Deregulated Wnt/beta-catenin program in high-risk neuroblastomas without MYCN amplification. - Wikidata - wikimedia - TriplyDB

Deregulated Wnt/beta-catenin program in high-risk neuroblastomas without MYCN amplification.

Deregulated Wnt/beta-catenin program in high-risk neuroblastomas without MYCN amplification.

http://www.wikidata.org/entity/Q40088682

about

The Wnt receptor FZD1 mediates chemoresistance in neuroblastoma through activation of the Wnt/beta-catenin pathway Evaluation and Structural Basis for the Inhibition of Tankyrases by PARP Inhibitors Molecular pathogenesis of peripheral neuroblastic tumors HOXD-AS1 is a novel lncRNA encoded in HOXD cluster and a marker of neuroblastoma progression revealed via integrative analysis of noncoding transcriptome Global MYCN transcription factor binding analysis in neuroblastoma reveals association with distinct E-box motifs and regions of DNA hypermethylation.Combinatorial regulation of neuroblastoma tumor progression by N-Myc and hypoxia inducible factor HIF-1alpha.WNT10A plays an oncogenic role in renal cell carcinoma by activating WNT/β-catenin pathway.Silencing of miR-370 in human cholangiocarcinoma by allelic loss and interleukin-6 induced maternal to paternal epigenotype switch Prostaglandin E2 promotes MYCN non-amplified neuroblastoma cell survival via β-catenin stabilization ODC1 is a critical determinant of MYCN oncogenesis and a therapeutic target in neuroblastoma.Multi-study integration of brain cancer transcriptomes reveals organ-level molecular signatures Characteristic Markers of the WNT Signaling Pathways Are Differentially Expressed in Osteoarthritic Cartilage.Autocrine Activation of the Wnt/β-Catenin Pathway by CUX1 and GLIS1 in Breast Cancers.Polymorphisms in WNT6 and WNT10A and colorectal adenoma risk.Planar cell polarity gene expression correlates with tumor cell viability and prognostic outcome in neuroblastoma Functional genomics identifies therapeutic targets for MYC-driven cancer.ATF4 regulates MYC-mediated neuroblastoma cell death upon glutamine deprivation.High Myc pathway activity and low stage of neuronal differentiation associate with poor outcome in neuroblastoma L1 and NCAM adhesion molecules as signaling coreceptors in neuronal migration and process outgrowth Exome and deep sequencing of clinically aggressive neuroblastoma reveal somatic mutations that affect key pathways involved in cancer progression Distinct transcriptional MYCN/c-MYC activities are associated with spontaneous regression or malignant progression in neuroblastomas.Disrupting polyamine homeostasis as a therapeutic strategy for neuroblastoma.Apoptotic cell death in neuroblastoma.p53 family: Therapeutic targets in neuroblastoma.Children's Oncology Group's 2013 blueprint for research: neuroblastoma.LGR5 regulates pro-survival MEK/ERK and proliferative Wnt/β-catenin signalling in neuroblastoma.Gene expression analysis predicts insect venom anaphylaxis in indolent systemic mastocytosis.Neuroblastoma in children: Update on clinicopathologic and genetic prognostic factors.TRPM7 maintains progenitor-like features of neuroblastoma cells: implications for metastasis formation.RLIP76 Inhibition: A Promising Developmental Therapy for Neuroblastoma.Transcriptional regulation of Wnt inhibitory factor-1 by Miz-1/c-Myc.Frizzled2 signaling regulates growth of high-risk neuroblastomas by interfering with β-catenin-dependent and β-catenin-independent signaling pathways.Wnt signalling is a bi-directional vulnerability of cancer cells.p53, SKP2, and DKK3 as MYCN Target Genes and Their Potential Therapeutic Significance.Overexpression of neurone glial-related cell adhesion molecule is an independent predictor of poor prognosis in advanced colorectal cancer.WNT signaling, the development of the sympathoadrenal-paraganglionic system and neuroblastoma.Therapeutically targeting glypican-2 via single-domain antibody-based chimeric antigen receptors and immunotoxins in neuroblastoma.The expression of SFRP1, SFRP3, DVL1, and DVL2 proteins in testicular germ cell tumors.Wnt Signalling Drives Context-Dependent Differentiation or Proliferation in Neuroblastoma.NEU4L sialidase overexpression promotes β-catenin signaling in neuroblastoma cells, enhancing stem-like malignant cell growth.

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P2860

Deregulated Wnt/beta-catenin program in high-risk neuroblastomas without MYCN amplification.

http://www.wikidata.org/entity/Q40088682

description

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2007年论文

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2007年论文

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Deregulated Wnt/beta-catenin p ...... as without MYCN amplification.

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Deregulated Wnt/beta-catenin p ...... as without MYCN amplification.

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Deregulated Wnt/beta-catenin p ...... as without MYCN amplification.

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A Cnaan

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H Zhao

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J Jagannathan

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J M Maris

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P2860

Identification of c-MYC as a target of the APC pathway

Activation of beta-catenin-Tcf signaling in colon cancer by mutations in beta-catenin or APC

Gene set enrichment analysis: a knowledge-based approach for interpreting genome-wide expression profiles.

Functional interaction between beta-catenin and FOXO in oxidative stress signaling

The Myc/Max/Mad network and the transcriptional control of cell behavior

The Hedgehog and Wnt signalling pathways in cancer

Transactivation of repair genes by BRCA1

Low expression of Wnt-5a gene is associated with high-risk neuroblastoma

Wnt signalling in stem cells and cancer

Regulation of cerebral cortical size by control of cell cycle exit in neural precursors

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