation of the gut in rotenone treated mice as possible early biomarker for Parkinson's disease.Reduced striatal tyrosine hydroxylase in incidental Lewy body disease.Neuropathological Staging of Brain Pathology in Sporadic Parkinson's disease: Separating the Wheat from the Chaff.Autophagic pathways in Parkinson disease and related disorders.Lewy pathology and neurodegeneration in premotor Parkinson's disease.The Lewy body in Parkinson's disease and related neurodegenerative disorders.Dysfunction of the locus coeruleus-norepinephrine system and related circuitry in Parkinson's disease-related dementia.Current concepts and controversies in the pathogenesis of Parkinson's disease dementia and Dementia with Lewy Bodies.Impaired baroreflex function in mice overexpressing alpha-synuclein.Accumulation of HtrA2/Omi in neuronal and glial inclusions in brains with alpha-synucleinopathies.Loss of native α-synuclein multimerization by strategically mutating its amphipathic helix causes abnormal vesicle interactions in neuronal cells.Lysia K.S. Forno, MD February 14, 1918-May 8, 2015.Alpha-synuclein pathology of the spinal and peripheral autonomic nervous system in neurologically unimpaired elderly subjects.Lewy body pathology involves cutaneous nerves.Lack of pathogenic potential of peripheral α-synuclein aggregates from Parkinson's disease patients.Reduction of Small Fibers of Thoracic Ventral Roots and Neurons of Intermediolateral Nucleus in Parkinson Disease and Dementia with Lewy Bodies.
P2860
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P2860
description
1976 nî lūn-bûn
@nan
1976年の論文
@ja
1976年学术文章
@wuu
1976年学术文章
@zh-cn
1976年学术文章
@zh-hans
1976年学术文章
@zh-my
1976年学术文章
@zh-sg
1976年學術文章
@yue
1976年學術文章
@zh
1976年學術文章
@zh-hant
name
Ultrastructure of Lewy bodies in the stellate ganglion.
@en
Ultrastructure of Lewy bodies in the stellate ganglion.
@nl
type
label
Ultrastructure of Lewy bodies in the stellate ganglion.
@en
Ultrastructure of Lewy bodies in the stellate ganglion.
@nl
prefLabel
Ultrastructure of Lewy bodies in the stellate ganglion.
@en
Ultrastructure of Lewy bodies in the stellate ganglion.
@nl
P356
P1476
Ultrastructure of Lewy bodies in the stellate ganglion.
@en
P2093
R L Norville
P2860
P2888
P304
P356
10.1007/BF00688674
P577
1976-03-01T00:00:00Z