Endotoxin priming of neutrophils requires NADPH oxidase-generated oxidants and is regulated by the anion transporter ClC-3.
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Transepithelial migration of neutrophils into the lung requires TREM-1Multiple Phenotypic Changes Define Neutrophil PrimingNOX2 protects against progressive lung injury and multiple organ dysfunction syndrome.Electrophysiology of reactive oxygen production in signaling endosomesActivation of swelling-activated chloride current by tumor necrosis factor-alpha requires ClC-3-dependent endosomal reactive oxygen productionThe role of reactive oxygen species in microvascular remodeling.Priming of neutrophils and differentiated PLB-985 cells by pathophysiological concentrations of TNF-α is partially oxygen dependentEndotoxin priming of neutrophils requires endocytosis and NADPH oxidase-dependent endosomal reactive oxygen speciesChloride Channel 3 Channels in the Activation and Migration of Human Blood Eosinophils in Allergic Asthma.Roles of neutrophils in the regulation of the extent of human inflammation through delivery of IL-1 and clearance of chemokinesA Common Genetic Variant in TLR1 Enhances Human Neutrophil Priming and Impacts Length of Intensive Care Stay in Pediatric Sepsis.Defective organellar acidification as a cause of cystic fibrosis lung disease: reexamination of a recurring hypothesis.Cell biology and physiology of CLC chloride channels and transporters.Analysis of electrophysiological properties and responses of neutrophils.K⁺-Cl⁻ cotransport mediates the bactericidal activity of neutrophils by regulating NADPH oxidase activation.Paradoxical effect of diphenyleneiodonium in inducing DNA damage and apoptosis.A critical role for chloride channel-3 (CIC-3) in smooth muscle cell activation and neointima formation.Endocytosis is required for exocytosis and priming of respiratory burst activity in human neutrophils.Neutrophil azurophilic granule exocytosis is primed by TNF-α and partially regulated by NADPH oxidase.ClC-3 deficiency prevents apoptosis induced by angiotensin II in endothelial progenitor cells via inhibition of NADPH oxidase.ClC-3 chloride channel/antiporter defect contributes to inflammatory bowel disease in humans and mice.CLC Chloride Channels and Transporters: From Genes to Protein Structure, Pathology and Physiology
P2860
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P2860
Endotoxin priming of neutrophils requires NADPH oxidase-generated oxidants and is regulated by the anion transporter ClC-3.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
Endotoxin priming of neutrophi ...... y the anion transporter ClC-3.
@en
type
label
Endotoxin priming of neutrophi ...... y the anion transporter ClC-3.
@en
prefLabel
Endotoxin priming of neutrophi ...... y the anion transporter ClC-3.
@en
P2093
P356
P1476
Endotoxin priming of neutrophi ...... by the anion transporter ClC-3
@en
P2093
A Paige Davis
Gail Bailey
James J Matsuda
Jessica G Moreland
Jessica S Hook
William M Nauseef
P304
33958-33967
P356
10.1074/JBC.M705289200
P407
P50
P577
2007-10-01T00:00:00Z