Sequence requirements of the Epstein-Barr virus latent origin of DNA replication.
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EBNA1-mediated recruitment of a histone H2B deubiquitylating complex to the Epstein-Barr virus latent origin of DNA replicationEBP2, a human protein that interacts with sequences of the Epstein-Barr virus nuclear antigen 1 important for plasmid maintenance.Architecture of the yeast origin recognition complex bound to origins of DNA replication.Characterization of the minimal replicator of Kaposi's sarcoma-associated herpesvirus latent origin.Functional analyses of the EBNA1 origin DNA binding protein of Epstein-Barr virus.The minimal replicator of Epstein-Barr virus oriP.The replicator of the Epstein-Barr virus latent cycle origin of DNA replication, oriP, is composed of multiple functional elements.The dyad symmetry element of Epstein-Barr virus is a dominant but dispensable replication origin.Epstein-Barr nuclear antigen 1 binds and destabilizes nucleosomes at the viral origin of latent DNA replication.Human DNA replication initiation factors, ORC and MCM, associate with oriP of Epstein-Barr virus.Human origin recognition complex binds to the region of the latent origin of DNA replication of Epstein-Barr virus.Maintenance of Epstein-Barr virus (EBV) oriP-based episomes requires EBV-encoded nuclear antigen-1 chromosome-binding domains, which can be replaced by high-mobility group-I or histone H1.The latency-associated nuclear antigen of Kaposi's sarcoma-associated herpesvirus supports latent DNA replication in dividing cells.Mitotic chromosome-binding activity of latency-associated nuclear antigen 1 is required for DNA replication from terminal repeat sequence of Kaposi's sarcoma-associated herpesvirusKaposi's Sarcoma-Associated Herpesvirus Genome Replication, Partitioning, and Maintenance in Latency.Cooperative assembly of EBNA1 on the Epstein-Barr virus latent origin of replicationLive-cell imaging reveals multiple interactions between Epstein-Barr virus nuclear antigen 1 and cellular chromatin during interphase and mitosisAnalysis of viral cis elements conferring Kaposi's sarcoma-associated herpesvirus episome partitioning and maintenance.Genetic control of translesion synthesis on leading and lagging DNA strands in plasmids derived from Epstein-Barr virus in human cells.The plasmid replicon of Epstein-Barr virus: mechanistic insights into efficient, licensed, extrachromosomal replication in human cells.Identifying a property of origins of DNA synthesis required to support plasmids stably in human cells.Telomere repeat binding factors TRF1, TRF2, and hRAP1 modulate replication of Epstein-Barr virus OriPInvolvement of SSRP1 in latent replication of Kaposi's sarcoma-associated herpesvirusThe Epstein-Barr Virus EBNA1 Protein.Efficient replication of Epstein-Barr virus-derived plasmids requires tethering by EBNA1 to host chromosomes.Analysis of cis-elements that facilitate extrachromosomal persistence of human papillomavirus genomes.Evidence that protein binding specifies sites of DNA demethylation.The linking regions of EBNA1 are essential for its support of replication and transcription.Establishment of an oriP replicon is dependent upon an infrequent, epigenetic event.Coupling of mitotic chromosome tethering and replication competence in epstein-barr virus-based plasmids.Specific methylation patterns in two control regions of Epstein-Barr virus latency: the LMP-1-coding upstream regulatory region and an origin of DNA replication (oriP).Rep*: a viral element that can partially replace the origin of plasmid DNA synthesis of Epstein-Barr virus.Replication from oriP of Epstein-Barr virus requires exact spacing of two bound dimers of EBNA1 which bend DNA.The cis-acting family of repeats can inhibit as well as stimulate establishment of an oriP repliconActivation of TRAF5 and TRAF6 signal cascades negatively regulates the latent replication origin of Epstein-Barr virus through p38 mitogen-activated protein kinase.Initiation of DNA replication within oriP is dispensable for stable replication of the latent Epstein-Barr virus chromosome after infection of established cell lines.Reconstitution of Epstein-Barr virus-based plasmid partitioning in budding yeastNucleosome assembly proteins bind to Epstein-Barr virus nuclear antigen 1 and affect its functions in DNA replication and transcriptional activation.Structural and Functional Basis for an EBNA1 Hexameric Ring in Epstein-Barr Virus Episome Maintenance.The plasmid replicon of EBV consists of multiple cis-acting elements that facilitate DNA synthesis by the cell and a viral maintenance element
P2860
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P2860
Sequence requirements of the Epstein-Barr virus latent origin of DNA replication.
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
1994年论文
@zh
1994年论文
@zh-cn
name
Sequence requirements of the Epstein-Barr virus latent origin of DNA replication.
@en
type
label
Sequence requirements of the Epstein-Barr virus latent origin of DNA replication.
@en
prefLabel
Sequence requirements of the Epstein-Barr virus latent origin of DNA replication.
@en
P2093
P2860
P1433
P1476
Sequence requirements of the Epstein-Barr virus latent origin of DNA replication.
@en
P2093
P2860
P304
P407
P577
1994-03-01T00:00:00Z