Suppression of anoikis by the neurotrophic receptor TrkB in human ovarian cancer.
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Tropomyosin-related kinase B/brain derived-neurotrophic factor signaling pathway as a potential therapeutic target for colorectal cancermiR-200c Regulation of Metastases in Ovarian Cancer: Potential Role in Epithelial and Mesenchymal TransitionBDNF is associated with SFRP1 expression in luminal and basal-like breast cancer cell lines and primary breast cancer tissues: a novel role in tumor suppression?TrkB is highly expressed in NSCLC and mediates BDNF-induced the activation of Pyk2 signaling and the invasion of A549 cells.Neuronal transcription factor Brn-3a(l) is over expressed in high-grade ovarian carcinomas and tumor cells from ascites of patients with advanced-stage ovarian cancermiR-200c targets a NF-κB up-regulated TrkB/NTF3 autocrine signaling loop to enhance anoikis sensitivity in triple negative breast cancerEpigenetic inactivation of EFEMP1 is associated with tumor suppressive function in endometrial carcinoma.Upregulation of TrkB promotes epithelial-mesenchymal transition and anoikis resistance in endometrial carcinoma.Notch3 overexpression promotes anoikis resistance in epithelial ovarian cancer via upregulation of COL4A2.A TrkB-STAT3-miR-204-5p regulatory circuitry controls proliferation and invasion of endometrial carcinoma cellsBrain-derived neurotrophic factor expression predicts adverse pathological & clinical outcomes in human breast cancer.Minocycline attenuates hypoxia-inducible factor-1α expression correlated with modulation of p53 and AKT/mTOR/p70S6K/4E-BP1 pathway in ovarian cancer: in vitro and in vivo studies.Targets of miR-200c mediate suppression of cell motility and anoikis resistanceThe PI3K/AKT/mTOR pathway is a potential predictor of distinct invasive and migratory capacities in human ovarian cancer cell linesRestoration of miR-200c to ovarian cancer reduces tumor burden and increases sensitivity to paclitaxelBrain-derived neurotrophic factor/tropomyosin-related kinase B pathway in gastric cancer.Profile of erlotinib and its potential in the treatment of advanced ovarian carcinomaSuppression of choriocarcinoma invasion and metastasis following blockade of BDNF/TrkB signaling.Targeting the epidermal growth factor receptor in epithelial ovarian cancer: current knowledge and future challenges.TrkB as a therapeutic target for ovarian cancer.On Trk--the TrkB signal transduction pathway is an increasingly important target in cancer biology.Influence of GRPR and BDNF/TrkB signaling on the viability of breast and gynecologic cancer cellsIntegration of autophagy and anoikis resistance in solid tumors.Update of molecular pathobiology in oral cancer: a review.The Effect of a Histone Deacetylase Inhibitor (AR-42) on Canine Prostate Cancer Growth and Metastasis.Elevated CDCP1 predicts poor patient outcome and mediates ovarian clear cell carcinoma by promoting tumor spheroid formation, cell migration and chemoresistance.TrkB inhibition by GNF-4256 slows growth and enhances chemotherapeutic efficacy in neuroblastoma xenografts.BRCA1-IRIS inactivation sensitizes ovarian tumors to cisplatin.More expressions of BDNF and TrkB in multiple hepatocellular carcinoma and anti-BDNF or K252a induced apoptosis, supressed invasion of HepG2 and HCCLM3 cells.Tropomyosin-related kinase B promotes distant metastasis of colorectal cancer through protein kinase B-mediated anoikis suppression and correlates with poor prognosis.TrkB expression level correlates with metastatic properties of L1 mouse sarcoma cells cultured in non-adhesive conditions.High TrkB expression levels are associated with poor prognosis and EMT induction in colorectal cancer cells.K252a induces anoikis-sensitization with suppression of cellular migration in Epstein-Barr virus (EBV)--associated nasopharyngeal carcinoma cells.Downregulation of HtrA1 promotes resistance to anoikis and peritoneal dissemination of ovarian cancer cellsBiological influence of brain-derived neurotrophic factor (BDNF) on colon cancer cellsTrks are novel oncogenes involved in the induction of neovascularization, tumor progression, and nodal metastasis in oral squamous cell carcinoma.Bioinformatics analysis of gene expression profiles to identify causal genes in luminal B2 breast cancer.Pan-Trk Immunohistochemistry Is an Efficient and Reliable Screen for the Detection of NTRK Fusions.Brain-derived neurotrophic factor/tropomyosin-related kinase B signaling pathway contributes to the aggressive behavior of lung squamous cell carcinoma.Expression of tyrosine kinase receptor B in eutopic endometrium of women with adenomyosis.
P2860
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P2860
Suppression of anoikis by the neurotrophic receptor TrkB in human ovarian cancer.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Suppression of anoikis by the neurotrophic receptor TrkB in human ovarian cancer.
@en
type
label
Suppression of anoikis by the neurotrophic receptor TrkB in human ovarian cancer.
@en
prefLabel
Suppression of anoikis by the neurotrophic receptor TrkB in human ovarian cancer.
@en
P2093
P2860
P1433
P1476
Suppression of anoikis by the neurotrophic receptor TrkB in human ovarian cancer.
@en
P2093
P2860
P304
P356
10.1111/J.1349-7006.2007.00722.X
P577
2008-01-14T00:00:00Z