Dystroglycan expression is frequently reduced in human breast and colon cancers and is associated with tumor progression.
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A role of fukutin, a gene responsible for Fukuyama type congenital muscular dystrophy, in cancer cells: a possible role to suppress cell proliferationHypoxic tumor cell modulates its microenvironment to enhance angiogenic and metastatic potential by secretion of proteins and exosomesAltered expression of natively glycosylated alpha dystroglycan in pediatric solid tumors.Large induces functional glycans in an O-mannosylation dependent manner and targets GlcNAc terminals on alpha-dystroglycanEZH2 depletion blocks the proliferation of colon cancer cells.Dystroglycan controls signaling of multiple hormones through modulation of STAT5 activity.Dystroglycan loss disrupts polarity and beta-casein induction in mammary epithelial cells by perturbing laminin anchoring.Design and screening of a glial cell-specific, cell penetrating peptide for therapeutic applications in multiple sclerosis.Sarcolemma instability during mechanical activity in Largemyd cardiac myocytes with loss of dystroglycan extracellular matrix receptor functionReduced glycosylation of α-dystroglycans on carcinoma cells contributes to formation of highly infiltrative histological patterns in prostate cancer.Downregulation of dystroglycan glycosyltransferases LARGE2 and ISPD associate with increased mortality in clear cell renal cell carcinoma.Different Array CGH profiles within hereditary breast cancer tumors associated to BRCA1 expression and overall survivalThe dystroglycan complex: from biology to cancer.The glycosyltransferase LARGE2 is repressed by Snail and ZEB1 in prostate cancer.Functional nsSNPs from carcinogenesis-related genes expressed in breast tissue: potential breast cancer risk alleles and their distribution across human populations.Increased expression of CD133 and reduced dystroglycan expression are strong predictors of poor outcome in colon cancer patients.Loss of LARGE2 disrupts functional glycosylation of α-dystroglycan in prostate cancer.Loss of alpha-dystroglycan laminin binding in epithelium-derived cancers is caused by silencing of LARGE.VHL-dependent regulation of a β-dystroglycan glycoform and glycogene expression in renal cancerPerlecan and Dystroglycan act at the basal side of the Drosophila follicular epithelium to maintain epithelial organization.RETRACTED: Dystroglycan and perlecan provide a basal cue required for epithelial polarity during energetic stress.Non-myogenic tumors display altered expression of dystrophin (DMD) and a high frequency of genetic alterations.Exosomes as mediators of platinum resistance in ovarian cancer.The LKB1/AMPK polarity pathway.The secretome signature of colon cancer cell lines.Increments of alpha-dystroglycan expression in liver metastasis correlate with poor survival in gastric cancer.Loss of dystroglycan function in oesophageal cancer.Identification of Endothelin-1 and NR4A2 as CD133-regulated genes in colon cancer cells.Evolution on experimental animal model for upper urothelium carcinogenesis.γ-Secretase Dependent Nuclear Targeting of Dystroglycan.Expression of beta-dystroglycan is reduced or absent in many human carcinomas.Recent advancements in understanding mammalian O-mannosylation.Dystroglycan expression is reduced during prostate tumorigenesis and is regulated by androgens in prostate cancer cells.Analysis of dystroglycan regulation and functions in mouse mammary epithelial cells and implications for mammary tumorigenesis.Boundary cells restrict dystroglycan trafficking to control basement membrane sliding during tissue remodelingActin-towards a deeper understanding of the relationship between tissue context, cellular function and tumorigenesis.Dystroglycan patterns on the prostate of non-obese diabetic mice submitted to glycaemic control.Differential expression of perlecan receptors, α-dystroglycan and integrin β1, before and after invasion of oral squamous cell carcinoma.Periacinar retraction clefting in nonneoplastic and neoplastic prostatic glands: artifact or molecular involvement.Direct Mapping of Additional Modifications on Phosphorylated O-glycans of α-Dystroglycan by Mass Spectrometry Analysis in Conjunction with Knocking Out of Causative Genes for Dystroglycanopathy.
P2860
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P2860
Dystroglycan expression is frequently reduced in human breast and colon cancers and is associated with tumor progression.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Dystroglycan expression is fre ...... ciated with tumor progression.
@ast
Dystroglycan expression is fre ...... ciated with tumor progression.
@en
type
label
Dystroglycan expression is fre ...... ciated with tumor progression.
@ast
Dystroglycan expression is fre ...... ciated with tumor progression.
@en
prefLabel
Dystroglycan expression is fre ...... ciated with tumor progression.
@ast
Dystroglycan expression is fre ...... ciated with tumor progression.
@en
P2093
P2860
P1476
Dystroglycan expression is fre ...... ociated with tumor progression
@en
P2093
Achille Cittadini
Alessandro Sgambato
Andrea Camerini
Carmen Losasso
Gian Paolo Trentini
Giovanni Capelli
Giulio Rossi
Mario Migaldi
Micaela Montanari
Rodolfo Cangiano
P2860
P304
P356
10.1016/S0002-9440(10)63881-3
P407
P577
2003-03-01T00:00:00Z