The kinetics of simian virus 40-induced progression of quiescent cells into S phase depend on four independent functions of large T antigen.
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Molecular biology, epidemiology, and pathogenesis of progressive multifocal leukoencephalopathy, the JC virus-induced demyelinating disease of the human brainThe replication protein A binding site in simian virus 40 (SV40) T antigen and its role in the initial steps of SV40 DNA replication.Interaction between T antigen and TEA domain of the factor TEF-1 derepresses simian virus 40 late promoter in vitro: identification of T-antigen domains important for transcription controlInteraction of the transcription factor TFIID with simian virus 40 (SV40) large T antigen interferes with replication of SV40 DNA in vitro.Transactivation of a ribosomal gene by simian virus 40 large-T antigen requires at least three activities of the protein.Activation of CREB/ATF sites by polyomavirus large T antigen.Intestinal dysplasia induced by simian virus 40 T antigen is independent of p53.Induction of duplication reversion in human fibroblasts, by wild-type and mutated SV40 T antigen, covaries with the ability to induce host DNA synthesis.Simian virus 40 large T antigen and two independent T-antigen segments sensitize cells to apoptosis following genotoxic damageHuman glial chimeric mice reveal astrocytic dependence of JC virus infectionCell cycle modulation by Marek's disease virus: the tegument protein VP22 triggers S-phase arrest and DNA damage in proliferating cells.Simian virus 40 large T antigen contains two independent activities that cooperate with a ras oncogene to transform rat embryo fibroblastsRole of c-myc in simian virus 40 large tumor antigen-induced DNA synthesis in quiescent 3T3-L1 mouse fibroblastsThe block of adipocyte differentiation by a C-terminally truncated, but not by full-length, simian virus 40 large tumor antigen is dependent on an intact retinoblastoma susceptibility protein family binding domain.Genetic analysis of polyomavirus large T nuclear localization: nuclear localization is required for productive association with pRb family members.A novel simian virus 40 early-region domain mediates transactivation of the cyclin A promoter by small-t antigen and is required for transformation in small-t antigen-dependent assays.Safety-modified episomal vectors for human gene therapy.Association of p300 and CBP with simian virus 40 large T antigenElevated recombination in immortal human cells is mediated by HsRAD51 recombinase.Transforming growth factor beta enhances the glucocorticoid response of the mouse mammary tumor virus promoter through Smad and GA-binding proteinsCell-type specific regulation of gene expression by simian virus 40 T antigens.JC virus inclusions in progressive multifocal leukoencephalopathy: scaffolding promyelocytic leukemia nuclear bodies grow with cell cycle transition through an S-to-G2-like state in enlarging oligodendrocyte nuclei.Polyomavirus BK and prostate cancer: an unworthy scientific effort?Ataxia telangiectasia-mutated damage-signaling kinase- and proteasome-dependent destruction of Mre11-Rad50-Nbs1 subunits in Simian virus 40-infected primate cellsLoss of p19(ARF) eliminates the requirement for the pRB-binding motif in simian virus 40 large T antigen-mediated transformation.Induction of p53-independent apoptosis by simian virus 40 small t antigen.Large T antigen promotes JC virus replication in G2-arrested cells by inducing ATM- and ATR-mediated G2 checkpoint signaling.An N-terminal deletion mutant of simian virus 40 (SV40) large T antigen oligomerizes incorrectly on SV40 DNA but retains the ability to bind to DNA polymerase alpha and replicate SV40 DNA in vitro.Adding an Rb-binding site to an N-terminally truncated simian virus 40 T antigen restores growth to high cell density, and the T common region in trans provides anchorage-independent growth and rapid growth in low serum concentrations.Tiny T antigen: an autonomous polyomavirus T antigen amino-terminal domain.Transactivation of E2F-regulated genes by polyomavirus large T antigen: evidence for a two-step mechanism.Multiple DNA damage signaling and repair pathways deregulated by simian virus 40 large T antigen.Replication stress and mitotic dysfunction in cells expressing simian virus 40 large T antigenRecombination and its roles in DNA repair, cellular immortalization and cancer
P2860
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P2860
The kinetics of simian virus 40-induced progression of quiescent cells into S phase depend on four independent functions of large T antigen.
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
1994年论文
@zh
1994年论文
@zh-cn
name
The kinetics of simian virus 4 ...... functions of large T antigen.
@en
type
label
The kinetics of simian virus 4 ...... functions of large T antigen.
@en
prefLabel
The kinetics of simian virus 4 ...... functions of large T antigen.
@en
P2093
P2860
P1433
P1476
The kinetics of simian virus 4 ...... functions of large T antigen.
@en
P2093
Dickmanns A
Simmersbach F
Wildeman AG
Zeitvogel A
P2860
P304
P407
P577
1994-09-01T00:00:00Z