Dystroglycan expression is reduced during prostate tumorigenesis and is regulated by androgens in prostate cancer cells.
about
Processing and secretion of the N-terminal domain of alpha-dystroglycan in cell culture mediaTumor suppressor function of laminin-binding alpha-dystroglycan requires a distinct beta3-N-acetylglucosaminyltransferaseFer kinase regulates cell migration through α-dystroglycan glycosylationDystroglycan is not required for maintenance of the luminal epithelial basement membrane or cell polarity in the mouse prostate.Microfabricated electrochemical cell-based biosensors for analysis of living cells in vitro.Reduced glycosylation of α-dystroglycans on carcinoma cells contributes to formation of highly infiltrative histological patterns in prostate cancer.The glycosyltransferase LARGE2 is repressed by Snail and ZEB1 in prostate cancer.Increased expression of CD133 and reduced dystroglycan expression are strong predictors of poor outcome in colon cancer patients.Loss of LARGE2 disrupts functional glycosylation of α-dystroglycan in prostate cancer.VHL-dependent regulation of a β-dystroglycan glycoform and glycogene expression in renal cancerNuclear targeting of dystroglycan promotes the expression of androgen regulated transcription factors in prostate cancer.Dystroglycan function is a novel determinant of tumor growth and behavior in prostate cancer.The tumor suppressor HINT1 regulates MITF and β-catenin transcriptional activity in melanoma cells.Loss of dystroglycan function in oesophageal cancer.Identification of Endothelin-1 and NR4A2 as CD133-regulated genes in colon cancer cells.Periacinar retraction clefting in nonneoplastic and neoplastic prostatic glands: artifact or molecular involvement.Nintedanib treatment delays prostate dorsolateral lobe cancer progression in the TRAMP model: Contribution to the epithelial-stromal interaction balance.Identification of Sp1 and GC-boxes as transcriptional regulators of mouse Dag1 gene promoter.Steroid hormone receptors, matrix metalloproteinases, insulin-like growth factor, and dystroglycans interactions in prostatic diseases in the elderly men.
P2860
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P2860
Dystroglycan expression is reduced during prostate tumorigenesis and is regulated by androgens in prostate cancer cells.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Dystroglycan expression is red ...... gens in prostate cancer cells.
@en
type
label
Dystroglycan expression is red ...... gens in prostate cancer cells.
@en
prefLabel
Dystroglycan expression is red ...... gens in prostate cancer cells.
@en
P2093
P2860
P50
P356
P1476
Dystroglycan expression is red ...... gens in prostate cancer cells.
@en
P2093
Boninsegna A
Cittadini A
De Paola B
Di Salvatore M
Faraglia B
Maiorana A
P2860
P304
P356
10.1002/JCP.21130
P577
2007-11-01T00:00:00Z